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Cirrhosis - a result of many diseases

Friday, 22 April 2011

Cirrhosis of the liver lesions in chronic liver fibrosis caused heavy

damage, spread in the liver lobes. Featuring a strong fibrous tissue,
while the primary structure and blood vessels of the liver lobes were
overturning an irreversible way.

Why have cirrhosis?There are many causes of cirrhosis: Because

diseases such as hepatitis viruses B, D, C; switch long bile, biliary
tract infections caused by stones, sticks or narrow in hepatic duct bile
duct; lack of nutrients, especially lack of protein, vitamin
deficiency, lack of fatty substances make fatty liver leading to
cirrhosis, parasitic infections: schistosomiasis, a small fluke ...
inflamed veins in the liver and rules vena cava below; disorders of iron
pigment in the infection, disease Willson (copper metabolic disorders);galactose
congenital blood disease, metabolic disorders pocphyrin; ... Since
sarcoidosis patients drink more alcohol per day for years. Use of
medicines by causing liver damage: oxyphenisatin (cure constipation),
clopromazin (psychiatric treatment), INH and rifampycin (TB). Caused by
toxic chemicals: aflatoxin, dioxin ... Alkaloids of some species of
plants. Hidden causes cirrhosis: a high rate of cirrhosis of unknown
cause.These factors make long-term impact parenchymal liver
necrosis and liver responded by enhancing cell regeneration and
proliferation of fiber. Fiber organization created walls around the door
with the fibers connecting the central regions of liver lobules,
separating the lobules.The department, created by the new islet
cells causing liver regeneration pinched, hindering the flow of the
portal vein and liver, leading to increased portal vein pressure. The
perimeter of the cavity in the Department of Renewable become
capillaries, leading off the straight vein hepatic vein, the fistula
created inside, making the liver cells anemia portal vein. Due to the
structure of the liver vascular system is disrupted, so the feeding is
declining liver cells, necrosis and status fibrosis is increasing.Depending
injury, one can distinguish three cirrhosis is cirrhosis of potential,
well compensated cirrhosis, fibrosis and advanced liver decompensation.Signs of cirrhosis What?Could
potentially cirrhosis: Although cirrhosis is not symptoms, diagnosis is
only by accident as by other abdominal surgery and treatment that show
cirrhosis.May well compensated cirrhosis: gastrointestinal
disorders such as dyspepsia, bloating, swollen slightly, slightly lower
rib pain right away; nosebleeds of unknown cause, usually deep yellow
urine, decreased sex: male the impotence, women, amenorrhea,
infertility. Check to see steam and make the liver, spleen slopes to the
shore; with capillaries in the back and chest, hairy armpit, in the
genitals are sparse, dry nails and white ...Assay:
Electrophoresis protein found decreased albumin, gamma globulin
increases. Maclagan over 10 units. Liver ultrasound to see. Illuminating
the abdomen and biopsy hurt cirrhosis. This fiber can have long-term
stability may in many years, but often progresses slowly in waves heavy,
especially when there are multiple infections cause disease became
decompensated cirrhosis or cause complications.Can cirrhosis:
Patients lean much worse, said khiu limbs, low blood pressure. Digestive
disorders such as anorexia, dyspepsia, loose bowel movements, fatigue,
less sleep, memory loss. Nosebleeds, bleeding tooth. Check to see dull
skin caused by melanin deposition. There are many clusters in the skin
bleeding feet, hands, shoulders and chest. There may be two feet in
line. Liver atrophy (or to), the density of strong, sharp edge on a
protuberance. Have ascites, collateral circulation. Splenomegaly than
normal, certain density.Assay: Electrophoresis protein found
decreased albumin, gamma globulin increases. Prothrombin ratio
decreased. Blood bilirubin increased, men transaminaza increase in the
sales progress. Erythrocytes, leukocytes, platelets are often reduced.
Capture detector and esophageal varicose veins can see the esophagus.Ultrasound
shows multiple nodules on the liver may sound bold, have doors pictures
varicose veins, varicose veins spleen. Illuminating the abdomen liver
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that can enlarge or shrink, faded, on the liver are the small lumps or
so to a small irregular, splenomegaly, abdominal outbreak. Cirrhosis
after having the symptoms, signs of decompensation, usually the
patient's survival time of approximately 4 years for alcoholic cirrhosis
by hepatitis virus; six years for secondary biliary cirrhosis; 7 years
with the development of cirrhosis

Treatment and prevention How?Prevention:

Use of measures to prevent, eliminate causes of cirrhosis such as viral
hepatitis B vaccination, quit drinking, eating enough, small liver
fluke infection prevention, better treatment of biliary tract diseases,
diseases acute hepatitis, chronic. Limiting the drugs are harmful to the
liver.Using testosterone to enhance protein metabolism, but
must monitor test. Coordination of traditional medicine such as ceiling,
Artichoke, ventricles, bear bile ... Blood transfusion, plasma TV or
alvezin, moriamin if patients with anemia, protein deficiency. Treatment
of ascites: use of diuretics as aldacton, furocemid; dig up water many
times ascites.During the progression of cirrhosis with ascites,
the patient should be absolute rest, preferably in hospital. Eating
enough quality, enough calories from 2500-3000 calories a day, eat
plenty of protein, about 100g/ngay, vitamins, limit fat foods, eat only
light when there is swelling.Only when patients show signs of
hepatic coma new money to limit the amount of protein. The drug improved
liver metabolism, such as high-dose vitamin C 1 g / day, vitamin B12
200 micrograms / day x 10 days; lipochol, cyanidanol ... glucocorticoid
drugs should be used only during the progression of cirrhosis due to
viral hepatitis, cirrhosis cholestatic liver.

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