Mechanical
Causes
Lumbar Strain
Degenerative
disease
Discs (spondylosis)
Facet joints
Spondylolisthesis
Herniated/Prolasp
ed disc
Spinal stenosis
Osteoporosis
Fractures
Congenital
disease
Severe kyphosis
Severe scoliosis
Nonmechanical
spine disease
Neoplasia
Multiple myeloma
Metastatic
carcinoma
Lymphoma and
leukemia
Spinal cord tumors
Retroperitoneal
tumors
Infection
Osteomyelitis
Septic discitis
TB
Inflammatory
arthritis (often
HLA-B27 associated)
Ankylosing
spondylitis
Psoriatic
spondylitis
Reiter's syndrome
Visceral
disease/
Referred Pain
Pelvic Organs
Prostatitis
Endometriosis
Chronic pelvic
inflammatory
disease
Renal disease
Nephrolithiasis
Pyelonephritis
Perinephric
abscess
Aortic aneurysm
Gastrointestinal
disease
Pancreatitis
Cholecystitis
Penetrating ulcer
Prolapsed disc
Degenerative spinal
disease
Bone infection or
tumour(pyogenic
osteomyelitis, spinal
TB, spinal
metastases)
Degenerative spinal
disease,
osteoporotic
vertebral
collapse,
malignancy(primary
or secondary from
lung, breast,
prostate,
thyroid, or kidney
Ca),
Multiple myeloma,
spinal stenosis, TB
spine
Red Flags!
Root canal
stenosis, spinal
stenosis
PATHOPHYSIOLOGY OF
DISC DEGENERATION
Glycosaminogl
ycan
diminished>poor water
retention>drying out
Secondary
changes :
segmental
instability
( displacemnet
of facet joints)
Osteophytes
formation
Clinical features
Hx of acute disc ruptured -> recurrent attacks of pain
over several years
Intermittent backache
Hard physical work, standing or walking a lot or sitting in
one position during a long journey
Relieve by laying down
X-rays
Radiographic
features of
intervertebral disc
degeneration
Flattening of disc
space
Marginal bony spur
(osteophytes)
OA of facet joints
Management
Asymptomatic dont need treatment
Conservative treatment
Modified activities
Exercise aerobic, stretching and muscle
strengthening
Wearing of a lumbar corset
Small doses of anti-inflammatory drug
Spinal Metastasis
If > 50 y.o., think bone mets > primary
malignancy of bone
Commonest sources : Ca of breast, prostate,
kidney, lung, thyroid, bladder and GIT.
Commonest site : vertebrae, pelvis, the
proximal half of the femur and the humerus.
Spread : hematogenous or direct
Metastases are usually osteoclastic and
pathological fractures are common.
Osteoblastic tumours are uncommon,
usually occur in prostatic carcinoma.
Clinical Features
Blastic =
abnormal
white area
Bone Scan
CT
Determining integrity of vertebral
column (if surgery indicated)
Management
Curative - Radical treatment (surgery +
radiotherapy) for both primary and secondary tumor
if possible
Prognosis for survival = almost hopeless
Focuses on relieving and preventing the suffering of
patients
Pain management- analgesia or radiotherapy
Fix fractures intramedullary nailing
Prophylactic fixation
Spinal stabilisation- fitted braces or operative stabilization
Hypercalcaemia hydration, reduce Ca intake,
biphosphanates
Cervical spondylosis
Cervical myelopathy
Defini
tion
Clinic
al
featur
es
-aged > 40
-neck pain and stiffness
-Gradual onset and worse
after waking up.
-Radiates to occiput, back of
shoulders or one or both
upper limbs.
-numbness, weakness and
clumsiness of hands
X ray
-Narrowing of one or more
Findin intervertebral disc.
gs
-Bony spur formation at the
anterior or posterior of disc.
-Osteophytes encroaching on
the intervertebral foramina.
Treat
-analgesics, collar,
OSTEOPOROSIS
-Abnormally LOW BONE MASS
-imbalance between bone resorption and
bone formation activity
-NORMAL mineralization
-high risk for FRACTURE!
Generalised
Primary/age
related
postmenouposa
l osteoporosis
>acute back
pain d/ t
vertebral
compression
Repeated minor
fractureprogressive
kyphosis
Localised
-due to
disuse/nearby
inflammation
Secondary
1-nutritional-scurvy
2-endocrine
disorders- high PTH,
cushings
3-malignancymultiple myeloma,
leukemia
4-non-malignantRA, AS, TB, CKD
5-ALCOHOL ABUSE!
DIAGNOSIS
Primary-Usually
obvious-need to
exclude secondary
causes
Dexa scan T score
<-2.5
X-ray