mediators
Membrane become permeable Plasma leaks out to the interstitial spaces Accumulation on the interstitial spaces Swelling / Edema Increase pressure in the site
Thrombocytes are stimulated 450 x 10 g/L Prothrombin becomes thrombin Thrombin converts fibrinogen Fibrin is produced fibrin barrier is produced
Rednes s
Warmt h
Chemotaxis
Clot Formation
Pain
Further damage and irritation of extensor halluces longus nerve ending Loss of Control and Function
Hyperthermi a
Ruptured Tendon