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Patient manifested: -fatigue -poor muscle tone -generalized weakness -polydipsia, polyuria, polyphagia. Due to lack of insulin in the body, the glucose level continuously rises because glucose canPt be utilized without the presence of insulin. The cells starved, leading to alteration of metabolism. There will be a breakdown of energy reserved from adipose tissue, muscles and liver (glucagons) this will result to
Patient manifested: -fatigue -poor muscle tone -generalized weakness -polydipsia, polyuria, polyphagia. Due to lack of insulin in the body, the glucose level continuously rises because glucose canPt be utilized without the presence of insulin. The cells starved, leading to alteration of metabolism. There will be a breakdown of energy reserved from adipose tissue, muscles and liver (glucagons) this will result to
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Patient manifested: -fatigue -poor muscle tone -generalized weakness -polydipsia, polyuria, polyphagia. Due to lack of insulin in the body, the glucose level continuously rises because glucose canPt be utilized without the presence of insulin. The cells starved, leading to alteration of metabolism. There will be a breakdown of energy reserved from adipose tissue, muscles and liver (glucagons) this will result to
Hak Cipta:
Attribution Non-Commercial (BY-NC)
Format Tersedia
Unduh sebagai DOCX, PDF, TXT atau baca online dari Scribd
CUES DIAGNOSIS EXPLANATION OUTCOME S> Ø Imbalanced Due to lack of Short term: >Ascertain >To determine Short term: O> Patient Nutrition: less insulin in the After 2 hours of understanding of what The pt shall manifested: than body body, the nursing individual nutritional information to have verbalized -fatigue requirement glucose level interventions, needs be provided to understanding -poor muscle r/t insulin continuously the pt shall client/SO on the tone deficiency rises because verbalize >Discuss eating >To achieve nutritional -generalized glucose can’t be understanding habits and health needs of dietary intake weakness utilized without on the encourage diabetic the patient with AEB taking high -polydipsia the presence of nutritional diet as prescribed the proper food caloric and -polyuria insulin. Glucose dietary intake by the Doctor diet for his protein rich -polyphagia is the source of AEB taking high disease foods like liver energy, while caloric and >Document actual >Patient may be and vegetables. VS taken as insulin is the protein rich weight, do not unaware of their follows: vehicle to foods like liver estimate actual weight or T-36.3 C(axilla) transport glucose and vegetables. weight loss due PR- 95 bpm to the body to estimating RR- 17 bpm BP- 110/70 mmHg tissues. Because weight of decrease >Note total daily >To reveal insulin level in Long term: intake including changes that Long term: the blood stream, After 2 weeks of patterns and time of should be made The pt shall the cells starved, nursing eating in client’s have gain at leading to interventions, dietary intake least 2lbs alteration of the pt. will gain- >Consult > For greater metabolism. The at least 2lbs dietician/physician understanding body needs for further and further glucose for assessment and assessment of metabolism; recommendation specific foods there will be a regarding food breakdown of preferences and energy reserved nutritional support from adipose tissue, muscles and liver (glucagons). This will result to weight loss. But the energy breaks down, the glucose level continuously increase because there is less amount of insulin. The body tissues need to be fed, this will lead to polyphagia and polydipsia because the tissue are not being fed and need glucose for metabolism.
PROBLEM #3 - ACTIVITY INTOLERANCE
ASSESSMENT/ NURSING SCIENTIFIC PLANNING INTERVENTION RATIONALE EXPECTED CUES DIAGNOSIS EXPLANATION OUTCOME S> Ø Activity Because of lack Short term: >Take and >To obtain Short term: O> Patient Intolerance r/t of insulin in the After 2 hours of record VS baseline data The pt shall have manifested: insulin body, there will nursing >Note presence >To identify identified -generalized deficiency / be cell starvation interventions, of factors causative techniques to weakness fatigue / leading to the pt will contributing to /precipitating enhance activity -slow, weak generalized decreased or identify fatigue factors tolerance AEB movement weakness alteration of techniques to >Evaluate >To provide adequate bed -body malaise energy or enhance activity current comparative rest. -fatigue metabolism. tolerance AEB limitations/degre baseline There will be adequate bed e of deficit in VS taken as decreased rest. light or visual follows: energy >Note client >To identify T-36.1 C(axilla) production AEB reports of causative/ PR- 97 bpm body malaise or weakness, precipitating Long term: RR- 16 bpm BP- 110/70 mmHg weakness. Long term: fatigue, difficulty factors The pt shall have Activities need After 3 days of accomplishing been able to energy and nursing tasks report glucose needs interventions, >Assess >To note for measurable insulin to be the pt. will be cardiopulmonary progression/acce increase in transferred to able to report response to lerating degree activity tolerance the body tissues. measurable physical activity of fatigue AEB (-) body Because of lack increase in including VS malaise. of insulin in the activity tolerance before, during body there will AEB (-) body and after activity be decrease malaise. >Adjust or >To assist the pt activity level reduce intensity while managing exerted by the level or activities patient. discontinue activities that cause undesired physiologic changes >Increase >To conserve exercise /activity energy level gradually >Plan care with >To reduce rest periods fatigue between activities >Promote >To enhance comfort ability to measures participate in activities >Plan for >To gradually maximal increase muscle activities within strength of the the client’s ability body PROBLEM #4 - IMPAIRED SKIN INTEGRITY
CUES DIAGNOSIS EXPLANATION OUTCOME S> Ø Impaired skin Type 2 Diabetes Short term: >Monitor and >To obtain Short term: O> Patient integrity r/t Mellitus is a After 2 hours of record VS baseline data The pt shall manifested: wound present condition of nursing >Asses >To determine have -disruption of on the left foot. fasting interventions, condition of skin the presence of demonstrated skin surface with hyperglycemia the pt will over bony necrotic tissue optimal white lesion on that occurs demonstrate prominence for skin/wound care the face despite the optimal breakdown routine AEB the -destruction of availability of skin/wound care every 2 hours absence of skin layers with endogenous routine AEB the >Establish local >To remove infection. varying wounds insulin. Glucose absence of wound care necrotic debris on the right and function as the infection. measure and to attain lest lower energy gives dressing and extremities. body, because treatment of lack of insulin regimens and VS taken as there would be Long term: achieve Long term: follows: inadequate After 3 days of advantageous The pt's skin T-36.2 C(axilla) glucose in the nursing wound healing shall have been PR- 98 bpm body that interventions, >Frequent >To prevent freed from RR- 15 bpm BP- 100/80 mmHg doesn’t supply the pt’s skin will turning and development of further skin other parts of be free of further repositioning other pressure breakdown AEB the organs. With skin breakdown ulcer the performance such having AEB the >Keep >To prevent of wound care. impaired skin performance of surrounding invasion of integrity. wound care. tissue free from microorganisms excess moisture