ASSESSMENT OF
THE CRITICALLY ILL PATIENT
Appearance Work of
breathing
Circulation
Abnormal
Abnormal
Abnormal
Abnormal
Abnormal
Assessment
Respiratory distress
Shock
Primary CNS disfungtion/
Metabolic abnormality
Cadiopulmonary failure
History
PHASE I
PHASE II
Subsequent reviews
(Secondary survey)
What is the underlying
cause?
Examination
Chart review,
documentation
Investigations
Treatment
LOOK for
cyanosis, altered respiratory pattern & rate, use of accessory
muscles, tracheal tug, altered level of consciousness,
protective airway reflect (cough, gag)
LISTEN for
noisy breathing (grunting, stridor, wheezing, gurgling)
complete obstruction results in silence
FEEL for
decreased or absent air flow
BREATHING
Cause of inadequate breathing
Depressed respiratory drive: e.g CNS depression
Depressed respiratory effort: muscle weakness, nerve/spinal
cord damage, debilitation, chest wall abnormalities, pain
Pulmonary disorders: pneumo/hemothorax, aspiration, chronic
obstructive pulmonary disease, asthma, pulmonary
embolus, lung contusion, acute lung injury, ARDS,
pulmonary edema
LOOK for
cyanosis, altered respiratory pattern & rate, equality & depth of
respiration, sweating, JVP, use of accessory muscles,
tracheal tug, altered of consciousness, SaO 2
LISTEN for
dyspnea, inability to talk, noisy breathing, percussion,
ausculatation
FEEL for
precordial cardiac pulsation, pulses (central &
peripheral) assessing rate, quality, regularity,
symmetry
Newborn
30 60
Infant
30 40
2 4 yr
20 30
4 7 yr
20 30
7 12 yr
16 20
> 13 yr
12 - 16
5.
3. Cheyne-Stokes respirations
brain damage, cardiogenic shock, uremia, drug induced resp.depress
Slow breathing
drug induced resp.depress, ICP, end-stege resp. muscle fatigue
tachycardia
Retractions
Nasal flaring
Grunting
Stridor or wheezing
Mottled color
Change in responsiveness
Hypoxemia, hypercarbia, decreased Hgb saturations
LATE : poor air entry, weak cry
apnea or gasping
deterioration in systemic perfusion
bradycardia
PULSE OXIMETRY
that estimates functional oxyhemoglobin saturation
Normal SpO2 96% - 99%
CAPNOGRAPHY
To measure PaCO2 level
end tidal CO2 (ET-CO2) CO2 alveolar
Circulation
Causes of circulatory inadequacy
primay-directly involving the heart
ischemia, conduction defects, valvular disorders,
cardiomyopathy
secondary-pathology originating elsewhere
drugs, hypoxia, electrolyte disturbances, sepsis
LOOK for
reduced perpheral perfusion (palor, ), hemorrhage
(obvious concealed), altered level of conciousness,
dyspnea, urine output
LISTEN for
additional or altered heart sounds, carotid bruits
FEEL for
precordial cardiac pulsasion, pulses (central &
peripheral) assessing rate, quality, regularity,
symetry
Assessment of circulation:
pulses rate, cardiac pulsasion, quality & regularity,
skin temperature, & blood pressure
Age (year)
Rate/minut
<1
110 160
12
100 150
25
5 12
> 12
95 140
80 120
60 - 100
Age (year)
Sistolic (mmHg)
<1
70 90
12
80 90
25
80 100
5 12
90 110
> 12
100 - 120
Disability
to assess signs of neurologic compromise precede loss
of conscious
AVPU
Awake
Responsive to voice
Responsive to pain
Unresponsive
4
3
2
1
5
4
3
2
1
6
5
4
3
2
1
4
3
2
1
5
4
3
2
1
6
5
4
3
2
1
Exposure
assessing the face, posture body & skin
rush, hematoma, temperature, etc
2. ASSES:
CIRCULATION
Obstructed breathing or
Central cyanosis or
Severe respiratory distress
C
Cold hands
with:
o
Capillary refill longer than
l
3 seconds, and
Weak and fast pulse
ANY SIGN
POSITIVE
Coma or
Convulsing (now)
IF COMA OR
CONVULSING
Manage airway
If convulsing, give diazepam or
paraldehyde rectally
Position the unconscious child (if head or
neck trauma is suspected, stabilize the
neck first)
Give IV glucose
DIARRHOEA
Plus TWO SIGNS
POSITIVE
Chedk for severe
malnutrition
Check for head/neck trauma before treating childdo not move neck if cervical spine injury possible
NO- URGENT
Proceed with assessment and for treatment according to the childs priority
Children
Slapping the back to clear airway obstruction
Heimlich manoeuvre
Child conscious
Inspect mouth and remove foreign body, if present (neutral
position)
Clear secretions from throat
Let child assume position of maximal comfort
Child unconscious
1.
2.
3.
4.
If the child is still not breathing with carrying out the above,
ventilate with bag & mask
NASAL PRONGS
Place the prongs just inside
the nostrils and secure with tape
NASAL CATHETER
Use an 8F size tube
Measure the distance from the side
of the nostril to the inner eyebrow margin
with the catheter
Insert the catheter to this depth
Secure with tape
SYOK
HIPOVOLEMIK: DEHIDRASI
PERDARAHAN
SYOK DISTRIBUTIF: SEPSIS, DSS,
ANAFILAKSIS
SYOK KARDIOGENIK: CHD,
KARDIOMIOPATI
SYOK OBSTRUKTIF: TAMPONADE
JANTUNG, TENSION
PNEUMOTHORAKS
TAKHIKARDI
TAKHIPNEU
PENGISIAN KAPILER>>
IRITABILITAS RINGAN
TEKANAN NADI MENYEMPIT
KULIT
TENTUKAN STATUS
KARDIOVASKULAR
2. TENTUKAN GANGGUAN
SIRKULASI TERHADAP
PERNAPASAN, TINGKAT
KESADARAN, PERFUSI, SUHU
DAN PRODUKSI URIN
1.
3.
1.
2.
3.
4.
UREUM, KREATININ
THORAKS FOTO
IRAMA JANTUNG?
PTT, aPTT, FDP, TROMBOSIT, D-DIMER
GANGGUAN SSP?
PADA
AWALNYA TERJADI
HIPERPIREKSIA, HIPERVENTILASI,
TAKIKARDI, GANGGUAN KESADARAN,
DAN AKRAL YANG HANGAT
KOMPENSASI CURAH JANTUNG DAN
TAHANAN VASKULAR SISTEMIK
STADIUM
DEKOMPENSASI
(COLD SHOCK)
JIKA TERAPI AWAL GAGAL DAN TUBUH
GAGAL MENGKOMPENSASI, TERJADI
HIPOTENSI, HIPOKSIA DAN METABOLISME
ANAEROB
1.
2.
3.
SYOK
Volume of RL or normal
saline
(20 ml/kg)
75 ml
100 ml
150 ml
250 ml
350 ml
0 min
5 min
15 min
Fluid Responsive
Observase
in PICU
Cathecholamine-resistant shock
At risk of adrenal insufficiency?
Not at risk?
Give hydrocortis
Do not Give hydrocortisone
60 menit
Normal Blood Pressure
Cold Shock
SVC O2 Saturation <70%
Add vasodilatoror
Type III PDE inhibitor
with volume loading
Titrate volume
And Epinephrine
Titrate volume
and norepinephrine
Vasopresin or
angiotensin
Refractory shock
ECMO
Recommnendation for stpewise management of hemodynamic support with
goals of normal perfusion and perfusion pressure (MAP-CVP) in infants and
children
Seizures
5-10 min
Seizures
Yes
No
STOP
5-10 min
prolonged seizure
Seizures
Yes
Airway-breathing-circulation
sign : trauma, infection, paresis
Vein access
Laboratorium : Blood :Glucose, electrolyte
No
STOP
Seizures
Yes
Status of Convulsions
No
12 hours later
fenitoin 5-7 mg/BW iv
Seizures
Yes
PICU, Intubation
No
12 hours later
fenitoin 5-7 mg/BW iv
+ fenitoin 5-7 mg/kg
PENANGANAN
EKSASERBASI AKUT
ASMA PADA BAYI DAN
ANAK
PATHOPHYSIOLOGY
Chemical mediators
Bronchoconstriction, mucosal edema, exes.secret
Airway obstruction
Atelectasis
Hyperinflation
Acidosis
Pulmonary
vasoconstriction
Nonuniform
ventilation
Hyperinflation
Mismatching V/P
Decreased
compliance
Alveolar
Hypoventilation
PaCO2
PaO2
Increased work
of breathing
KLASIFIKASI EKSASERBASI
RINGAN
SEDANG
BERAT
ANCAMAN
HENTI NAPAS
LANJUTAN
LANJUTAN
1.
2.
3.
4.
MEREDAKAN OBSTRUKSI
MENGURANGI HIPOKSEMIA
MENORMALKAN FAAL PARU
MERENCANAKAN
PENANGANAN JANGKA
PANJANG SESUAI KLASIFIKASI
PENYAKIT
LANJUTAN
LANJUTAN
LANJUTAN