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Pernicious anemia results from autoimmune destruction of gastric parietal cells that produce intrinsic factor needed for vitamin B12 absorption. This leads to vitamin B12 deficiency and anemia. Predisposing factors include age, lifestyle, and genetics. Bacteria like H. pylori can penetrate the gastric mucosa, weakening the protective mucous layer and allowing acid and pepsin to damage the gastric lining. This causes bleeding, edema, and tissue injury or ulceration in the stomach. Loss of parietal cells and intrinsic factor results in vitamin B12 malabsorption and deficiency, leading to megaloblastic anemia known as pernicious anemia.
Pernicious anemia results from autoimmune destruction of gastric parietal cells that produce intrinsic factor needed for vitamin B12 absorption. This leads to vitamin B12 deficiency and anemia. Predisposing factors include age, lifestyle, and genetics. Bacteria like H. pylori can penetrate the gastric mucosa, weakening the protective mucous layer and allowing acid and pepsin to damage the gastric lining. This causes bleeding, edema, and tissue injury or ulceration in the stomach. Loss of parietal cells and intrinsic factor results in vitamin B12 malabsorption and deficiency, leading to megaloblastic anemia known as pernicious anemia.
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Pernicious anemia results from autoimmune destruction of gastric parietal cells that produce intrinsic factor needed for vitamin B12 absorption. This leads to vitamin B12 deficiency and anemia. Predisposing factors include age, lifestyle, and genetics. Bacteria like H. pylori can penetrate the gastric mucosa, weakening the protective mucous layer and allowing acid and pepsin to damage the gastric lining. This causes bleeding, edema, and tissue injury or ulceration in the stomach. Loss of parietal cells and intrinsic factor results in vitamin B12 malabsorption and deficiency, leading to megaloblastic anemia known as pernicious anemia.
Hak Cipta:
Attribution Non-Commercial (BY-NC)
Format Tersedia
Unduh sebagai DOC, PDF, TXT atau baca online dari Scribd