Objectives
By the end of this session, the students will be able to: Will be able to record good technical E.C.G Understand basic trouble-shooting Identify normal ECG wave.
Electrocardiogram Introduction
The electrocardiogram (ECG) is a diagnostic tool that measures and records the electrical activity of the heart in exquisite detail. Interpretation of these details allows diagnosis of a wide range of heart conditions. These conditions can vary from minor to life threatening.
Electrocardiogram Introduction
The term electrocardiogram was introduced by Willem Einthoven in 1893 at a meeting of the Dutch Medical Society. In 1924, Einthoven received the Nobel Prize for his life's work in developing the ECG.
It is called a 12-lead ECG because it examines the electrical activity of the heart from 12 points of view.
This is necessary because no single point (or even 2 or 3 points of view) provides a complete picture of what is going on.
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Electrocardiogram Introduction
To fully understand how an ECG reveals useful information about the condition of your heart requires a basic understanding of the anatomy (that is, the structure) and physiology (that is, the function) of the heart.
Conducting Tissues
Sino-atrial node (SAN) Fibrous septum Atrio-ventricular node Specialised Conducting Tissue
SAN Depolarisation The events of the cardiac cycle are initiated by depolarisation of the sinoatrial node Atrial Depolarisation The wave of electrical depolarisation is conducted through the cardiac muscle of both atria
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SAN Depolarisation
Atrial Depolarisation
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Atrial Contraction The depolarising wave causes contraction of the atria puching blood into the ventricles AVN Depolarisation The wave of depolarisation reaches: The fibrous septum which does not contract The AVN which depolarises and conducts but slows the wave
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Atrial Contraction
AVN Depolarisation
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Ventricular Depolarisation The wave of depolarisation moves quickly through the specialised conductive tissues Ventricular muscle is depolarised in a cocoordinated, synchronised manner
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Ventricular Contraction The co-coordinated synchronised depolarisation produces an effective contraction of both ventricles Blood is pushed into the aorta and pulmonary arteries
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Ventricular Repolarisation After depolarisation and contraction the ventricles repolarise The bulk of the ventricle means that this electrical event is easily identified on the ECG Repolarisation returns the ventricles back to their resting potential
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SAN Depolarisation The events of the cardiac cycle are initiated by depolarisation of the sinoatrial node
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Atrial Depolarisation The wave of electrical depolarisation is conducted through the cardiac muscle of both atria
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Atrial Contraction The depolarising wave causes contraction of the atria pushing blood into the ventricles
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AVN Depolarisation The wave of depolarisation reaches: The fibrous septum which does not contract The AVN which depolarises and conducts but slows the wave
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Specialised Conducting Tissue The AVN conducts the depolarisation to the Bundle of HIS The Bundle of HIS bifurcates into the left and right bundle branches The impulse is rapidly conducted through these tissues
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Waves of an ECG
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Intervals of an ECG
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ECG Paper
The ECG is recorded on a moving paper ruled at 1mm intervals with darker lines every 5mm At the standard paper speed of 25mm/sec each 1mm horizontally represents 40msec and each 5mm interval 200msec The space between the vertical lines from the top of the paper is 25mm which is equal to 1 second In the vertical dimension, each 10mm represents 0.1mv of electrical potential
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300 divided by number of large squares between R-R 1500 divided by number of small squares between R-R
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Atrial depolarization Lead II will have the tallest P wave AVR will have a negative P wave lead III or AVL will have the smallest P wave. Ventricular repolarization T waves should be upright in leads I, II, AVL, AVF, V2-V6 inverted in AVR T waves are variable in leads III and V1.
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Limbs Lead
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Right wrist = aVR Left wrist = aVL Left leg = aVF Right leg (earth)
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V1 - 4th ICS RSE (Red) V2 - 4th ICS LSE (Yellow) V3 - midway between V2 & V4 (Green) V4 - 5th ICS MCL ( Brown) V5 - 5th ICS AAL (Black) V6 - 5th ICS MAL (Purple)
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The chest leads look at the heart across the horizontal plane The limb leads look at the heart in the vertical plane Leads aVR, aVL and aVF look from three separate directions Leads I,II and III are summation of potential differences between limb leads
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Inferior leads: leads II, III and aVF Look at electrical activity from the inferior or diaphragmatic wall of the left ventricle Septal leads: Leads V1 and V2 looks at electrical activity from the septal wall of the left ventricle. They are often grouped together with the anterior leads
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Rate: 60 to 100/mins Regularity: regular PQRS relationship: present G/C: alert, restful Rx: observe
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Sinus Bradycardia
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Sinus Bradycardia
Rate: < 60/mins Regularity: regular PQRS relationship: present G/C: May be symptomatic i.e. fainting Causes: Sleep, well conditioned athletes, druginduced, SSS or hypothyroidism Rx: Observe, may need IV Atropine
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Sinus Tachycardia
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Sinus Tachycardia
Rate: > 100/mins Regularity: regular PQRS relationship: present Causes : Anxiety, exercise, fever, drug-induced, pain or medical condition - CCF, AMI, etc Rx: Treat underlying cause(s)
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Ventricular Fibrillation
Rate: undeterminable Regularity: chaotic PQRS relationship: absent Causes: MI, drug induced, lightning injury, electrocution Rx: DC shock, AICD
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Ventricular Tachycardia
Rate: > 150/min Regularity: regular PQRS relationship: absent Causes: CAD, MI, dilated cardiomyopathy, electrolytes disturbances, sepsis, etc Rx: If pulseless treat as VF. Pulse VT is unstable, treat with cardio-version, or antiarrhythmias, electrolytes replacement, override pacing
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Asystole
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Questions?
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ECG machine
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ECG machine
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ECG machine
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ECG machine
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End Of Presentation
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