Pathophysiology: Book-Based
a. Hernia
>Heavy-lifting
>Excessive coughing/
Non - Modifiable Risk
sneezing (COPD)
Factors:
>Strenuous Activities
> Age
>Constipation
> Sex
>Obesity
>smoking
Increased
pressure in the
compartment
in the abdomen
is developed
Intra-
abdominal wall
of inguinal
canal becomes
weakened
Preventing
inguinal ring
from closing
Evolves into a
hole or a defect
Fat or part of
the small
intestine slide
through the
inguinal canal
Swollen or enlarged
inguinal area on the
abdomen/ swollen or
enlarged scrotum
>bloated abdomen
b. Ileus / Ileal Perforation
>Abdominal surgery
>Thrombosis, embolism
Cessation of
peristalsis
MECHANICAL
OBSTRUCTION
Gases and
fluids Bacterial
Borborygmi Activity
accumulate in
the area
Increased contractions
intestine
Increased
intra-luminal Persistent
pressure vomiting
Severe
colicky
abdominal
pain
Inc
secretions into Loss of
hydrogen ions,
the intestine
potassium
Compression
of veins
Metabolic
alkalosis:
Increased venous >Confusion
pressure Hand tremor
>Lightheadedness
>Muscle twitching
> Prolonged
muscle spasms
Decreased
(tetany)
absorption
Edema of the
intestine
Dec. arterial
blood supply
Compression of
terminal branches
of mesenteric
Ischemia
artery
Perforation of Bacteria or
Necrosis necrotic toxins leak
segments into:
Gangrenous
intestinal wall
Blood
Peritoneal
cavity supply
Decreased
bowel Cessation of
sounds peristalsis
Bacteremia,
Peritonitis
Septicemia
Complications:
> Peritonitis
a. Hernia
Factors:
>Heavy-lifting
>Age: 45 y/o
>Strenuous Activities
Increased
pressure in the
compartment
in the abdomen
is developed
Intra-
abdominal wall
of inguinal
canal becomes
weakened
Preventing
inguinal ring
from closing
Evolves into a
hole or a defect
Part of the
small intestine
slide through
the inguinal
canal
Swollen or enlarged
mass at inguinal area
on the abdomen
>Bloated abdomen
b. Ileus / Ileal Perforation
—————————————
————————————————-
Cessation of
peristalsis
MECHANICAL
OBSTRUCTION
Gases and
fluids Bacterial
Borborygmi
accumulate in Activity
the area
Increased contractions
intestine
Increased
intra-luminal Persistent
pressure vomiting
Severe
colicky
abdominal
pain
Inc secretions
into the
Compression
of veins
Increased venous
pressure
Decreased
absorption
Edema of the
intestine
Dec. arterial
blood supply
Compression of
terminal branches
of mesenteric
artery
Ischemia
Perforation of
Bacteria or
Necrosis necrotic
toxins leak
segments
into:
Gangrenous
intestinal wall
Blood
Decreased
bowel Cessation of
sounds peristalsis
Bacteremia,
Septicemia