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  • Inflammation Fractures

    Diunggah oleh

    Arvin Ian Penaflor
    130 tayangan2 halaman
    The document discusses the pathophysiology of inflammation following a femur fracture with severe blood loss. It describes the inflammatory process that begins with disruption of cell membranes allowing pyrogens and infectious agents to enter. This starts the inflammatory response mediated by the hypothalamus and various cells and chemicals, resulting in fever, vasodilation, increased white blood cells, swelling, and exudate formation if not treated with antibiotics. If left untreated, it can lead to sepsis, septic shock, and potentially death.

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    The document discusses the pathophysiology of inflammation following a femur fracture with severe blood loss. It describes the inflammatory process that begins with disruption of cell membranes allowing pyrogens and infectious agents to enter. This starts the inflammatory response mediated by the hypothalamus and various cells and chemicals, resulting in fever, vasodilation, increased white blood cells, swelling, and exudate formation if not treated with antibiotics. If left untreated, it can lead to sepsis, septic shock, and potentially death.

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    Attribution Non-Commercial (BY-NC)
    Unduh sebagai DOC, PDF, TXT atau baca online dari Scribd
    Tandai sebagai konten tidak pantas
    130 tayangan2 halaman

    Inflammation Fractures

    Diunggah oleh

    Arvin Ian Penaflor
    The document discusses the pathophysiology of inflammation following a femur fracture with severe blood loss. It describes the inflammatory process that begins with disruption of cell membranes allowing pyrogens and infectious agents to enter. This starts the inflammatory response mediated by the hypothalamus and various cells and chemicals, resulting in fever, vasodilation, increased white blood cells, swelling, and exudate formation if not treated with antibiotics. If left untreated, it can lead to sepsis, septic shock, and potentially death.

    Hak Cipta:

    Attribution Non-Commercial (BY-NC)
    Unduh sebagai DOC, PDF, TXT atau baca online dari Scribd
    Tandai sebagai konten tidak pantas
    dari 2

    Pathophysiology of Inflammation 1 Blood Transfusion supracondylar femur Ruptured of femural arteries and other Severe Blood loss Blood

    vessels that supplying the femur Hypotension and Tachycardia Disruption of Cell Membrane entrance of pyrogens and invasion of infective agents Start of Inflammatory Process Hypothalamus ostaglandin production) Mast Cells (Pr Bone Fracture open type 3

    Fever (value)

    Bradykinin interleukin - 1

    Prostaglandin

    Histamine

    Leukotrienes

    Vasoconstriction Vasodilation increase WBC = (value) Prevent spill of blood Increase permeability Diapedesis or Accumulation of WBC to the interstitial compartment Phagocytosis Swelling sepsis Exudates or suppuration ruptured of the affected part

    Pain treated INFLAMMATION If treated (strong antibiotics) if NOT

    RECOVERY

    Septic shock:

    LEGEND: Blood Pressure BOLD signs/symptoms Decreased Blood Volume NORMAL process ITALIC complications/treatment

    Decrease

    DEATH

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