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  • Pathophysilogy of Primary Hypertension

    Diunggah oleh

    romeo rivera
    7K tayangan1 halaman
    Using the book of Joyce Black (Medical -surgical Nursing) this study guide was made for nursing students. presented using schematic diagram of the process of acquiring primary hypertension.

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    Using the book of Joyce Black (Medical -surgical Nursing) this study guide was made for nursing students. presented using schematic diagram of the process of acquiring primary hypertension.

    Hak Cipta:

    Attribution Non-Commercial (BY-NC)
    Unduh sebagai DOC, PDF atau baca online dari Scribd
    Tandai sebagai konten tidak pantas
    7K tayangan1 halaman

    Pathophysilogy of Primary Hypertension

    Diunggah oleh

    romeo rivera
    Using the book of Joyce Black (Medical -surgical Nursing) this study guide was made for nursing students. presented using schematic diagram of the process of acquiring primary hypertension.

    Hak Cipta:

    Attribution Non-Commercial (BY-NC)
    Unduh sebagai DOC, PDF atau baca online dari Scribd
    Tandai sebagai konten tidak pantas
    dari 1

    PATHOPHYSIOLOGY OF PRIMARY (ESSENTIAL) HYPERTENSION

    1.) Baroreceptor (major 2.) Chemoreceptor reflex activation 3.) Humoral (RAAS)
    stretch receptor) activation by decrease O2 & pH

    Abnormality in the
    Vasomotor center Cardioregulatory center stimulation Increase blood volume
    transport of Na in
    activation
    renal tubules
    Increase venous Increase heart pumping action
    Vasoconstriction return Increase peripheral
    Kidney produces renin vascular resistance
    Increase peripheral Increase blood Increase cardiac Increase in heart rate and
    resistance volume output stroke volume Rennin catalyzes Increase
    Angitensinogen BP/ HPN

    Above normal increase Angiotensinogen convert Vasoconstriction Increase blood


    Prolonged and sustained of blood pressure angiotensin I to II volume
    increase in BP Sympathetic stimulation Inhibition of
    Angiotensin II converted sodium excretion
    to III H2O
    Baroreceptors retention
    continue to “reset” Alteration in chemoreceptor
    autoregulation Angiotensin I and II
    Arterial baroreceptors stimulates adrenal cortex
    Chronic hypertension
    become desensitized
    Sodium reabsorption
    Adrenal cortex
    and potassium
    release aldosterone
    excretion

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