Exam breakdown(?): 15 for anat, 15 for physio, 15 for neurology, 20 for psych
No histology from Kruger, but know the coursepack learning topics for everyone, not just
Kruger. She didnt ask anything about parkinsons, epilepsy, nothing about optic nerve
specifically this refers to the optic vision. All clinical conditions, symptoms syndromes,
Cranial nerves are important where does it come from, which foramen does it go through
those with 2, 3 nuclei these are important what happens if only one nucleus is affected,
what happens when the affected nuclei are shared amongst others. Spinal tracts, upper
motor and lower motor neuron. 3 learning topics in week 1 belongs to Kruger embryo,
spina bifida, motor and sensory function (sans bladder function), congenital malformation of
CNS, anatomy of memory dementia.
Station 1
Station 2
1. Check slide
2. What is the function: scanning movement both eyes are conjugate/do the same thing
conjugate movement
3. Right frontal eye field a lesion would result in conjugate deviation to the same side as
the lesion
4. Broca motor, expressive speech expressive aphasia understanding but not able to
verbalize. Expressive part cant write as they lack the inability to express, but the
comprehension is intact. They know that they do not make sense. They cannot use sign
language, as it uses expressive part = physically they can, but cannot express. They can
read and comprehend.
5.
6. Receptive aphasia they do not understand, but form proper words, but no structure
random words. They dont know what theyre speaking. They cannot follow instructions.
Station 3
1. Thalamus
3. Receive sensory (primary), interpretive part (association). Receive and interpret signals.
Station 4
Station 5
1. Temporal (some visual association occurs) there are two important components of the
limbic system
2. Hippocampus/hippocampal complex (dentate and parahippocampal), amygdaloid
4. Kluver-Bucy syndrome: (amygdaloid body and hippocampal formation removal results in)
docility, loss of ability to learn, excessive exploratory behavior, (male) abnormal, bizarre and
increased sexual activity. Only amygdaloid bodies removed: most of the symptoms, except
sexual activity is not so pronounced. Only hippocampal gyri: (lack of emotional reponse,
special memory and )inability to learn. Removal of entire temporal lobe = Excessive
Exploratory behavior temporal vision association = put everything in mouth = trying to
figure out what stuff is by putting it in the mouth.
Station 6
1. PPT netters
4. Superior cerebellar tract superior cerebellar midbrain, middle pons, inferior medulla
5. ---------
6. ---------
7. Archicerebellum = balance, cortex of floccuolodular node and vestigial nucleus. Input comes
from vestigial nucleus. Output/efferent is towards the vestigial nucleus (its about balance
and equilibrium). Paleocerebellum: vermis together with globus and emboliform nuclei.
Input is the dorsal and ventral spinocerebellar tract, output is contralateral red nucleus,
cross again to rubrospinal tract, result in ipsilateral due to double decussation. It is
responsible for muscle tone and posture. Neocerebellum: cortex of the cerebellar
hemisphere together with dentate. From the contralateral part of pons, cross over to
neocerebellum, cross over to contralateral part of thalamus, up to motor cortex. Double
decussation = ipsilateral.
Station 7
2. Cerebellar nystagmus: Depending what type results in where the problem is. It is in the
horizontal plane. It is more pronounced when the eyes are looking at one side: most
exaggerated when looking at the side of lesion/ipsilateral. This is due to damage to
flocculonodular.
5. Asynergy: lack of co-ordination in complex task such as buttoning up, speed and skills
affected
7. Dysarthria: motor speech disorder slowness, slurring of speech as due to physical inability
to talk.