Trauma
EMG Team
Introduction
Trauma is the leading cause of death < 40
y.o.
Abdominal trauma is responsible for 10%.
The findings to look for in abdominal trauma
are the following:
Hemoperitoneum
Contrast blush consistent with active
extravasation
Laceration: Linear shaped hypodense
areas
Hematomas: oval or round shaped areas
Contusions: vague ill-defined hypodense
areas that are less well perfused
Pneumoperitoneum
Devascularization of organs or parts of
organs
Subcapsular hematomas
Introduction
Sign of shock in CT-Scan :
Collapse of inferior vena cava
Small aorta
Persistent nephrogram without excretion
Hypodense spleen, without enhancement and
normal vascular pedicle
Increased enhancement of the small bowel wall
Increased enhancement of the adrenal glands
Sometimes findings of right cardiac
insufficiency with reflux into the hepatic veins
Introduction
Systematic
approach :
1.Crania caudal
2.Superficial deep
3.One organ other
organ in one plane
Introduction
Initial evaluation :
Blunt trauma
Penetrating trauma
Trauma Protocol
Blunt injury
Simple protocol
The portal venous phase ~ 70 secand
Delayed scan ~ 3-5 minutes later injury is
detected on the initial scan.
No oral contrast is administered.
Penetrating injury
Same as blunt injury
risk injury : flank great risk for bowel perforation.
No reason for immediate surgery (initial scan)
additional scan after the administration of rectal contrast
(50 ml contrast in 1000 ml saline).
Introduction
Hemoperitoneum
Hyperdense intraperitoneal fluid collection
020HU
Preexisting ascites
Bile
Urine
Digestive fluid
Diluted or old blood
3045HU
Free Unclotted
intraperitoneal blood
4570HU
>100 HU
Extravasation of contrast
medium
(vascular or urinary)
Spleen
Spleen most
commonly injured
(25%)
AAST grading limited
value not predict the
succes rate of NOM
Contrast extravasation
(not part of grading)
active bleeding
failure of NOM (80%)
Grade
IV
Grade
V
Parenchymal Contus
Hypodense
intraparenchymal area
Parenchymal Laceration
Superficial, linear
hypodensity, usually
less than 3 cm in
length
Fracture - involves
two visceral surfaces,
or if its length is
more than 3 cm
Multiple fractures Scattered spleen
Subcapsular
Hematoma
Crescent-shaped perisplenic
Compresses the splenic parenchym
Spleen
The shortecommings of
this grading scale are :
Often underestimates
injury extent
Significant
interobserver variability
Does not include :
Active bleeding
Contusion
Post traumatic infarcts
Liver
Second most common
solid organ injury
Most common death
more vessels
Posterior segment of
right liver lobe
frequently injured
retroperitoneal
bleeding
Liver
The findings are :
1.Green arrow : oval
shaped hypodense
hematoma
2.Yellow arrow : linear
shaped hypodense
laceration
3.Blue arrow : vague ill
defined hypodens
contusion
4.Fluid around the liver
5.Transection of the
liver
Liver
Shown to be
unreliable in
predicting need for
surgery
Helpful in guiding
management
Positive correlation
between grade of
injury vs likelihood
of failed NOM
Classification
(AAST)
I-Subcapsular hematoma<1cm,
superficial laceration<1cm deep.
IV-Parenchymal/supcapsular
hematoma> 10cm in diameter,
lobar destruction,
V- Global destruction or
devascularization of the liver.
Liver
The findings are
the following :
1.Complete
devascularization
of the right lobe
2.Contrast blush
intraparenchymal
at lateral margin
and lower level
3.Hemoperitoneum
Liver
The findings are the
following :
1.Subcapsular
hematoma > 10 cm
2.Contrast blush
3.No hemoperitoneum
Lacerations can be :
Stellate
Branching
Renal
90% due to blunt trauma
Renal is the 3rd most common organ
involved in blunt trauma
10% of solid visceral injury
Evaluation for :
Parenchymal injury
Vascular injury
Collecting system injury
Computed Tomography
Early and delayed CT scans through the
kidneys are necessary
Excretory-phase contrast (3min)
The preferred technique
Helical CT performed from the dome of the
diaphragm
Subcapsular hematoma
(category I)
Laceration
Hypodense, irregularly linear areas,
typically distributed along the vessels
and filled with blood.
They are best analyzed at arterial
phase
Superficial (<1 cm from the renal cortex)
Deep (>1 cm from the renal cortex)
Renal medulla
Collecting tubule system
Segmental Infarct
Triangular parenchymal area, with a
widest part at the cortex, which is
not enhanced during the different
phases, with clear delineation
Traumatic
occlusion of the
main renal
artery
(category III)
Urinoma/Urohematoma
Presence of a more or less significant
breach of the collecting tube system,
with urine escape reflected by
extravasation of contrast medium on
delayed imaging, in an extrarenal
location
Renal
The findings are the
following :
1.Extravasation and fluid
paracolic gutter
peritoneal violation
2.Hematoma perirenal
space
3.Violation of the
collecting system
Renal
Some final remarks on
renal injury :
CT has facilitated shift
toward NOM
98% renal injuries
NOM
Injury delayed
imaging evaluate
collecting system
Penetrating trauma
rectal contrast
bowel injury
Pancreas
Uncommon injury
0.4% overall
incidens
1.1% in
penetrating trauma
and 0.2% in blunt
trauma
Rarely an isolated
injury
Type of
Injury
Description of Injury
Hematoma
Laceration
Hematoma
Laceration
III
Laceration
IV
Laceration
Laceration
II
Pancreas
Typical left sided package
injury
Pancreatic tail injury
Renal Injury
Pneumoperitoneum
Pancreas
Right sided
package
injury
Liver
laceration
Transection
of the
pancreas
Bowel
The findings are the following :
1.Hypoperfusion of the spleen
(yellow arrow)
2.Multiple area of contrast
extravasation (green arrow)
3.Hemoperitoneum
4.Pneumoperitoeum (blue arrow)
Bowel
Pneumoperitoneum uncommon
findings if bowel injury present
Known false positive :
Peritoneal Lavage
Foley insertion intraperitoneal bladder
rupture
Translocation from thorax diaphragm
injury
Small Bowell
Most common findings :
Unexplained non-physiologic free air fluid (84%)
Diffuse wall thickening :
Hypoperfused shock bowell active bleeding
Focal thickening :
Direct injury usualy non-transmural injury
Spesific findings :
Oral contrast/bowel content extravasation
Focal bowel wall discontinuity
Other findings :
Mesenteric stranding
If in combination,
Focal bowel thickening
very suggestive
for bowell injury
Interloop fluid
Periduodenal hemorrhage
Bladder
If # pelvic bladder
rupture only 10%
If bladder rupture
almost always #
pelvic
1/3 rupture at site
of # due to pelvic
#
2/3 rupture
opposite site
shearing force
Bladder
The findings are the
following :
1.Contrast in the
bladder surrounding
foley catheter
2.Extravasation of
contrast in pre-vesicle
space
Molar Tooth Sign
extraperitoneal bladder
rupture
Bladder
Have good
distention of the
bladder
Do not administer
contrast in the
bladder and I.V
contrast at the
same time
bladder rupture or
active bleeding
Thank You