Hypertensive crises is a critical elevation in the BP with markedly elevated diastolic BP ( !"# $"mmHg% which incl&des HP' Emergency and (rgency
Hypertensive Emergencies is a spectr&m of clinical presentation where &ncontrolled BPs lead to progressive or impending end#organ dysf&nction)damage
Ac&te end#organ damage may incl&de ne&rological, cardiovasc&lar, renal, retinopathy, or eclampsia
,ook for patients with end organ damage and re-&ire immediate I. therapy
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The EP must be capable of treating the patient and not the number
2ost commonly presents with C.A, p&lmonary oedema, hypertensive encephalopathy and CC3
Cere/ral a&toreg&lation is the a/ility of the cere/ral vasc&lat&re to maintain a constant (CB3% across a wide range of perf&sion press&res4
1apid rises in /lood press&re can ca&se hyperperf&sion and increased CB3, which can lead to increased intracranial press&re and cere/ral edema45 6
The left ventricle is unable to compensate for an acute rise in SVR that leads to left ventricular failure and pulmonary edema or myocardial ischemia.[4 ]
hen renal autoregulatory system is disrupted! intraglomerular pressure starts to vary directly "ith the systemic arterial pressure! thus offering no protection to the #idney during $P fluctuations % this can lead to acute renal ischemia.[4 ]
&eath from both ischemic heart disease and stro#e increase progressively as the $P increases
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2ost patients have a history of inade-&ate hypertensive treatment or an a/r&pt discontin&ation of their medications
Subarachnoid Hemorrhage
Encephalitis
An"iety
Systemic 'upus Erythematosus (lomerulonephritis, Acute &o"icity, Amphetamine Headache, Cluster &o"icity, #hencyclidine Headache, &ension
#regnancy, Eclampsia
Differential Diagnoses
Ac&te lowering of BP in the narrow window of the ED visit does not improve long#term mor/idity and mortality rates
2AP sho&ld /e lowered /y no more than !"? in the first ho&r of treatment, then /e lowered to @") ""# " mm Hg in the ne7t !#@ ho&rs if sta/le
Don8t treat BP in ac&te ischemic stroke &nless C!!") !" +1 yo& want to give fi/rinolysis
'reat Ac&te ICB more aggressively with target 0BP D"mmHg for E days
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2aintain 0BPF @"mmHg in 0AH &ntil definitive management and add Gimodipine
.asodilators and nitroglycerin is the preferred agent for ac&te heart fail&re
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Cocaine to7icity associated HP' and tachycardia doesn8t re-&ire specific treatment /&t alpha#adrenergic antagonists to /e given once AC0 kicks in
All hypertensive emergency cases need cons<ation whereas hypertensive &rgencies m&st have an early follow &p
Admit HP' emergency cases to intensive care &nit for close monitoring
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Always watch o&t for complications and treat it like a ticking time /om/
'hank :o&
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Dr&g Apppendi7
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1eferences
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