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  • Acute Glomerulonephritis

    Diunggah oleh

    Chinita Sangbaan
    56 tayangan1 halaman
    The document discusses manifestations and treatment of glomerulonephritis. It lists signs and symptoms across several body systems including hematologic, cardiovascular, integumentary, gastrointestinal, musculoskeletal, neurological and eyes. Common manifestations include anemia, hypertension, fever, edema, azotemia, hyperlipidemia, headache, decreased visual acuity, nausea, vomiting, muscle wasting and acute pain or flank pain. Treatments mentioned include spironolactone, furosemide, captopril, penicillin V potassium. Risk factors include prior streptococcal infection, age, gender and genetics.

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    The document discusses manifestations and treatment of glomerulonephritis. It lists signs and symptoms across several body systems including hematologic, cardiovascular, integumentary, gastrointestinal, musculoskeletal, neurological and eyes. Common manifestations include anemia, hypertension, fever, edema, azotemia, hyperlipidemia, headache, decreased visual acuity, nausea, vomiting, muscle wasting and acute pain or flank pain. Treatments mentioned include spironolactone, furosemide, captopril, penicillin V potassium. Risk factors include prior streptococcal infection, age, gender and genetics.

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    56 tayangan1 halaman

    Acute Glomerulonephritis

    Diunggah oleh

    Chinita Sangbaan
    The document discusses manifestations and treatment of glomerulonephritis. It lists signs and symptoms across several body systems including hematologic, cardiovascular, integumentary, gastrointestinal, musculoskeletal, neurological and eyes. Common manifestations include anemia, hypertension, fever, edema, azotemia, hyperlipidemia, headache, decreased visual acuity, nausea, vomiting, muscle wasting and acute pain or flank pain. Treatments mentioned include spironolactone, furosemide, captopril, penicillin V potassium. Risk factors include prior streptococcal infection, age, gender and genetics.

    Hak Cipta:

    Attribution Non-Commercial (BY-NC)
    Unduh sebagai DOC, PDF, TXT atau baca online dari Scribd
    Tandai sebagai konten tidak pantas
    dari 1

    Manifestations

    Hematologic Cardiovascular Integumentary GIT Muscoskeletal Neurological Eyes

    Anemia Hypertension Fever Azotemia Hyperlipidemia Edema Hyperlipidemia Hypoproteinuria Cyanosis Hypoproteinuria Treatment: Anorexia Vomiting Body malaise Acute pain/Flank pain Peritoneal edema Nausea Muscle wasting Headache Decreased visual acuity

    Spirolanacton e

    (Aldactone)

    Lasix

    L E G E N D: Risk factor

    Pathophysiology

    Risk Factors

    Treatment

    Precipitating: Antigens AB Hemolytic Streptococci

    Manifestations

    Morbillovirus Rubella virus Varicella

    Treatment:

    Predisposing: Age (common during childhood 2-3 y/o but may occur during adulthood. Gender (more common in men) Diet Genetics Proliferation Creatinin, Healthcare Delivery System of endothelial cells lining serum Acute Nausea the glomerular capillary & mysangial BUN Depressionpain/ Altered taste of Decreased Irreversible glomerular & cells lying between endothelium & Increased immunologic Captopril intracranial visual Socioeconomic System Deposition in Glomerulus bud = elevated Feve levels Scarring loss of damage if last for 3 days AntigenInflammatory&response on in vomitin Papillede Na Hypertensio & H2O retention Thickening of response Bacterial/Viral/Potozoal/Fungal triggersepitheliumthe immune pressure defenses BUN (capoten) Toxicit Oliguri Uremi Glomerularacuity damage injury/ =elevate Stimulation of nerve glomerular filtration Antigen-antibody glomerular capillary r glomerular filtration blooda g Headache sensation Decreased GFR ma Flank y n a fibers membrane Treatment:

    ,,

    Penicillin V Potassium

    CBC: Decreased RBC


    Urinalysis: Decreased presence of blood in urine Compensatory Hyperlipidemi Hypoproteinu Cyanosi synthesis of Decreased a ria proteins by liver

    Urinalysis: Capillary membrane swells System permeable to Presence of protein Creatin & become Antistreptolysin-O = presence of streptococcal Crackl Decreased Decreased albumin Proteinur in urine ic in Shortness plasma plasma proteins & blood Muscle es on blood Complement assay = increasedInfectionlevel inoncotic formation Impaired skin of Fatigu Anemi antigen-antibody cells increased vascular Pleural effusionpressure blood of clearan Protein lungs Edema Cola-colored urine antinuclear breathe ia onloss in Decreased o2 supply Edematous alveoli waisting antibodies integrity lungs Hematuria e a ce

    malnutriti Malais on e

    Body

    Risk for infection Anorex Imbalanced nutrition less than body ia

    Hgb Hct

    Triglycerides: Triglycerides: Decreased level of Increased fats in protein in blood blood

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