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PATHOPHYSIOLOGY

PHYSIOLOGICAL CAUSES ANATOMICAL CAUSES OTHER CAUSES

Menstrual reflux Decreased tubal Congenital anomalies STDs PID Previous ectopic IUD History of
motility pregnancy abortion

Backflow of Prevents
Salphingitis egg cell Endometrial
matured egg cell Slow movement
Scar Adhesion movement scarring
of matured egg
cell

Narrowing of tubal diameter

Sperm
cell Stasis
FERTILICATION FERTILICATION
enters

Tubal Cervical
Ovarian Abdominal implant implant
implant implant

Cell division via meiosis and mitosis Manifestations Uterine enlargement


of some signs
of pregnancy Elevated HCG level
Increase in size

Stretching of maternal tissues (site of implantation) Nausea and vomiting

Amenorrhea
Activation/release Creation of Unrecognized/ignored
of pain chemicals microscopic cuts
Continues to enlarge
Pain perception Spotting Abnormal
menstruation Inflicts more damage
Dull pain
More pain chemicals are released /activated More microscopic cuts/bigger tissue injuries

Acute lower Nausea and Kehrs sign Bleeding


abdominal vomiting
pain
Peritoneal Vaginal Cul-de-sac

Neck pain Rectal pressure


Rupture

Signs of shock