2.
3.
4.
1. Atrial fibrillation
2. Atrial flutter with variable
conduction
3. Multifocal atrial tachycardia
(MAT).
1. Atrial tachycardia
Note that the P wave is not
upright in lead II (indicating that
it originates at a site other than
the sinus node, a.k.a. "ectopic").
Each P wave is followed by a QRS
complex which is narrow
(supraventricular in origin). Click
below for a detalied review of
ECG findings in ectopic atrial
1. Sinus tachycardia
2. Acute anterior myocardial infarction
This ECG demonstrates an "extensive anterior" and
"tombstoning" of the ST segment seen during a large
acute anterior myocardial infarction. This is usually the
result of thrombosis of the left anterior descending
coronary artery. In this ECG, the thrombosis would be
proximal in the left anterior descending since the septal
leads (V1 and V2) are involved. Also, note the inferior
changes. Some left anterior descending coronary arteries
"wrap around" the cariac apex and can supply part of the
inferior wall as well which was indeed the case in this
situation. For a complete, detalied review of the ECG
changes during an anterior myocardial infarction, click the
Anterior MI ECG Review button below.
Normal
yolo
1. Sinus tachycardia
2. Left ventricular hypertrophy
1. Sinus tachycardia
2. Anterior myocardial infarction
3. Hyperacute T wave abnormality
Many different T wave abnormalities exist.
Hyperacute T waves are the very first sign of a
myocardial infarction, however are frequently missed
on the ECG since they are only transient during the
first few minutes of the infarction. Hyperacute T
waves tend to be a bit more symmetric and broad at
the top than those "peaked" T waves seen in
hyperkalemia, although this is difficult to distinguish
at times. On this ECG, the ST elevation gives away the
fact that the T waves are hyperacute. Some different
T wave abnormalities are pictorally shown below: