1. Introduction
Ocular trauma is a disease with bimodal age distribution; late of adolescence, early adulthood, & older than 70. Severe ocular trauma, vision threatening eye injuries, effects men 3-5 times as frequently as women Significant cause of visual loss Largely preventable, especially in workplace Ocular trauma is a recurrent disease
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2. Type of injuries
Mechanical injuries
Sharp trauma Blunt trauma
Non-mechanical injuries:
Chemical injuries Photic trauma Electrical trauma
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Immediately transferred to emergency room: Respiratory distress Cardiovascular instability Massive bleeding Acutely impaired mental status
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3.2. History
Details of the traumatic incident should be recorded: 1. Date, time and location of incident 2. Mechanism of injury 3. Accidental, intentional, or self-inflicted injury 4. Accident setting 5. Use of contact lenses, corrective glasses, or safety glasses at a time of accident 6. Presence of witnesses to the accident
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3.3. Examination
Visual acuity Pupils Brightness testing and color vision Visual fields Extraocular motility Intraocular pressure External examination: head, face, periorbital area, eyelid
3.4. Examination
Conjuctiva Cornea Anterior chamber Iris Lens Vitreous Retina and choroid Optic nerve
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Subconjunctival hemorrhage may be spontaneous or the result of trauma. In this patient, the hemorrhage was spontaneous.
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Small metallic foreign bodies have a predilection for the superior tarsal conjungtival surface. In this patient a small fragment of metal is adherent to the conjungtiva
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A.Corneal abrasion stained with fluorescein and illuminated with white light B.Corneal abrasion stained with fluorescein and illuminated with blue light
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Lower lid gently pulled down to show a conjunctival foreign body. The cornea has also been perforated 24
Chemical Injuries
Alkalies
Sulfuric (H2SO4) Sulfurous (H2SO3) Acetic (CH3COOH) - Hydrochloric (HCl) - Chromic (Cr2O3)
Acids
Ammonia (NH3) - Mg(OH)2 Lye (NaOH) - Ca(OH)2 Potassium hydroxide (KOH)
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Chemical Injuries
cont..
Chemical injuries are a true ocular emergencies The amount of tissue damage is directly related to the length of time the chemical remains in contact with the eye Immediate irrigation is vital Chemical composition is also important Alkaline agent tend to penetrate the eye than acids
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Grade I chemical injury :clinical appearance. Epithelial defect involving one quadrant without significant limbal ischemia or evidence of limbal stem cell loss
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Grade II chemical injury : clinical appearance. In the quadrant with epithelial defect there is obvious limbal ischemia and probable lpss of limbal stem cells 29
Traumatic mydriasis
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Iridodialysis
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Management of Hyphema
1. 2. 3. 4. 5. Topical prednisolone acetate 1% 4x daily Cycloplegia is maintained with atropine Worn eye shield full-time Maintain bed rest with minimal ambulatory Keep the head of their be angled at more than 45 degrees 6. Warning sign of rebleeding and elevated IOP 7. Daily follow-up
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Lens-induced glaucoma
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Retinal detachment
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Vascular choroid
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Marginal superior eyelid lacerations Non-marginal inferior eyelid lacerations Superior canalicular lacerations
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Shuttlecocks and squash balls fit neatly inside the orbital rim hence potential for severe injury to the globe larger objects such as footballs hit the orbital rim first.
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Radiograph showing blowout fracture of the left orbit with fluid in the maxillary sinus
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Orbital absess associated with proptosis, restricted extraocular muscle movement, fever, and malaise
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Extensive subconjungtival hemorrhage due to trauma. The examiner needs to consider the possibility of globe rupture or laceration
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Ocular Trauma Score (OTS): Predicting the final vision in the injured eye
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Thank You
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