0 penilaian0% menganggap dokumen ini bermanfaat (0 suara)
38 tayangan1 halaman
Diabetic nephropathy is a type of kidney disease caused by diabetes. It results from high blood glucose levels over many years that damage the tiny blood vessels in the kidneys. This causes the kidneys to not function properly by filtering waste from the blood. Symptoms include swelling in the legs and feet, fatigue, numbness, and passing more urine than normal. If left untreated, diabetic nephropathy can lead to kidney failure.
Diabetic nephropathy is a type of kidney disease caused by diabetes. It results from high blood glucose levels over many years that damage the tiny blood vessels in the kidneys. This causes the kidneys to not function properly by filtering waste from the blood. Symptoms include swelling in the legs and feet, fatigue, numbness, and passing more urine than normal. If left untreated, diabetic nephropathy can lead to kidney failure.
Hak Cipta:
Attribution Non-Commercial (BY-NC)
Format Tersedia
Unduh sebagai DOC, PDF, TXT atau baca online dari Scribd
Diabetic nephropathy is a type of kidney disease caused by diabetes. It results from high blood glucose levels over many years that damage the tiny blood vessels in the kidneys. This causes the kidneys to not function properly by filtering waste from the blood. Symptoms include swelling in the legs and feet, fatigue, numbness, and passing more urine than normal. If left untreated, diabetic nephropathy can lead to kidney failure.
Hak Cipta:
Attribution Non-Commercial (BY-NC)
Format Tersedia
Unduh sebagai DOC, PDF, TXT atau baca online dari Scribd
nephropathy kidney glucose level function Thickening in the Fluid retention Beta cells Increased blood stimulated to Ede vessels pressure produce insulin ma caused by the urine Increased Impaired insulin Altered pancreatic Blood protein leak tissue production insulin secretions into the urine resistance to insulin Glucose Ineffective Stressed kidney insulin accumulation in filtration the blood stream/ mechanism hyperglycemia Glucose cannot enter the cells Prolonged Glucosuria without insulin elevation of Cells become Increased glucose in starved of glucose peripheral Polyuria resistance Increased blood viscosity Polyphagia Polydipsia UTI Increased BP Sluggish circulation Increased breakdown of Weight loss protein as source of Fatigue Decreased blood energy Increased flow susceptibilit y to Decreased protein Microvascular Decreased infection disease circulation in lower
Retinopathy Poor wound
Leg Weak Numbness/ Intermittent healing amputat immune tingling claudication ion system Blurred vision