Note: We did not include the x-ray frames and pictures shown in the lecture
since they were the same as those used in lecture 09 (Overview of Heart
Diseases in Children). Please refer to the pictures under the Congestive Heart
Failure section of the said transcription. Thank you.
I. INTRODUCTION
Congestive Heart Failure
ULTIMATE GOAL: RECOGNIZE and initially treat a CHILD in
HEART FAILURE (HF)
A clinical syndrome in which the heart is unable to pump enough
blood to meet its needs, to dispose of venous return adequately,
or a combination of the two
Definition: inadequate oxygen delivery by the heart or circulatory
system to meet the demands of the body
o Pallor
Increased Sympathetic Stimulation
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o Tachycardia
o Diaphoresis (excessive sweating)
o Arrhythmia
Increased Afterload
o Vasoconstriction cold extremities because of peripheral
vasoconstriction brain heart kidney priority, warmth means
good perfusion less severe heart failure
To perfuse distal organs
C. Physical Examination
General Survey
Failure to thrive (FTT)
o Evaluated either by a low weight for the child's age, or by a low
rate of increase in the weight
Wasting or acute malnutrition
o Caused by an extremely low energy intake, nutrient losses due
toinfection, or a combination of the two
Chromosomal syndromes
o In infants w/ Downs Syndrome, 50% may have cardiac
problems, most of w/c have CAVSD (Complete Atrio-Ventricular
Septal Defect)
o Edwards syndrome (Trisomy 18) 98% will have congenital
heart disease
o So due to such statistics, it is recommended to have a 2D echo
in such cases even if no abnormalities were heard in
ausculation
Color: pale, cyanotic (can help narrow down possible CHDs),
jaundiced (i.e. in liver congestion)
Anxious/irritable compromised oxygen to the brain
Signs Diaphoresis - sweat on the forehead sympathetic
activation
Signs of Cardiorespiratory distress
o Dyspnea, Shortness of breath, Tachypnea
o Subcostal Retractions = Harrisons groove (make sure to note
its presence or absence in routine PE) which indicates poor lung
compliance. But this can also be sings of asthma and
pneumonia
Vital Signs
Sinus tachycardia results in decreased cardiac output
o Sustained CR > 220 /min infants
o Sustained CR > 150/min older children
CR = cardiac rate
Consider Supraventricular Tachycardia
o Abnormal increase in HR HF
o There are instances when tachycardia is the cause of HF
arryhtmia causing HF
o Results in decreased cardiac output
Diastolic filling time decreased in tachycardia decreased stroke
volume
Decreased preload
Hypotension
o May lead to shock
Tachypnea
o Pediatric Respiratory Rates (RR)
Table 1. Normal breath rates per pediatric age group
Age
Rate (breaths/min)
Infant (birth1 yr)
3060
Toddler (13 yrs)
Preschooler (36 yrs)
2440
2234
(compromised
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o Grunting
o Hacking cough
bronchial mucosal edema
CLASS III
CLASS IV
o Rales or crackles
o Wheezing
continuous, coarse, whistling sound
congestion of airway due to pulmonary edema
not exclusive to asthma; can be also cardiac in etiology; but all who
have asthma have wheezing
wheezing despite giving beta-agonists = most probably cardiac in
origin
Abdominal Examination
Hepatomegaly
Ascites (accumulation of fluid in the peritoneal cavity)
Splenomegaly
o PE: dullness to percussion over Traube's space
o NOT associated with CHF but due to hematologic disorders like
beta-thalassemia and leukemia
Examination of the Extremities
Cool extremities:
o warm extremities = well-perfused; most probably, heart does
well
Peripheral pulse
Capillary refill;
o While legs are raised the legs, blood flow should return in < 2
seconds after blanching
Peripheral edema
o Extremely rare in infants; edema found only in dependent area
o Edema usually due to presence of TR (Tricuspid Regurgitation)
o Systemic congestion-dependent edema in adults
HEENT (Head-Eye-Ear-Nose-Throat) Examination
Neck vein distention/engorgement
Difficult to evaluate, small in children; not too common in infants;
in acquired heart disease
o Constrictive pericarditis
o Pericardial effusion
Facial edema/Puffy eyelids
o Again, usually point to a renal etiology in infants
The three-minute examination for CHF (from 2013)
CLASS II
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VI. SUMMARY
Pathophysiology is basically similar to adult CHF.
Clinical manifestations and treatment are grounded on the same
principles as adult CHF.
Diagnosis of CHF is based on History and PE
Diagnosis of cause is based on presentation & age group
Chest x-ray, ECG, & echocardiogram are essential in diagnosis
and treatment of CHF.
Medical treatment is geared towards augmenting oxygen
delivery, decreasing oxygen demand, and tempering
compensatory mechanisms.
Surgery and/or cardiac catherization intervention is often
necessary for definitive treatment.
END OF TRANSCRIPTION
B. Surgical
Definitive depends on the type of CHD (e.g. VSD closure, PDA
transection/ligation, arterial switch operation)
Palliative done to postpone surgery for 6 months until definitive
surgery can be done; a usual occurrence in PGH due to high costs
of definitive surgery; usually costs around 20k only (e.g.
pulmonary artery banding, man-made pulmonic valve stenosis)
C. Pharmacologic
Enhance Oxygen Delivery/Increase Cardiac Output
o Augment Myocardial Contractility (for acute HF)
DIGOXIN/LANOXIN
-AGONISTS: Dobutamine, Dopamine
BIPYRIDINES: Milrinone (an inotrope vasodilator)
o Decrease Afterload
ACE INHIBITORS: Captopril, Enalapril
BIPYRIDINES: Milrinone
ARTERIAL VASODILATORS: Nitroglycerin
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