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Hemorrhage or other fluid loss decreases intravascular volume

Cardiac output
Tissue perfusion
Compensatory mechanisms are actived
Epinephrine & norepinephrine release


RAAS stimulation
ADH release
ICF shifts to the Ivasc. space


Blood volume
Cardiac output
Cardiac output
HR & SVR
BP
perfusion of vital organ
MSOF
Compensatory mechanisms fail
Development
of hypovolemic
shock
Myocardial injury (acute or progresive)
Tissue perfusion
Compensatory mechanisms are actived
Massive vasoconstriction
Perfusion of vital organ
MSOF
Contractility
Pulmonary edema
Blood pressure
Cardiac output
Development
of cardiogenic
shock
Septicemia
Capillary permiability
Inadequate tissue perfusion
Compensatory mechanisms are actived
Perfusion of vital organ
MSOF
Inflamatory response
Third-spacing of fluid
Massive vasodilasion
The immune system release histamine & many other chemical mediator
Development
of septic shock
Spinal cord injury, spinal anesthesia, or vasomotor center depression
Hypotension
Warm, dry, flushed skin
Perfusion of vital organ
MSOF
Sympathetic tone
Arterial /venous blood pooling
Arterial & venous dilation
Bradycardia
Development
of neurogenic
shock
Antigen re-exposure
Capillary permiability
Angioedema
Urticaria
Pulmonary congestion
Airway obstruction
Respiratory arrest
Cardiac arrest
Hypersensitive antibody response
Vascular collapse
Massive vasodilasion
Vasoactive mediator release
Profound hypovolemia
Development
of anaphylactic
shock