Failure
Dr. Muhammad Fadil, SpJP
Department of Cardiology and Vascular Medicine
Medicine Faculty of Universitas Andalas/ Dr. M. Djamil Hospital
Padang
th
4 SymCARD 2014
Introduction
In heart failure patient population, cardiac arrhythmias
frequently contribute to worsened symptoms, periodic
decompensations, and increased mortality
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0.4
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The incidence of atrial fibrillation in recent heart failure and arrhythmia trials
Thomas SA, et al. AACN Clin Iss 2001; 12(1):156163.
Mechanism of AF in HF
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McMurray JJV, et al. European Heart Journal (2012) 33, 17871847 4 SymCARD 2014
Management
1.Rate Controlled
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McMurray JJV, et al. European Heart Journal (2012) 33, 17871847 4 SymCARD 2014
th
McMurray JJV, et al. European Heart Journal (2012) 33, 17871847 4 SymCARD 2014
Extreme case
AV node ablation and pacing may be required
CRT may be considered instead of conventional pacing
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McMurray JJV, et al. European Heart Journal (2012) 33, 17871847 4 SymCARD 2014
th
McMurray JJV, et al. European Heart Journal (2012) 33, 17871847 4 SymCARD 2014
th
McMurray JJV, et al. European Heart Journal (2012) 33, 17871847 4 SymCARD 2014
Management
2.Rhythm Controlled
In patients with Chronic HF, a rhythm-control strategy has not been demonstrated to be
superior to a rate-control strategy in reducing mortality or morbidity
In patient with Acute HF with haemodynamic instability emergency cardioversion
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4 SymCARD 2014
Treatment
Most patients with systolic HF will have a risk score consistent with a firm
indication for (score2) or preference for an oral anticoagulant (score=1)
although
bleeding risk must also be considered
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McMurray JJV, et al. European Heart Journal (2012) 33, 17871847 4 SymCARD 2014
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4 SymCARD 2014
Ventricular Arrhytmias and Sudden
Cardiac Death
Sudden cardiac death : 20% to 50% of the mortality in HF
Tedrow U and Stevenson WG. Management of Atrial and Ventricular Arrhythmias in Heart Failure. Marcel Dekker New York. 2005
Monomorphic Ventricular Tachycardia
Ischemic
Mechanisme of VT
Patients with Ischemic Cardiomyopathy typically have
large areas of infarction. Surviving myocyte bundles
present within the infarction create channels for
conduction set up reentry circuits VT
Mechanisme of VT
Patients with non Ischemic Cardiomyopathy who develop
sustained monomorphic VT, most have evidence of large areas
of ventricular scar associated with a reentry circuit
Mechanisme of VT
Electrophysiological changes that accompany ventricular hypertrophy in
chronic heart failure may increase susceptibility to torsades de pointes