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  • Case Hematology + Genetics I: Β Thalassemia/Hbe Disease

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    Man Lor
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    Thalassemia Decreased beta chains production Mutation of b chain of the 26th amino acid: Glutamic acid Lysine Unstable hemoglobin RBC deformity Increased RBC destruction at spleen change in RBC morphology Bone change Hypochromic microcytic RBC with poikilocytosis.

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    Thalassemia Decreased beta chains production Mutation of b chain of the 26th amino acid: Glutamic acid Lysine Unstable hemoglobin RBC deformity Increased RBC destruction at spleen change in RBC morphology Bone change Hypochromic microcytic RBC with poikilocytosis.

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    11 tayangan1 halaman

    Case Hematology + Genetics I: Β Thalassemia/Hbe Disease

    Diunggah oleh

    Man Lor
    Thalassemia Decreased beta chains production Mutation of b chain of the 26th amino acid: Glutamic acid Lysine Unstable hemoglobin RBC deformity Increased RBC destruction at spleen change in RBC morphology Bone change Hypochromic microcytic RBC with poikilocytosis.

    Hak Cipta:

    Attribution Non-Commercial (BY-NC)
    Unduh sebagai DOCX, PDF, TXT atau baca online dari Scribd
    Tandai sebagai konten tidak pantas
    dari 1

    CASE HEMATOLOGY + GENETICS I

    + thalassemia/HbE disease

    + + thalassemia thalassemia

    HbE disease (Hemoglobinopathy)

    Decreased beta chains production

    Mutation of chain of the 26th amino acid:


    Glutamic acid Lysine

    Unstable hemoglobin

    RBC deformity

    Increased RBC destruction at spleen Change in RBC morphology

    Polychromasia

    5400054 5406181

    Bone change Hypochromic microcytic RBC with poikilocytosis Compensation Extramedullary hemopoiesis

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