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Asuhan Keperawatan

Klien dengan Trauma dada

Tuti Herawati, SKp, MN
• Struktur Organ: Jantung, pembuluh darah besar,
esofagus, trakeobronkial dan paru-paru
• 25% of kematian akibat KLL disebabkan karena trauma
• Trauma abdomen umumnya disertai dengan trauma
• Penyebab: trauma tumpul dan trauma tajam
• Fokus pencegahan
Sistem yang memperbaiki keselamatan penumpang
seperti airbags, safety belt
Anatomi & Fisiologi Thorax
• Tulang dinding thorax
– 12 pasang tulang-tulang iga yang berbentuk C
• Ribs 1-7: Join at sternum with cartilage end-points
• Ribs 8-10: Join sternum with combined cartilage at 7 th rib
• Ribs 11-12: No anterior attachment
– Sternum
• Manubrium
– Joins to clavicle and 1st rib
– Jugular Notch
• Body
– Sternal angle (Angle of Louis)
» Junction of the manubrium with the sternal body
» Attachment of 2nd rib
• Xiphoid process
– Distal portion of sternum
• Trauma Tajam
Panah, pisau, handguns,
Shotguns, tergantung jarak
dengan senjata dan kaliber.
Type I: >7 meters: injuri jaringan
Type II: 3-7 meters : penetrasi
ke fascia dan organ internal
Type III: <3 meters: kerusakan
jaringan yang hebat.
Penyebab Trauma dada
Trauma Tumpul
Injuri yang menyertai trauma pada
• Closed pneumothorax • Tracheobronchial tree
• Open pneumothorax lacerations
(including sucking chest • Esophageal lacerations
wound) • Penetrating cardiac
• Tension pneumothorax
• Pericardial tamponade
• Pneumomediastinum
• Spinal cord injuries
• Hemothorax • Diaphragm trauma
• Hemopneumothorax • Intra-abdominal
• Laceration of vascular penetration with
structures associated organ injury
Dinding dada
• Contusion
– Umumnya disebabkan trauma tumpul
– Signs & Symptoms
• Erythema
• Ecchymosis
• Nyeri saat bernafas
• Suara nafas yang menurun Limited breath sounds
• HYPOVENTILAsi (nyeri saat bernafas)
• Crepitus
• Gerakan dinding dada paradox
Dinding dada
• Fraktur Iga
– >50% trauma dada disebabkan oleh trauma tumpul
– Compressional forces flex and fracture ribs at
weakest points
– Iga 1-3 diperlukan kekauatan yang besar bila terjadi
fraktur, dapat menuebabkan injuri paru
– Iga 4-9 tempat yang paling umum terjadi farktur
– Iga 9-12 jarang terjadi fraktur
• Transmisikan energy trauma ke organ internal
• Bila fraktur, curigai adanya injuri hepar dan limpa
– Hypoventilasi umum terjadi karena nyeri
Dinding dada
• Sternal Fracture & Dislocation
– Associated with severe blunt anterior trauma
– Typical MOI
• Direct Blow (i.e. Steering wheel)
– Incidence: 5-8%
– Mortality: 25-45%
• Myocardial contusion
• Pericardial tamponade
• Cardiac rupture
• Pulmonary contusion
– Dislocation uncommon but same MOI as fracture
• Tracheal depression if posterior
Dinding dada
• Flail Chest
– Segment of the chest that becomes free to move with
the pressure changes of respiration
– Three or more adjacent rib fracture in two or more
– Serious chest wall injury with underlying pulmonary
• Reduces volume of respiration
• Adds to increased mortality
– Paradoxical flail segment movement
– Positive pressure ventilation can restore tidal volume
Paradoxical chest wall movement
The point of insertion in the chest most commonly occurs on the
side (lateral thorax), at a line drawn from the armpit (anterior
axillary line) to the side (lateral) of the nipple in males, or to the
side (about 2 in [5 cm]) above the sternoxiphoid junction (lower
junction of the sternum, or chest bone) in females.
Injury Paru-Paru
• Simple Pneumothorax
– Closed Pneumothorax
• Progresses into Tension Pneumothorax
– Occurs when lung tissue is disrupted and air leaks
into the pleural space
– Progressive Pathology
• Air accumulates in pleural space
• Lung collapses
• Alveoli collapse (atelectasis)
• Reduced oxygen and carbon dioxide exchange
• Ventilation/Perfusion Mismatch
– Increased ventilation but no alveolar perfusion
– Reduced respiratory efficiency results in HYPOXIA
Injury Paru-Paru
• Open Pneumothorax
– Free passage of air between atmosphere and pleural
– Air replaces lung tissue
– Mediastinum shifts to uninjured side
– Air will be drawn through wound if wound is 2/3
diameter of the trachea or larger
– Signs & Symptoms
• Penetrating chest trauma
• Sucking chest wound
• Frothy blood at wound site
• Severe Dyspnea
• Hypovolemia
Injuri Paru-paru
• Tension Pneumothorax
– Buildup of air under pressure in the thorax.
– Excessive pressure reduces effectiveness of
– Air is unable to escape from inside the pleural
– Progression of Simple or Open Pneumothorax
Pulmonary Injuries
Tension Pneumothorax Signs & Symptoms

• Dyspnea • Diminished then absent

– Tachypnea at first breath sounds on injured
• Progressive side
ventilation/perfusion • Cyanosis
mismatch • Diaphoresis
– Atelectasis on • JVD
uninjured side • Hypotension
• Hypoxemia • Hypovolemia
• Hyperinflation of injured • Tracheal Shifting
side of chest – LATE SIGN
• Hyperresonance of
injured side of chest
Injuri Paru-Paru
• Hemothorax
– Accumulation of blood in the pleural space
– Serious hemorrhage may accumulate 1,500 mL of
• Mortality rate of 75%
• Each side of thorax may hold up to 3,000 mL
– Blood loss in thorax causes a decrease in tidal
• Ventilation/Perfusion Mismatch & Shock
– Typically accompanies pneumothorax
• Hemopneumothorax
Hemothorax sign & symptoms
• Blunt or penetrating chest trauma
• Shock
– Dyspnea
– Tachycardia
– Tachypnea
– Diaphoresis
– Hypotension
• Dull to percussion over injured side
Pulmonary Injury
• Pulmonary Contusion
– Soft tissue contusion of the lung
– 30-75% of patients with significant blunt chest trauma
– Frequently associated with rib fracture
– Typical MOI
• Deceleration
– Chest impact on steering wheel
• Bullet Cavitation
– High velocity ammunition
– Microhemorrhage may account for 1- 1 ½ L of blood
loss in alveolar tissue
• Progressive deterioration of ventilatory status
– Hemoptysis typically present
Cardiovascular injury
• Myocardial Contusion
– Occurs in 76% of patients with severe blunt chest trauma
– Right Atrium and Ventricle is commonly injured
– Injury may reduce strength of cardiac contractions
• Reduced cardiac output
– Electrical Disturbances due to irritability of damaged myocardial cells
– Progressive Problems
• Hematoma
• Hemopericard
• Myocardial necrosis
• Dysrhythmias
• CHF & or Cardiogenic shock
Myocardial contusion sign & symptom

• Bruising of chest wall

• Tachycardia and/or irregular rhythm
• Retrosternal pain similar to MI
• Associated injuries
– Rib/Sternal fractures
• Chest pain unrelieved by oxygen
– May be relieved with rest
• Similar signs and symptoms of medical chest pain
Cardiovascular injury
• Pericardial Tamponade
– Restriction to cardiac filling caused by blood or other
fluid within the pericardium
– Occurs in <2% of all serious chest trauma
• However, very high mortality
– Results from tear in the coronary artery or penetration
of myocardium
• Blood seeps into pericardium and is unable to escape
• 200-300 ml of blood can restrict effectiveness of cardiac
– Removing as little as 20 ml can provide relief
Pericardial Tamponade Signs & Symptoms

• Dyspnea • Kussmaul’s sign

– Decrease or absence of
• Possible cyanosis JVD during inspiration
• Beck’s Triad • Pulsus Paradoxus
– JVD – Drop in SBP >10 during
– Distant heart tones
– Due to increase in CO2
– Hypotension or during inspiration
narrowing pulse • Electrical Alterans
pressure – P, QRS, & T amplitude
changes in every other
• Weak, thready pulse
cardiac cycle
• Shock • PEA
Cardiovascular injury
• Traumatic Aneurysm or Aortic Rupture
– Aorta most commonly injured in severe blunt or penetrating trauma
• 85-95% mortality
– Typically patients will survive the initial injury insult
• 30% mortality in 6 hrs
• 50% mortality in 24 hrs
• 70% mortality in 1 week
– Injury may be confined to areas of aorta attachment
– Signs & Symptoms
• Rapid and deterioration of vitals
• Pulse deficit between right and left upper or lower extremities
Assessment of the Thoracic
Trauma Patient
• Scene Size-up
• Initial Assessment
• Rapid Trauma Assessment
– Observe
• JVD, SQ Emphysema, Expansion of chest
– Palpate
– Auscultate
– Percuss
– Blunt Trauma Assessment
– Penetrating Trauma Assessment
• Ongoing Assessment
General Management of the Chest Injury Patient

• Ensure ABC’s
– High flow O2 via NRB
– Intubate if indicated
– Consider overdrive ventilation
• If tidal volume less than 6,000 mL
• BVM at a rate of 12-16
– May be beneficial for chest contusion and rib fractures
– Promotes oxygen perfusion of alveoli and prevents atelectasis
• Anticipate Myocardial Compromise
• Shock Management
– Fluid Bolus: 20 mL/kg
Management of the Chest Injury
• Rib Fractures
– Consider analgesics for pain and to improve
chest excursion
• Morphine Sulfate
• Nitrous Oxide
– May migrate into pleural or mediastinal space and
worsen condition
Management of the Chest Injury
• Sternoclavicular Dislocation
– Supportive O2 therapy
– Evaluate for concomitant injury
• Flail Chest
– Place patient on side of injury
• ONLY if spinal injury is NOT suspected
– Expose injury site
– Dress with bulky bandage against flail segment
• Stabilizes fracture site
– High flow O2
• Consider PPV or ET if decreasing respiratory status
Management of the Chest Injury
• Open Pneumothorax
– High flow O2
– Cover site with sterile
occlusive dressing
taped on three sides
– Progressive airway
management if
Management of the Chest Injury
• Tension Pneumothorax
– Confirmation
• Auscultaton & Percussion
– Pleural Decompression
• 2nd intercostal space in
mid-clavicular line
• Consider multiple
decompression sites if
patient remains
• Large over the needle
catheter: 14ga
Management of the Chest Injury
• Hemothorax
– High flow O2
– 2 large bore IV’s
• Maintain SBP of 90-100
• EVALUATE BREATH SOUNDS for fluid overload
• Myocardial Contusion
– Monitor ECG
• Alert for dysrhythmias
– IV if antidysrhythmics are needed
Management of the Chest Injury
• Pericardial Tamponade
– High flow O2
– IV therapy
– Consider pericardiocentesis; rapidly deteriorating
• Aortic Aneurysm
– AVOID jarring or rough handling
– Initiate IV therapy enroute
• Mild hypotension may be protective
• Rapid fluid bolus if aneurysm ruptures
– Keep patient calm
Diagnosa keperawatan
1. Ketidakefektifan pola pernapasan berhubungan dengan ekpansi paru yang
tidak maksimal karena akumulasi udara/cairan.
2. Inefektif bersihan jalan napas berhubungan dengan peningkatan sekresi
sekret dan penurunan batuk sekunder akibat nyeri dan keletihan.

3. Perubahan kenyamanan : Nyeri akut berhubungan dengan trauma jaringan

dan reflek spasme otot sekunder.

4. Kerusakan integritas kulit berhubungan dengan trauma mekanik terpasang

bullow drainage.

5. Hambatan mobilitas fisik berhubungan dengan ketidakcukupan kekuatan

dan ketahanan untuk ambulasi dengan alat eksternal.

6. Risiko terhadap infeksi berhubungan dengan tempat masuknya organisme

sekunder terhadap trauma.
Ketidakefektifan pola pernapasan berhubungan dengan ekspansi paru
yang tidak maksimal karena trauma.

Tujuan : Pola pernapasan efektive.

Kriteria hasil :
o Memperlihatkan frekuensi pernapasan yang efektive.
o Mengalami perbaikan pertukaran gas-gas pada paru.
o Adaptive mengatasi faktor-faktor penyebab.

Intervensi :
Berikan posisi yang nyaman, biasanya dnegan peninggian kepala tempat
tidur. Balik ke sisi yang sakit. Dorong klien untuk duduk sebanyak mungkin.
Observasi fungsi pernapasan, catat frekuensi pernapasan, dispnea atau
perubahan tanda-tanda vital.
Jelaskan pada klien bahwa tindakan tersebut dilakukan untuk menjamin
Intervensi …
• Jelaskan pada klien tentang etiologi/faktor pencetus adanya sesak
atau kolaps paru-paru.
• Pertahankan perilaku tenang, bantu pasien untuk kontrol diri
dnegan menggunakan pernapasan lebih lambat dan dalam.
• Perhatikan alat chest drainase berfungsi baik, cek setiap 1 – 2 jam
• Periksa pengontrol penghisap untuk jumlah hisapan yang benar.
• Periksa batas cairan pada botol penghisap, pertahankan pada batas
yang ditentukan.
• Observasi gelembung udara botol penampung.
• Posisikan sistem drainage slang untuk fungsi optimal, yakinkan
slang tidak terlipat, atau menggantung di bawah saluran masuknya
ke tempat drainage. Alirkan akumulasi drainase bila perlu.
• Catat karakter/jumlah drainage selang dada.
• Kolaborasi dengan tim kesehatan lain
Pemberian analgetika.
Konsul photo toraks.
Inefektif bersihan jalan napas berhubungan dengan peningkatan sekresi
sekret dan penurunan batuk sekunder akibat nyeri dan keletihan.

Tujuan : Jalan napas lancar/normal

Kriteria hasil :
• Menunjukkan batuk yang efektif.
• Tidak ada lagi penumpukan sekret di sal. pernapasan.
• Klien nyaman.

Intervensi :
• Jelaskan klien tentang kegunaan batuk yang efektif dan mengapa
terdapat penumpukan sekret di saluran pernapasan.
• Ajarkan latihan pernapasan dan batuk efektif
• Auskultasi paru sebelum dan sesudah klien batuk.
• Ajarkan mempertahankan hidrasi yang adekuat; meningkatkan
masukan cairan 1000 sampai 1500 cc/hari bila tidak kontraindikasi.
• Dorong atau berikan perawatan mulut yang baik setelah batuk.
• Lakukan penghisapan lendir jika diperlukan
• Kolaborasi dengan tim kesehatan lain : Pemberian expectoran,
pemberian analgesik,