Cardiac Arrhythmias
ROBERT J. THOMAS, SUDHANSU CHOKROVERTY,
MEETA BHATT, AND TAMMY GOLDHAMMER
ognize the basic abnormalities. The following 1Q PSG segments show some of the cardiac arrhythmias seen during
overnight PSG recording.
171
172
Cardiac Arrhythmias
Alte - HOST v 1.6.03 FUlplronlca C 2001
STAG
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FIGURE 6-1. Severe disease-related bradyarrhythmia in rapid eye movement (REM) sleep, A 56-year-old man with severe daytime
sleepiness. The left side of the snapshot shows a heart rate less than 50, the termination of an apnea, and severe hypoxia (saturation in
the mid-70s). Just prior to the arousal, there is a 4-second period with no electrocardiographs (EKG) rhythm (possible sinus arrest or
sinoatrial exit block), followed by a ventricular escape, and post-arousal tachycardia, with a near doubling of heart rate. The EKG R
wave amplitude also shows fluctuations that track respiratory effort: This EKG-derived respiration signal is secondary to changes in the
cardiac axis generated by positional variation of the heart in the thoracic cavity associated with respiration.
Cardiac Arrhythmias
173
99
99
99
99
99
99
99
99
99
99
99
99
99
99
99
99
99
99
99
99
99
99
99
99
99
98
99
98
98
98
SAO2
Vtot
941
1176
902
1059
1255
941
1137
980
1059
1020
Vest
STM5I REM REM REM REM REM REM REM REM REM REM REM REM REM REM REM REM REM REM REM REM REM REM REM REM REM REM REM REM REM REM
10-
15"
20"
25"
30"
FIGURE 6-2. Ventricular ectopy in REM sleep. Bigeminy and trigeminy is noted in this 26-year-old woman who presented with
nonhypoxic obstructive sleep-disordered breathing. This finding was not seen in non-REM (NREM) sleep. There was excessive use of
caffeine (four to five large cups of strong coffee across the day). The patient has no palpitations during the day or night.
174
Cardiac Arrhythmias
SR SR SR SR SR SR SR SR SR SR SR SR SR SR SR SR SR SR SR SR SR SR SR SR SR SR SR 5R SR SR
C4A1
C3A2
O2A1
O1A2
ROC
LOG
CHIN
EKG
8 8 /
4
-1,6
RR
R-LEG
L-LEG
IPAP 0
EPAP D
0
O
0
O
98
98
98
98
98
98
98
98
99
98
98
98
98
98
98
99
99
99
99
99
59
99
99
SNOR
SA02
Vtot
-39
-39
-39
-39
-39
-39
-39
-39
-39
-39
Vest
STAG
NK WK WK WK WK WK WK WK WK WK WK WK WK WK WK WK WK WK WK WK WK WK WK WK WK WK WK
4:37:51 AM
10"
15"
20"
25"
WK WK WK
30"
FIGURE 6-3. Second-degree heart block, Wenckebach. Progressive increase in PR interval preceding dropped beats in a 48-year-old
patient with delayed sleep phase syndrome, seen unchanged during wake and sleep. This is an innocent arrhythmia and requires no
treatment.
Cardiac Arrhythmias
175
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RAW DATA
FIGURE 6-4. Supraventricular tachycardia. Atrial tachycardia with varying block or a run of atrial flutter with varying block. The
abrupt change in P wave morphology is not associated with any change in QRS morphology. The ability of a single EKG channel in
precisely determining the origin of rhythms can be limited. This patient was not on any medication such as digitalis.
(Rem
C4-A1
01-A2
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Q2-A1
R-EOG
L-EOG
CHN
BCD
Nual Prinure
CHEST
ABDOMEN
SaQ2
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FIGURE 6-5. REM-bradyarrhythmia. A 26-year-old asymptomatic man presented for the evaluation of snoring. Complete heart
block.
CHEST
ABDOMEN
StGQ
FIGURE 6-6. Postarousal tachycardia. A 36-year-old woman presents for the. evaluation of excessive sleepiness in the setting of refractory depression. This pattern of postaronsal bursts of heart rate increase was noted throughout the study. Such cyclic variations in heart
rate have been used to develop EKG-based screening tools for sleep-disordered breathing. One disadvantage of such tools is that severity information cannot be assessed from the degree of heart rate change, and some patients with the worst disease (e.g., heart failure)
may show very little RR variability.
Cardiac Arrhythmias
177
SRSRSRSRSR3RSRSRSRSRSRSRSRSRSRSRSRSR3RSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSRSR
C4A1
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LOG
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EKG
L-LEG
FLOW
NAF
ABD
SAO2
STAG
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FIGURE 6-7. EKG-derived respiration signal. Cyclic variation in RR amplitude (rather than interval) induced by respiratory-related
fluctuations in cardiac positions. This signal has also been evaluated as a noninvasive EKG-derived method to assess sleep respiration.
The disadvantages are similar to RR variabilityit is rarely as prominent as in this sample.
178
Montage:
Cardiac Arrhythmias
PSG Umbs-PFLOW
High Cut: 70 Hz
Low Cut:
0 . 5 3 Hz
Sensitivity:
7 jiV/mm
Speed:
30 s/page
F3-C3
F7-T3
T3-T5
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C3-A2
C4-A1
LT EOG
RT EOG
Chin EMG
P FLOW
Oronasal
Chest
Abdomen
Snore
EKG
Us bpm
Heart Rate
|43 tpm
42 bpm
|42 bpm
Unscored
FIGURE 6-8. Venfricu/ar premature contractions. A 30-second epoch from an overnight PSG study of a 40-year-old woman with a
history of restless legs syndrome reveals the presence of ventricular ectopic beats. The patient reported occasional palpitations. This phenomenon may be rate dependent, and a faster heart rate may not be associated with the ventricular ectopic beats. Sinus arrhythmia is
also noted.
Cardiac Arrhythmias
Montage:
PSG limbs-PFLOW
High Cut:
70 Hz
Low Cut:
0.53 Hz
Sensitivity:
179
7 iiV/nrn
Speed:
30 s/page
F3-C3
F7-T3
T3-T5
T5-01
F4-C4
F8-T4
T4-T6
T6-O2
C3-A2
C4-A1
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LT EOG
Chin EMG
L t . Tib EMG
R t . Tib EMG
P FLOW
Chest
Abdomen
Snore
EKG
FIGURE 6-9. Atrial fibrillation. A 47-year-old man with a history of atrial fibrillation was referred for evaluation of sleep apnea. A 30second epoch of REM sleep from an overnight PSG demonstrates the presence of atrial fibrillation. The association of sleep apnea and
recurrence/triggering of atrial fibrillation has been reported.
180
Cardiac Arrhythmias
High Cut: 50 Hz
LOC/A2
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ROC/A1
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Low Cut;:
0 . 5 3 Hz
Sensitivity:
7 ^V/mm
Speed:
30 s/page
Chin-EMG
C3-A2
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01-A2
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ECG
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Lt Leg EMG
Rt Leg EMG
O2 0.0 1pm
0 cm H2O
Unscored
FIGURE 6-10. Aberrant conduction. A 51-year-old woman was referred for the evaluation of sleep apnea. A 30-second epoch from the
PSG shows premature ventricular complexes with slight widening of the QRS but maintained axis. This may be an aberrant beat
secondary to ajunctional ectopic.