CHRONIC HYPERTENSION
Adult Phase II
Submitted by: Daryl C. Diez
INTRODUCTION
Chronic kidney disease (CKD) is defined as persistent kidney
damage accompanied by a reduction in the glomerular filtration rate (GFR)
causing build up of dangerous levels of fluid, electrolytes and wastes in the
body.
Furthermore, hypertension is one of the leading causes of CKD
due to the deleterious effects that increased BP has on kidney vasculature.
Long-term, uncontrolled, high BP leads to high intraglomerular pressure,
impairing glomerular filtration. Damage to the glomeruli lead to an increase
in protein filtration, resulting in abnormally increased amounts of protein in
the
urine
(microalbuminuria
or
proteinuria).
Microalbuminuria
is
the
presentation of small amounts of albumin in the urine and is often the first
sign of CKD. Proteinuria (protein-to-creatinine ratio 200 mg/g) develops as
CKD progresses, and is associated with a poor prognosis for kidney disease.
Hypertension has been reported to occur in 85% to 95% of
patients with CKD (stages 35). The relationship between HTN and CKD is
cyclic in nature. Uncontrolled HTN is a risk factor for developing CKD, is
associated with a more rapid progression of CKD, and is the eighth leading
cause of ESRD in the Philippines that necessitates the need for replacement
therapy, including dialysis or kidney transplantation (WHO, 2014).
Brief Description:
Statistical Data:
According to the data published by the World Health Organization
(WHO) in May 2014, deaths in the Philippines due to kidney disease reached
15,873 or 3.04% of total deaths. The age adjusted Death Rate is 25.71 per
100,000 of population ranks Philippines number 13 th in the world.
Impact of the disease:
Based on the information from the WHO data last May 2014,
CKD is the eighth leading cause of deaths in the Philippines with
hypertension as one of its main cause. It is also one of the top diseases with
the greatest burden, not only must be dealt with lifelong but consumes great
amount of money. The kidney is a vital part of the body that helps excrete
wastes and maintain homeostasis. Uncontrolled hypertension for a long
period damages the kidneys causing a lot of dysfunctions and problems
affecting different systems of the body that can greatly impact a patients
life.
Lipid Profile
Fasting Blood Sugar
Blood typing
Hep panel
Results
Normal Values
Total Cholesterol
192.05mg/dL
Up to 200 mg/ dL
Triglycerides
253.47mg/dL
Up to 150 mg/dL
HDL cholesterol
37.66 mg/dL
> 35 mg/dL
VLDL cholesterol
50.69mg/dL
< 40 mg/dL
LDL cholesterol
135.05 mg/dL
FBS
104.34 mg/dL
70 105 mg/dL
Serum Creatinine
Blood Urea Nitrogen
14.24mg/dL
65.22 mg/dL
Sodium
155mEq/L
H
4.8mEq/ L
1.08
10.42mg/dl
34g/l
Potassium
Ionized Calcium
Phosphorus
Albumin
Hepatitis B antigen
Hepatitis C antigen
0.5- 1.3mg / dL
7-20 mg / dL
135-145 mEq/L
H
3.5-4.5 mEq/L
1.13-1.32 mmol/L
H 2.6-4.5 mg/ dl
L 35-45 g/l
NEGATIVE
NEGATIVE
Parameter
Reference Range
WBC
9.72 x 10^9/L
5.00 10.00
Neutrophils %
79.1%
Lymphocytes %
11.2%
Monocytes %
8.4%
H 1.0 6.0
Eosinophils %
1.1%
0.0 3.0
Basophils %
0.2%
0.0 1.0
Neutrophils #
7.69 x 10 ^9/L
H 2.00 7.00
Lymphocytes#
1.09 x 10 ^9/L
0.80 4.00
Monocytes#
0,82 x 10 ^9/L
0.12 1.20
Eosinophils#
0.10 x 10 ^9/L
0.02 0.05
Basophils#
0,02 x 10^9/L
0.00 0.10
RBC
3.05 x 10^12/L
HGB
90 g/L
L 140 -180
HCT
22.0 %
Blood type
50.0-70.0
20.0 40.0
4.00 5.00
42.0 54.0
O Positive
Chest X ray revealed increased hydrostatic pulmonary capillary pressure indicative of fluid buildup
(pulmonary congestion). Correlate with other diagnostics
Renal disorder impairs glomerular filtration that resulted to fluid overload. With
fluid volume excess, hydrostatic pressure is higher than the usual pushing excess fluids into
the interstitial spaces. Since fluids are not reabsorbed at the venous end, fluid volume
overloads the lymph system and stays in the interstitial spaces leading the patient to have
edema, weight gain, pulmonary congestion and HPN at the same time due to decrease GFR,
nephron hypertrophied leading to decrease ability of the kidney to concentrate urine and
impaired excretion of fluid thus leading to oliguria/anuria.
Nursing Diagnosis: Excess fluid volume related to compromised regulatory
mechanisms of the body as evidenced by pulmonary congestion on cxr, increased
blood pressure, tissue edema of the lower extremities, weight gain and decreased
urine output
Goal: The patient will be able to display appropriate Maintenance of ideal body
weight without excess fluid as manifested by near normal; stable weight, vital
signs within patients normal range; and absence of edema
Nursing Interventions
INDEPENDENT
1. Assess fluid status:
a. Daily weight
b. Intake and output
balance
Rationale
1. Assessment provides
baseline and ongoing
database for
monitoring changes
and evaluating
interventions.
d. Distention of neck
veins
e. Blood pressure,
pulse rate, and
rhythm
3. Unrecognized sources of
excess fluids may be
identified.
f. Respiratory rate
and effort
4. Understanding promotes
patient and family
cooperation with fluid
restriction.
5. Increasing patient
comfort promotes
compliance with
dietary restrictions.
6. Oral hygiene minimizes
dryness of oral
mucous membranes.
Expected Outcomes
Demonstrates no
rapid weight
changes
Maintains dietary
and fluid
restrictions
Exhibits normal
skin turgor
without edema
Exhibits normal
vital signs
Exhibits no neck
vein distention
Reports no
difficulty
breathing or
shortness of
breath
Performs oral
hygiene
frequently
Reports
decreased thirst
Reports
decreased
dryness of oral
mucous
membranes.
1.
a. Weight changes
b. Laboratory values
(serum electrolyte,
BUN, creatinine,
protein, transferrin,
and iron levels)
2. Assess patient's nutritional
dietary patterns:
a. Diet history
b. Food preferences
c. Calorie counts
3. Assess for factors
contributing to altered
nutritional intake:
Increased dietary
intake is encouraged.
5.
Complete proteins
are provided for positive
nitrogen balance needed
for growth and healing.
6.
c. Depression
d. Lack of understanding
of dietary restrictions
e. Stomatitis
4. Provide patient's food
preferences within dietary
restrictions.
5. Promote intake of high
biologic value protein foods:
eggs, dairy products, meats.
7.
8.
Consumes
protein of high
biologic value
Chooses foods
within dietary
restrictions that
are appealing
Explains in own
words rationale
for dietary
restrictions and
relationship to
urea and
creatinine levels
Takes
medications on
schedule that
does not produce
anorexia or
feeling of
fullness
Consults written
lists of
acceptable foods
Reports
increased
appetite at
meals
Exhibits no rapid
increases or
decreases in
weight
Demonstrates
normal skin
turgor without
edema; wound
healing and
2.
a. Anorexia, nausea, or
vomiting
b. Diet unpalatable to
patient
9.
Reduces source of
restricted foods and
proteins and provides
calories for energy,
sparing protein for
tissue growth and
healing.
Ingestion of
medications just before
meals may produce
anorexia and feeling of
fullness.
Promotes patient
understanding of
relationships between
diet and urea and
creatinine levels to renal
disease.
Lists provide a
positive approach to
dietary restrictions and
a reference for patient
Provide pleasant
surroundings at meal-times.
Weigh patient daily.
Assess for evidence of
inadequate protein intake:
a. Edema formation
b. Delayed wound
healing
c. Decreased serum
albumin levels
COLLABORATIVE
1. Consult with dietitian
support team
DEPENDENT
1. Administer medications
as indicated. Antiemetics:
prochlorperazine
(Compazine),
trimethobenzamide
(Tigan
Unpleasant factors
that contribute to
patient's anorexia are
eliminated.
11.
Allows monitoring of
fluid and nutritional
status.
12.
Inadequate protein
intake can lead to
decreased albumin and
other proteins, edema
formation, and delay in
wound healing.
1. Determines individual
calorie and nutrient
needs within the
restrictions, and
identifies most effective
route and product (oral
supplements, enteral or
parenteral nutrition).
1. Given to relieve N/V and
may enhance oral intake.
acceptable
plasma albumin
levels
Discharge Summary:
The disease has long term effects to the body that can affect the
patient holistically. Lifestyle habits, dietary and activity are some of the changes
that must be thought. The following are some of the activities that must be
educated to the patient:
You will need to return for tests to monitor your kidney function. You may also
be referred to a kidney specialist.
Follow your healthcare provider's directions on how to manage high blood
pressure. These conditions can make CKD worse that may resut to death. Talk
to your healthcare provider before you take over-the-counter medicine.
Weigh yourself daily: Ask your healthcare provider what your weight should be.
Ask how much liquid you should drink each day. CKD may cause you to gain or
lose weight rapidly. Weigh yourself every day. Write down your weight, how
much liquid you drink or eat, and how much you urinate each day. Contact your
healthcare provider if your weight is higher or lower than it should be.
Exercise 30 to 60 minutes a day, 4 to 7 times a week, or as directed. Ask about
the best exercise plan for you. Regular exercise can help you manage CKD and
high blood pressure.
Follow your healthcare provider's advice about what to eat and drink. He may
tell you to eat food low in sodium (salt), potassium, phosphorus, or protein. You
may need to see a dietitian if you need help planning meals. Ask how much
liquid to drink each day and which liquids are best for you.
Do not smoke. Nicotine and other chemicals in cigarettes and cigars can cause
lung and kidney damage. Ask your healthcare provider for information if you
currently smoke and need help to quit. E-cigarettes or smokeless tobacco still
contain nicotine. Talk to your healthcare provider before you use these
products.