GENERAL BACKGROUND
Definition
Tissue injury, protein denaturation, edema, and loss of intravascular fluid resulting from exposure to or contact with a causative agent such as heat, electricity, chemicals, radiation, friction, or cold.
Classification
Classification Systems
As to CAUSATIVE AGENT As to DEPTH OF INVOLVEMENT As to SEVERITY
As to CAUSATIVE AGENT
Thermal
Heat Cold
As to DEPTH OF INVOLVEMENT
Old terminology New terminology
OLD TERMINOLOGY
1st degree epidermis only
Edema and redness with necrosis
OLD TERMINOLOGY
Most sources describe burn depth only up to 3rd degree and include the structures affected in 4th degree burns
NEW TERMINOLOGY
Superficial Superficial partial thickness Deep partial thickness Full thickness Subdermal
http://en.wikipedia.org/wiki/Burn_(injury)
As to SEVERITY
American Burn Association (ABA) Classification of Burn Severity
Minor Moderate Major
Epidemiology
For all burns:
1/70 hospitalized Majority are males 17 30 y.o. 66% home-related
Etiology
Thermal Chemical Radiation Electrical Mechanical
Thermal
Heat
Due to rapid heating Types:
Flame burns Scalds Contact burns Flash burns
Cold
Due to rapid freezing
Chemical
Due to exposure to various chemical agents Agents:
Acids Alkali
Radiation
Usually due to exposure to electromagnetic energy radiating agents Agents:
UVR Electrical Radar Radioactive elements (uranium, plutonium)
Electrical
Due to exposure to electrical currents Types:
Low-voltage
<1000V usually 110V (US) or 220V (other countries), both with AC 60 Hz current
High-voltage
>1000V
Mechanical
Usually due to friction
Pathophysiology
Because the skin also serves as protection from infection, the loss of the cutaneous barrier facilitates entry of the patient's own flora and of organisms from the hospital environment into the burn wound. The wound often contains devitalized or frankly necrotic tissue that quickly becomes contaminated with bacteria. Invasive infection (localized and/or systemic) occurs when bacteria penetrate viable tissue, usually below the eschar.
Epidermal Healing
Occurs if there are viable epithelial cells lining the wound Consists of 3 stages
Epithelial Migration Contact Inhibition Epithelial Proliferation
Dermal Healing
Scar formation occurs Even if each phase is described separately, they occur on a continuum and one phase often overlaps another Consists of 3 stages
Inflammatory Phase Proliferative Phase Maturation Phase
Clinical Manifestations
Local Effects:
Loss of ability to regulate evaporative water loss Susceptibility to infection Loss of massive amounts of body fluids, especially in open wounds Local burn wound sepsis 2 to bacterial contamination
Zones of Burn:
Zone of Coagulation Zone of Stasis Zone of Hyperemia
Zone of Coagulation
Dying cells with irreversible damage If not controlled, affectation may involve next zone
Zone of Stasis
Injured cells which may die in 1 2 days if no intervention is done Infection, drying, inadequate wound perfusion in this zone will result in conversion of potentially salvageable tissue to completely necrotic tissue
Zone of Hyperemia
Minimal cell damage with possible recovery up to 7 days without any lasting effects
Complications
SYSTEMIC EFFECTS / PRIMARY COMPLICATIONS:
Acute hypovolemia with loss of fluid to extravascular compartment Pulmonary changes hyperventilation Oxygen consumption If inhalation injury, may lead to pneumonia Acute gastric dilatation/gastrointestinal ileus within 1st 3 days post-burn
SECONDARY COMPLICATIONS
Infection LOM 2 to soft tissue contracture Muscle strength 2 to disuse or nerve involvement Sensory loss 2 to destruction of sense receptors in skin or nerve involvement Auto-amputation
In electrical burns, usually toes/fingers
SECONDARY COMPLICATIONS
Disfigurement, usually 2 to scarring Heterotopic Ossification Associated injuries such as:
Visual loss Neurovascular damage Fracture
Diagnosis
Major concerns as to severity:
Percentage of total body surface area (TBSA) burned Depth of burn
New terminology is used now due to difficulty in specifying actual tissue involvement using old terminology
Differential Diagnosis
Often limited to identifying the causative agent
Prognosis
Factors affecting the severity of a burn injury and its prognosis are:
Depth Extent Age of patient General condition Position/location of the burn Delay of treatment Type of first aid given prior to treatment Other complications present Etiologic agent
MEDICAL MANAGEMENT
Pharmacologic
The main goal is to prevent infection
Through the use of topical antibacterial agents
Tetanus prophylactics are indicated in full thickness burns Sedatives may be applied in major burns due to extreme pain
Medical
Immediate treatments are:
For minor & moderate burns
Ice or cold water Cleaned with soap & warm water Remove loose epithelium Wound dressing Antibacterial agents Tetanus prophylactics if full thickness
Surgical
Escharotomy
To relieve pressure on underlying arteries and veins
Fasciotomy
For persistent impairment of peripheral blood flow
Biologic dressings
For:
Immediate coverage of superficial partial thickness burn Test dressing Wound coverage after escharotomy
Types:
Skin grafts from cadavers Human fetal membranes (homograft or allograft) Skin grafts from pigs (heterografts or xenografts)
Synthetic dressings
Types:
Spray-on polymerics
Only for superficial partial thickness burns < 20% BSA and possible donor sites
Debridement
Types:
Mechanical Enzymatic
Sutilains Travase / Elase
Surgical
Fascial Tangential
CO2 LASER
Other Rehabilitative
Respiratory therapy may be indicated in inhalation injury Speech pathologists may participate if speech is affected due to an inhalation injury Occupational therapists provide:
Skills retraining if affected Dysphagia management if affected due to an inhalation injury
Psychiatric counseling may be indicated if any psychological impact to the injury is noticed
Functional assessment
To assess patients functionality in performing ADL, including:
Basic ADL Instrumental ADL
Problem List
Burn patients often present with the following problems for physical therapy:
Pain Impaired skin integrity
With subsequent affectations in musculoskeletal system
Impaired integumentary integrity associated with full-thickness skin involvement and scar formation Impaired integumentary integrity associated with skin involvement extending into fascia, muscle, or bone and scar formation
If other examination findings indicate inclusion in other diagnostic criteria, patients are placed in other diagnostic labels depending on the other systems affected such as:
Impaired circulation and anthropometric dimensions associated with lymphatic system disorders
Impaired ventilation, respiration/gas exchange and aerobic capacity/endurance associated with airway clearance dysfunction Impaired peripheral nerve integrity and muscle performance associated with peripheral nerve injury Impaired joint mobility, motor function, muscle performance, and range of motion associated with bony or soft tissue surgery Impaired joint mobility, motor function, muscle performance, and range of motion associated with localized inflammation
Plan of Care
Acute care:
Allow rapid wound healing Resolve edema Preserve function Prevent / minimize hypertrophic scarring Prevent respiratory complications Achieve good cosmetic outcome
Interventions
Wound care Functional activities and exercises Scar management techniques Post-healing education
Wound care
Cleansing Debridement Topical agents Dressings
Cleansing
Local wound care Hydrotherapy
Immersion Non-submersion (spray technique)
Debridement
May be done during or after hydrotherapy
Topical agents
Applied after cleansing and debridement
Dressings
Applied after cleansing, debridement, and application of topical agents
Ambulation Strengthening
PRE for involved & uninvolved areas
Endurance
Hips no flexion or external rotation & abducted to 10 from midline Knees extended Ankles dorsiflexed
Compression garments
For management of hypertrophic scarring and edema Worn 24 hours a day up to 1 year until scar matures Types:
Elastic cloth garment Silastic mask Clear plastic mask
Friction massage
To align collagen in healing skin Not done after grafting for at least 5 days Initially gentle and then more aggressive
Post-healing education
Moisturizing newly-healed skin Avoiding direct sunlight
Use of sunscreen Covering affected area with clothing Planning activities in early morning and late evening
After discharge, the patient is followed-up less intensively in physical therapy Depending upon the extent of the burn, the patient will need only 2-3 sessions per week of supervised PT Follow-up the severely burned patient for at least 18-24 months until the scar is completely matured and all rehabilitation complications have been resolved
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