Note: This compensation pattern may be driven by ankle or hip dysfunction. If ankle dysfunction is to
blame it will be necessary to release and lengthen the calf complex and activate the tibialis anterior.
Short/Overactive Long/Underactive
Femoral External Femoral External Rotators
Rotators
Hip External Rotator Gluteus Maximus Gluteus Maximus
Piriformis Flexibility Gluteus Medius Activation
Biceps
Femoris Lower Leg Flexibility Gluteus Medius
Adductor Activation
Magnus
Tibialis Anterior
Evertors: Invertors: Activation
Special notes:
This is a tricky dysfunction to analyze. Although you may be tempted to label this Abduction of the
Hip, this leads to the ineffective practice of inhibiting an underactive gluteus medius and activating
the commonly overactive adductors. Practice has shown that the overactive synergists of external
rotation are the primary culprit driving this dysfunction as they attempt to compensate for an
inhibited glute complex during extension (or eccentric flexion).
Believe it or not, if correcting this dysfunction results in Knees Bow In, this is an improvement. This
sign is one of our first compensations within a compensation. If the knees bow in on reassessment
treat the dysfunction as such and use the corrective strategy implied by the table knees bow in.
Excessive Forward Lean = Hip Flexion & Lack of dorsiflexion (a.k.a. excessive plantar flexion)
Short/Overactive Long/Underactive
Hip Flexion Hip Flexor Flexibility Hip Extensors Gluteus Maximus
Tensor Fasciae Adductor Flexibility Gluteus Maximus Activation
Latae (TFL) Lower Leg Flexibilty Semitendinosus & Tibialis Anterior
Psoas Semimembranosus Activation
Iliacus *Biceps Femoris
Rectus Femoris *Posterior Fibers *Tibial Internal
Sartorius of Adductor Rotator Activation
Anterior Magnus
Adductors
Plantar Flexors: Dorsiflexors:
Soleus Tibialis Anterior
Gastrocnemius
Special notes:
In this dysfunction we are forced to confront our first set of strange muscles marked with
an *. By strange I mean they pair a length and activity relationship that is not common.
The muscles denoted by an * are long, but over-active. These are not muscles we want to
stretch, or activate; however, release techniques may be effective for improving function.
The Tibial Internal Rotator Activation is only added as a means of increasing semitendinosus
and semimembranosus activity.
Anterior Pelvic Tilt (Excessive Lordosis) = Hip Flexion & Lumbar Extension
Short/Overactive Long/Underactive
Hip Flexion Hip Flexor Flexibility Hip Extensors Gluteus Maximus
Activation
Tensor Fasciae Adductor Flexibility Gluteus Maximus
Latae (TFL) Semitendinosus & TVA Activation
Psoas Lumbar Extensor Semimembranosus
Iliacus Flexibility *Biceps Femoris Intrinsic Stabilization
Rectus Femoris *Posterior Fibers Subsystem Activation
Sartorius of Adductor
Anterior Magnus Anterior Oblique
Adductors Subsystem Integration
Lumbar Extensors: Trunk Flexors:
In this dysfunction we are forced to confront our first set of strange muscles marked with an *.
By strange I mean they pair a length and activity relationship that is not common. The muscles
denoted by an * are long, but over-active. These are not muscles we want to stretch, or activate;
however, release techniques may be effective for improving function.
Similarly the anterior trunk musculature makes up the Anterior Oblique Subsystem
Asymmetrical Weight Shift Left = Knee Bows Out on Right + Knee Bows in on Left
Note: A single direction was chosen for ease of visualization. Reverse rights and lefts if dysfunction
occurs to the opposite side.
Short/Overactive Long/Underactive
Right Femoral Femoral Left Tibial Tibial Right Invertors Glutues
External Rotators External External Rotators External Tibialis Medius
Piriformis Rotator TFL (via Rotator Anterior Activation
Biceps Flexibility ITB) Flexibility Tibialis Gluteus
Femoris Biceps Lower Posterior Maximus
Adductor Femoris Leg Activation
Magnus Lateral Flexibilty Tibialis
Gastroc Posterior
Right Ankle Lower Left Femoral Left Femoral External Activation
Evertors Leg Internal Rotators Rotators Tibialis
Fibularis Flexibility TFL Gluteus Anterior
(Peroneals) Gluteus Maximus Activation
Lateral Minimus Gluteus
Gastroc Adductors Medius
Special notes:
In future articles this dysfunction will be discussed in more detail. Often what cause an
asymmetrical weight shift is simply having lower leg dysfunction on one side.
This is an abbreviated analysis and solution, for a more thorough look at this dysfunction see
my article Sacroiliac Joint Motion and Predictive Model of Dysfunction
Most often this dysfunction is a compensation within a compensation. A corrective
strategy that resulted in a symmetrical compensation such as, Anterior Pelvic Tilt, Knees
Bow In, or an Excessive Forward Lean would be an improvement.
Arms Fall Forward = Shoulder Internal Rotation
Note: The muscles that cause the shoulders to internally rotate in static standing posture are the
same muscles that would cause extension/adduction of the arms from an overhead position.
Short/Overactive Long/Underactive
Shoulder Internal Pectoralis Major, Shoulder External Rotators External Rotator
Rotators Minor and Infraspinatus Activation
Latissimus Subscapularis Teres Minor
Dorsi Flexibility *Posterior Deltoid
Pectoralis Lumbar Extensor
Major Flexiblity (Lats)
Subscapularis
Special notes:
In this dysfunction we are forced to confront our second strange muscle marked with an
*. By strange I mean it pairs a length and activity relationship that is not common. The
posterior deltoid is long, but over-active. This is not a muscle we want to stretch, or activate;
however, release techniques may be effective for improving function.
It is very rare that shoulder dysfunction exists without scapula and thoracic spine dysfunction.
Most often a corrective strategy would include many of the techniques recommended in the
graph below Shoulders Elevate
Short/Overactive Long/Underactive
Downward Rotators Scapular Muscle Upward Rotators Serratus Anterior
Pectoralis Flexibility Upper and Activation
Minor Lower Trapezius Activation
Levator Trapezius
Scapula Serratus
Rhomboids Anterior