Form D GU
Form D GU
Kelamin :Perempuan
Program :Keperawatan
Paralel :A
Photo
Tanda Tangan,
(......................................)
FORM “D” Adult Assessment Case
REPORT OF PRACTICAL EXPERIENCE
MEDICAL SURGICAL NURSING PRACTICE II
I. Demographic Data
Pt. Name : Pasien a
Room/ Bed # :-
Medical Record # : diabetes mellitus
Age :58 year(s)
Gender : male female
Marital status : single married divorced
Address : Jl. Prof. Soemantri Brodjonegoro No.1
Occupation : indonesia
Religion :-
Medical Diagnosis : Diagnosa gagal ginjal kronik stadium V ec.
Nefropati diabetes + Diabetes Melitus tipe 2 + Hipertensi grade I + Ulkus diabetikum
Physician Name : dr. Sandra Rini, S.Ked.
II. Definition of Disease** (must be with at least four definition and four references)
Gagal ginjal kronik ditandai dengan adanya gangguan fungsi ginjal dan penurunan progresif dan
irreversible dari laju filtrasi glomerulus (GFR). Gagal ginjal kronik telah menjadi masalah
kesehatan dunia.
1 Prevalensi gagal ginjal kronik di seluruh dunia sebesar 8 – 16%.
2 Penyakit ini bukan hanya menyebabkan masalah kesehatan tetapi juga menimbulkan masalah
ekonomi. Di Amerika Serikat setiap tahunnya dibutuhkan biaya $49.3 juta untuk pengobatan
GGK.
3 Komplikasi dari gagal ginjal kronik adalah penurunan kognitif, anemia, gangguan tulang dan
mineral hingga kematian. Dimana diabetes mellitus merupakan penyebab utama dari gagal ginjal
di dunia.
Penyakit ginjal kronik (Chronic Kidney Desease) adalah keadaan dimana terjadi penurunan fungsi
ginjal yang cukup berat secara perlahan – lahan (menahun) disebabkan oleh berbagai penyakit ginjal.
Peyakit ini bersifat progresif dan umumnya tidak dapat pulih kembali (irreversibel). Gejala penyakit
ini umumnya adalah tidak ada nafsu makan, mual, muntah, pusing, sesak nafas, rasa Lelah, edema
pada kaki dan tangan serta uremia. Apabila nilai Glomerulo Filtration Rate (GFR) atau Tes Kliren
Kreatinin (TKK) < 25 ml/menit, diberikan Diet Rendah Protein (Almatsier, 2004).
Reference(s): 1. Levey AS, Coresh J. Chronic Kidney Disease. Lancet. 2012; 379(9811):165-80. 2. Jha V, Garcia G,
Iseki K, Li Z, Naicker S, Plattner B, Saran R, Wang AY, Yang CW. Chronic Kidney Disease: Global Dimension and
Perspectives. Lancet. 2013; 382(9888): 260-72.
III.Etiology* must be with at least four references
- Perokok aktif
- Etiologi memegang peran penting dalam memperkirakan perjalanan klinis Gagal Ginjal
Kronik (GGK) dan penaggulangannya. Penyebab primer Gagal Ginjal Kronik (GGK) juga
akan mempengaruhi manifestasi klinis yang akan sangat membantu diagnose, contoh: gout
akan menyebabkan nefropati gout. Penyeban terbanyak Gagal Ginjal Kronik (GGK) dewasa
ini adalah nefropati DM, hipertensi, glomerulus nefritis, penyakit ginjal herediter, uropati
obstruki, nefritis interstitial. Sedangkan di Indonesia, penyebab Gagal Ginjal Kronik (GGK)
terbanyak adalah glomerulus nefritis, infeksi saluran kemih (ISK), batu saluran kencing,
nefropati diabetic, nefrosklerosis hipertensi, ginjal polikistik, dan sebagainya (Irwan, 2016).
Reference(s): . Sharad P. Pendsey. Understanding Diabetic Foot. Int J Diabetes Dev Ctries. 2010; 30(2): 75–9 .
Reference(s):
Patofisiologi penyakit ginjal kronis berupa kerusakan ginjal yang direpresentasikan oleh penurunan
laju filtrasi glomerulus yang berujung pada berbagai komplikasi.
Ginjal normal memiliki 1 juta nefron (unit satuan ginjal) yang berpengaruh terhadap laju filtrasi
glomerulus. Ginjal memiliki kemampuan untuk menjaga laju filtrasi glomerulus dengan
meningkatkan kerja nefron yang masih sehat ketika ada nefron yang rusak. Adaptasi ini
menyebabkan hiperfiltrasi dan kompensasi hipertrofi pada nefron yang sehat. Hipertensi dan
hiperfiltrasi pada glomerulus merupakan faktor yang berpengaruh besar dalam progresivitas penyakit
ginjal kronis
Laju aliran darah ke ginjal berkisar 400 mg / 100 gram jaringan per menit. Laju ini lebih banyak
dibandingkan dengan aliran ke jaringan lain seperti jantung, hati dan otak. Selain itu, filtrasi
glomerulus bergantung pada tekanan intra dan transglomerulus sehingga membuat kapiler
glomerulus sensitif terhadap gangguan hemodinamik
Peningkatan dasar plasma kreatinin dua kali lipat kurang lebih merepresentasikan penurunan laju
filtrasi glomerulus sebanyak 50%. Contoh: plasma kreatinin dasar senilai 0.6 mg/dL yang meningkat
menjadi 1.2 mg/dL, (masih dalam batas normal), menggambarkan terdapat 50% kerusakan massa
nefron[2].
Peningkatan tekanan kapiler glomerulus dapat menjadi cikal bakal glomerulosklerosis fokal dan/atau
segmental yang kemudian dapat berakhir menjadi glomerulosklerosis global. Membran filtrasi
glomerulus memiliki muatan yang negatif, sehingga membuat hal tersebut menjadi penghalang dari
makromolekul anionik. Dengan penghalang elektrostatik ini, protein pada plasma dapat menembus
filtrasi glomerulus
References:
VI. Assessment
A. Health History
1. Chief Complaints
2. History of Present Illness
3. Past History
a. Childhood Illness : diabetes mellitus sejak 10 tahun
b. Accident(s)
i.Type :
ii.Date/month/year :
c. Allergy :
d. Hospitalization(s)
i.Cause(s) : Nefropati diabetes + Diabetes Melitus tipe 2 + Hipertensi grade I + Ulkus diabetikum
ii.
iii.Date/month/year :
e. Medications : Terapi medikamentosa berupa cairan intravena IVFD NaCl 0,9 % X TPM, Captopril 2
x 12,5 mg, Furosemid Injeksi/ 8 Jam, asam folat 2 x 1 mg dan Glimepiride 1 x 2 mg
=Female
= Male
= Dead Female
= Dead Male
= Pt (male)
= Pt (female)
= Divorced
= Twin
= Adopted
5. Lifestyle
Diet Pattern
Frequency : Frequency :
Contents : Contents :
Others : ____________________________________________________________________________
Frequency : Frequency :
Contents : Contents :
Others : ____________________________________________________________________________
Sleep well Frequently awakened (__x) Sleep well Frequently awakened (__x)
Sleeping position : _____ º ______ pillows
Sleeping position : _____º ______ pillows
Supine Semi Fowler’s High Fowler’s
Supine Semi Fowler’s High Fowler’s
Others (specify)____________
Others (specify)____________
Others : ____________________________________________________________________________
Elimination Pattern
Frequency : Frequency :
Consistency : Consistency :
Diarrhea
Bowel Incontinence
Others : ____________________________________________________________________________
Others : ____________________________________________________________________________
Others : ____________________________________________________________________________
Others : ____________________________________________________________________________
B. Physical Examination
1. Vital Signs
T: 36,6ºC P : 96 x/menix/m R : 24 x/menit x/m
BP: 150/90 mmHg , MAP:
Weight______Kg
Height______Cm
BMI 20,7 kg/m2_ : _(Normoweight)
Nursing Diagnoses:
Hypothermia
Hyperthermia
Level of Consciousness :
Compos Mentis Apathy Somnolence Stuppor Coma
Nursing Diagnoses:
Deficient Fluid Volume
Imbalanced Nutrition: More than body requirements
Skin Integrity, Impaired
Ineffective Airway Clearance
Self-care Deficits
Risk of Aspiration
Communication, Impaired Verbal
______________________________________________________________________
3. Head and neck
a. Head mesocephal nodulelesions scar hematoma bruits sound
normal sinuses tenderness pain facies leonine deformities
&
pale _______________ _________________ ________________
face
Trigeminal (V) nerve function positive negative
Hair scaly dry oily fall bald ____________________
Temporal artery weak strong regular irregular absent
Nursing Diagnoses:
Deficient Fluid Volume
Infection, Risk for
Imbalanced Nutrition: More than body requirements
Hyperthermia
Swallowing, Impaired
Oral Mucous Membrane, Impaired
Skin Integrity, Impaired
Acute/Chronic Pain
Ineffective Airway Clearance
______________________________________________________________________
4. Chest
a. Inspection accessory respiratory muscle use lesions symmetrical thorax
redness scar dry skin warm squama swelling
gynecomastia retraction Barrel chest _________ ________
Breast symmetrical smooth extreme vascularity retraction
bulging discharge bleeding scaly indurations masses
______________
b. Palpation
Apical pulse weak strong regular irregular absent
Tactile fremitus normal increased decreased
c. Percussion resonance hyper-resonance dullness tympani
Diaphragmatic excursion 3-5 cm < 3 cm >5 cm *(______cm)
d. Auscultation bronchovesicular vesicular bronchial tubular/tracheal
crackles wheezing _________________ _______________
Nursing Diagnoses:
Skin Integrity, Impaired
Activity Intolerance
Cardiac Output, Decreased
Ineffective Airway Clearance
Ineffective Breathing Pattern
Impaired Gas Exchange
Self-care Deficit
Tissue Perfusion, Altered
Acute/Chronic Pain
______________________________________________________________________
5. Upper Extremities
a. Inspection anemia jaundice hyper-pigmented vitiligo striae cyanosis
clubbing finger onycholysis ulcer sikatriks nevi deformities
spider nevi hirsutism ecchymosed paronychia ____________
b. Palpation warm tenderness poor skin turgor ____________ ___________
Brachial pulses weak strong regular irregular absent
Radial pulses weak strong regular irregular absent
Ulnar pulses weak strong regular irregular absent
Capillary refill normal slow
Acute/Chronic Pain
Skin Integrity, Impaired
Tissue Perfusion, Altered
Disturbed Body Image
Coping, Ineffective
Infection, Risk for
_____________________________________________________________________
6. Abdomen
a. Inspection symmetrical bulges flat rounded ascites deformities
b. Auscultation
Bowel sounds
c. Percussion
o RLQ resonance hyper-resonance dullness tympani
Nursing Diagnoses:
Acute/Chronic Pain
Activity Intolerance
Constipation
Diarrhea
_____________________________________________________________________
7. Lower Extremities
a. Inspection anemia jaundice hyper-pigmented vitiligo cyanosis striae
excoriations Ulcer sikatriks nevi spider nevi hirsutism
ecchymosed alopecia ___________ ____________________
b. Palpation papule vesicle pustule squama crust nodule tumor
fissure sub-cutis emphysema dry skin moist warm
tenderness poor skin turgor _________________ _______________
Edema pitting grade: 0 (no pitting) +1(mild pitting)
+2 (moderate pitting) +3 (deep pitting) +4 (severe pitting)
Femoral Pulses weak strong regular irregular absent
Senses Left Right
Light/Deep Touch positive negative positive negative
Nursing Diagnoses:
Infection, Risk for
Acute or Chronic Pain
Skin Integrity, Impaired
_________________________________________________________________________________
C. Social Data
Social status single married widow divorced
Social activities organization(s) :_________________________________________________
not involved
Nursing Diagnoses:
Social Interaction, Impaired
Social Isolation
Loneliness, Risk for
Coping, Ineffective
_________________________________________________________________________________
D. Spiritual Data
Worship attendance always often sometimes rare never
Needs priests/friends to pray yes no
Nursing Diagnoses:
Spiritual Distress
Hopelessness
____________________________________________________________________________
E. Psychological Data
Expression sad frowning smiling comfort appearance _________
Emotion anxious afraid angry irritability relax _____________
Coping strategy independent need assistance
Nursing Diagnoses:
Anxiety
Hopelessness
Coping, Ineffective
_________________________________________________________________________________
- Gallop S3
- Nadi diskenetik
- Sinosis perifer
- Ikterik
- Eritema Palma
r - Spider Angiomata
- Gynecomastia pada
lakilaki
- Asterixis
- Uremic fetor
Other Tests
VIII. Medication and Treatment
No. Drug’s Name Classification Dose Contents *Indications *Contraindications *Adverse Reactions *Nursing Implications
3
4
*Indication, contraindication, adverse reactions and nursing implications must be at least one reference
IX. Data Analysis
5
Form NCP (Nursing Care Plans)
Pt. Name: Age: Room/Bed: Medical Diagnosis: Physician’s Name:
Date/ Planning
No. Nursing Diagnosis* Implementation Evaluation
Time Goal* Interventions* Rationale*
Nama Klien:
Room:
Diagnosa:
References: