Left Main CA Circumflex Left Anterior Descending CA Right CA Marginal Branch Layers of the Arterial Wall PENDAHULUAN. Anatomi arteria coronarria dan jantung normal Pengertian volume ventrikel kiri
End Systolic Volume (ESV) Volume akir sistol (akir kontraksi ventrikel)
Stroke Volume (SV) = EDV - ESV
Ejection Fraction (EF) = SV EDV Normal darah yg dipompo ventrikel kiri: 62% Hambatan pompa jantung adalah indikator terbaik dari kemampuan kerja jantung dan prognosa kondisi jantung. End Diastolic Volume (EDV) Volume akir diastol (volume akir pengisian ventrikel) 1. Cardiac Output (COP) = Heart Rate X Strooke Volumes 2. Cardiac Index = COP body surface area 3. Preload: Volume darah yang masuk ventrikel saat diastole (End Diastole Volume= reflects stretch of the cardiac muscle cells) 4. Afterload: Tahanan ventricular selama systole (Kemampuan otot ventrikel untuk mendorong darah ke aorta) 5. Frank Starling Law of the Heart - Kemampuan kontraksi otot ventrikel terbesar mulai pre load secara bertahap. 6. Myocardial Contractility Kekuatan kontraksi otot jantung dan perkembangannya sampai preload. 7. Regulated by: 1. sympathetic nerve activity (most influential) 2. catecholamines (epinephrine norepinephrine) 3. amount of contractile mass 4. drugs Pengertian Starlings Law of the Heart and Contractility SV
left ventricular performance preload (venous return) u contractility normal contractility d contractility (heart failure) 0 120 dP/dt dP/dt Normal Heart Failure (lemah jantung) dP/dt = change in pressure per unit of time Gambaran peningkatan saat dan akhir tekanan diastol Curves saat tekanan ventrikel indikasi kemampuan kontraksi Dan fungsi jantung. Perubahan tekanan per tahap Pada jantung normal SV= 60-80 CC Jantung terlatih SV= 90-250 CC Jantung sakit SV = 40-50 CC Kemampuan kontraksi dipengaruhi oleh 1. Kekuatan otot jantung. 2. Fleksibilitas otot jantung. 3. Tahanan perifer (aorta, jaringan, vena 4. Peningkatan max selisih preload dan afterload (dP/dt from LV pressure curve) 5. Pengaruh Positive/negative iontropic. 6. Ejection fraction (EF = SV/EDV) used in clinical practice 7. Hormonal (epineprin atau norepineprin) increase contractility assumed with increase EF with Ca, NE, digitalis, exercise; with [K]o, [Na]o
Contractility related to : 1. sympathetic adrenergic nerves a. catecholamines: epinephrine norepinephrine b. Obat: digitalis sympathomimetics anesthetics, barbiturates 2. Hilangnya kemampuan kontraksi otot misalnya MCI, cardiomyopathy.
1. Perbedaan tekanan oksigen antara darah arteri dan vena. Arteriovenous Oxygen Difference (AVO 2 D) PENGUKURAN DALAM ml % - ml O 2 / 100 ml blood 2. Oxygen Consumption (VO 2 ) Jumlah oksigen yang dibutuhkan darah untuk metabilism dalam menghasilkan energi/ 1. absolute measures: L / min , ml / min 2. relative measures: ml / kg body wt. / min 3. Fick equation: VO 2 = COP X Selisih O2 arteri dan vena 3. Maximum Oxygen Consumption (VO 2max ) Jumlah oksigen yang mampu disediakan secara maksimal per menit untuk metabolism dalam menghasikan energi 1. Tak langsung 220-usia = 60-80 % VO2 maks. 2. Spirometri .
Definisi
4. Myocardial Oxygen Consumption VO 2 of the heart muscle (myocardium) "estimated" by RPP: HR X Sistole BP.
5. Functional Aerobic Impairment:
predicted VO 2max - attained VO 2max predicted VO 2max mild 27% - 40% moderate 41% - 54% marked 55% - 68% severe > 69% Definisi 1. Systolic Blood Pressure (SBP) pressure measured in brachial artery during systole (ventricular emptying and ventricular contraction period) 2. Diastolic Blood Pressure (DBP) pressure measured in brachial artery during diastole (ventricular filling and ventricular relaxation) 3. Mean Arterial Pressure (MAP) "average" pressure throughout the cardiac cycle against the walls of the proximal systemic arteries (aorta) 1. estimated as: .33(SBP - DBP) + DBP 4. Total Peripheral Resistance (TPR) - the sum of all forces that oppose blood flow 1. length of vasculature (L) 2. blood viscosity (V) 3. hydrostatic pressure (P) 4. vessel radius (r)
Definitions TPR = ( 8 ) ( V ) ( L ) ( p ) ( r 4 ) This examination is a particularly important skill to master but is also one of the more complex ones. It not only involves a thorough examination of the heart, but also of the hands, face, neck and other areas of the body. 1. Start, as with all examinations, by introducing yourself and explaining to the patient what you plan to do and ensure that you have their consent. You should also have a chaperone present. Now is a good point to ask the patient to remove their top so that the chest is entirely exposed and place them on the bed with their trunk at 45.
Perform a patient introduction
2. Firstly, you should start by observing the patient from the end of the bed. You should note whether the patient looks comfortable. Are they cyanosed or flushed? Is their respiration rate normal? Are there any clues around the bed such as PCA machines, GTN sprays or an oxygen mask? You should comment on each of the areas to the examiner.
Observe the patient from the end of the bed
3. Next you should look at the patients hands. Initially note how warm they feel as this gives an indication of how well perfused they are. Particular signs which you should be looking for are nail clubbing, splinter haemorrhages, palmar erythema and nicotine staining.
Look at the patients hands
4. Now is a good time to take the radial pulse. It is not a suitable pulse for describing the character of the pulsation, but can be used to assess the rate and rhythm. At this point you should also check for a collapsing pulse a sign of aortic incompetence. Remembering to check that the patient doesnt have any problems with their shoulder, locate the radial pulse and place your palm over it, then raise the arm above the patients head. A collapsing pulse will present as a knocking on your palm. Locate the radial pulse and place your palm over it
Raise the arm above the patients head
5. Examine the extensor aspect of the elbow for any evidence of xanthomata. 6. At this point you should say to the examiner that you would like to take the blood pressure. They will usually tell you not to and give you the value. 7. Next you should move up to the face. Look in the eyes for any signs of jaundice (particularly beneath the upper eyelid), anaemia (beneath the lower eyelid) and corneal arcus. You should also look around the eye for any xanthelasma.
Look in the eyes for any signs of jaundice, anaemia, and corneal arcus
8. Whilst looking at the face, check for any malar facies, look in the mouth for any signs of anaemia such as glossitis, check the colour of the tongue for any cyanosis, and around the mouth for any angular stomatitis another sign of anaemia.
Exam around the patient's face
9. Next, move to the patients neck to assess their jugular venous pressure (JVP). Ask them to turn their head to look away from you. Look across the neck between the two heads of sternocleidomastoid for a pulsation. If you do see a pulsation you need to determine whether it is the JVP if it is then the pulsation is non-palpable, obliterable by compressing distal to it and will be exaggerated by performing the hepatojugular reflex. Having warned the patient that it may cause some discomfort, press down on the liver. This will cause the JVP to rise further. If you decide the pulsation is due to the JVP, note its vertical height above the sternal angle.
Assess the patient's jugular venous pressure (JVP)
10. It is now time to move the examination to the chest, or praecordium. Start by inspecting the area, particularly looking for any obvious pulsations, abnormalities or scars, remembering to check the axillae as wel 11. Palpation of the praecordium starts by trying to locate the apex beat. Start by doing this with your entire hand and gradually become more specific until it is felt under one finger and describe its location anatomically. The normal location is in the 5th intercostals space in the mid-clavicular line. However, it is not uncommon to not feel the apex beat at all. Try to locate the apex beat
12.Next you should palpate for any heaves or thrills. A thrill is a palpable murmur whereas a heave is a sign of left ventricular hypertrophy. A thrill feels like a vibration and a heave feels like an abnormally large beating of the heart. Feel for these all over the praecordium.
Palpate for any heaves or thrill
13. Palpate for any heaves or thrill 14. You now move onto auscultation. This is done for all four valves of the heart in the following areas: Mitral valve where the apex beat was felt. Tricuspid valve on the left edge of the sternum in the 4th intercostal space. Pulmonary valve on the left edge of the sternum in the 2nd intercostal space. Aortic valve on the right edge of the sternum in the 2nd intercostal space. 15. You should listen initially with the diaphragm noting how many heart sounds you can hear are there any extra to the two normal sounds? Are there any murmurs? Are the heart sounds normal in character? Can you hear any rub? If you hear any abnormal sounds you should describe them by when they occur and the type of sound they are producing. Feeling the radial pulse at the same time can give good indication as to when the sound occurs the pulse occurs at systole. Furthermore, if you suspect a murmur, check if it radiates. Mitral murmurs typically radiate to the left axilla whereas aortic murmurs are heard over the left carotid artery.
16.You may also wish to listen with the bell of your stethoscope for any low pitched murmurs. Mitral valve location
Tricuspid valve location
Pulmonary valve location
Aortic valve location
17.To further check for mitral stenosis you can lay the patient on their left side, ask them to breathe in, then out and hold it out and listen over the apex and axilla with the bell of the stethoscope. Further check for Mitral Stenosis 18.Aortic incompetence can be assessed in a similar way but ask the patient to sit forward, repeat the breathe in, out and hold exercise and listen over the aortic area with the diaphragm. Assess for Aortic incompetence 19.Finally you should assess for any oedema. Whilst the patient is sat forward, feel the sacrum for oedema and also assess the ankles for the same. Assess for any oedema