faculty of nursing
maternal and child health department
pediatric health coarse (clinical 301348).
Case presentation about meningitis .
Prepared by: heba al-khozae.
Student no. : 200711008 .
Clinical area : al ـbasheer hospital .
Group no.: 1.
Dr. nehad qasem.
cl. inst. : miss. Amneh.
Child’s name:ي نا لف ايز محمد علوش
Sex: male.
Age: 2, 5 months. Date of birth: 14\2\2010.
Body weight: 4, 1 kg.
Height: 56 cm.
Head circumference: 40 cm.
Open and mild plugging anterior fontanel.
Temp.: 38, 5 c.
Resp.: 55 breath\min.
Pulse: 160 beat\min.
Condition on arrival: holding by his father.
Date of admission: 21\4\2010. Via: emergency.
Floor: medical. Room no.: 8.
Source of data: child’s parent’s, patient file and medical health
team.
Confirmed medical diagnosis: bacterial meningitis.
Complains upon admission: fever, hypo activity, tachycardia
and poor feeding from 5 days.
Past medical and surgical history: history of neonatal jaundice
treated at home , upper respiratory infection and no any past
surgical history.
Perinatal history:
Gestational period: 39 weeks.
Type of delivery: c\s due to previous c\s.
Birth weight: 3,2 kg.
Without any complications immediately after delivery , and not
admitted to NICU .
Immunization status: BCG only.
No any family history of hereditary or chronic diseases , and no any
history of abortion , stillbirth , or congenital anomalies.
Nutrition and metabolic pattern:
Loss of 1 kg, anorexia, breast feeding 2-3 times\day , low amounts.
Other body systems appear normal without any complications.
30
28
years
years
A,W A,W
2,5 3
month years
s
Meningitis A,W
Bacterial meningitis is a life-threatening illness that results
from bacterial infection of the meninges. Beyond the
neonatal period, the 3 most common organisms that cause
acute bacterial meningitis are Streptococcus pneumoniae,
Neisseria meningitidis, and Haemophilus influenzae type b
(Hib).
Inflammation of the meninges and ventricles produces a
polymorphonuclear response, an increase in cerebrospinal fluid
(CSF) protein content, and utilization of glucose in CSF.
Inflammatory changes and tissue destruction in the form of
empyema and abscesses are more pronounced in gram-negative
meningitis. Thick inflammatory exudate causes blockage of the
aqueduct of Sylvius and other CSF pathways, resulting in both
obstructive and communicating hydrocephalus.
According to text book: in my patient:
Poor feeding Poor feeding
Lethargy Irritability
Irritability Fever
Apnea hypoactive