PREDISPOSING

Non-Modifiable
AGE (loss of blood vessel
elasticity)
GENDER
FAMILY (hereditary)
Modifiable Insufficient insulin
LIFESTYLE secretion/ production of the
DIET (high fat, sweet) pancreas

Glucose is not metabolize

PRECIPITATING
by the body
Diabetes Mellitus

Accumulation of glucose in
the blood stream

Increase fat deposit on Increase viscosity of blood/

the walls of the blood sluggish flow of blood
vessels

Narrowing of blood vessel Atherosclesrosis Evaluate level of

(coronary artery) blood glucose/
increase
concentration of
Pressure on the wall of blood sugar in blood
Decrease blood flow
through the coronary
S/sx: BP: 140/90 mmHg Increase Peripheral Vascular
Dyspnea resistance
S/sx:
Some fragments joins in circulation CBG
CORONARY ARTERY (EMBOLISM) monitoring:
103-166mg/dL
Goes to small vessels and clog (deep
Decrease blood flow
penetrating arteries especially lacunar
to the heart
region)

Increase blood Obstruction/ interruption of O2 supply due

pressure to decreased blood supply

HCVD
Lacunar infarct
Severe elevation in BP
w/o progressive target
organ Decrease oxygen Influences autonomic nervous
supply to basal system
Hypertensive ganglia (L)
Responsible for cerebellar
INFARCTION function
Prognosis
Also affects motor skills of
chewing & swallowing
(DYSPHAGIA)
If treated:
If untreated:
Further Difficulty in chewing &
• Nursing complication
Management swallowing could lead to
can occur: decrease appetite
• Diet • Ischemia
Management • Angina
• Pharmacologic pectoris Causing weight loss
Medication: • MI
(Aspirin, ACE • Heart
inhibitor, attack
Nitroglycerin for
• CVA
angina)
• Oxygen Therapy

Recove Death
ry