2017 Psikopatologi RBM
2017 Psikopatologi RBM
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Gangguan Kesadaran
– somnolen
– sopor
– koma
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Gangguan Kesadaran
Gangguan Kesadaran Kualitatif: kondisi individu yang
mengalami gangguan pada persepsi, pikiran, mood/afek,
ingatan dan motoriknya:
– Delirium – Karakteristik: disorientasi, gangguan
persepsi, mudah disugesti, mudah mengalami
misinterpretasi dan gangguan mood
– obnubilation (twilight state) – mulai dan berakhir tiba-
tiba, amnesia; pasien kacau, perilakunya samasekali tak
bertujuan, kadang-kadang agresif, sulit dimengerti
stuporous
vigilambulant
delirious
Ganser syndromemulyowarnonagoro@msn.com
Gangguan Persepsi
Persepsi: proses memahami sensasi yang diterima oleh
panca indera
Image: suatu pengalaman dalam ingatan, tanpa sensasi
nyata walaupun itu bagian dari realitas
Pseudoilusi – gangguan persepsi terhadap objek yang
sebetulnya tampak samar
Ilusi – adalah fenomena psikopatologis;
terjadi terutama pada kondisi gangguan kesadaran yang
secara kualitatif berubah
Halusinasi – adalah persi tanpa adanya stimulus
apapun terhadap panca indera;
pasien tak dapat membedakannya
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dari realitas
Gangguan Persepsi
Halusinasi:
auditorik (akustik) – pendengaran
Visual – penglihatan
Olfaktorik – penciuman
Gustatorik – Pengecapan
taktil – perabaan (sensasi lebih dalam)
Intrapsikik (lebih ke gangguan pikiran)
hypnagogic and hypnopompic (hypnexagogic)
Fungsi-fungsi Kognitif
Gangguan pikiran:
– kuantitatif
– kualitatif
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Gangguan Pikiran Kuantitatif
Blocking - berhenti
Perseverasi
Asosiasi longgar
Neologisme
Verbigerasi
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Gangguan Pikiran Kualitatif
Gangguan pikiran secara kualitatif (gangguan isi pikir):
Delusi (waham):
a) keyakinan yang tidak mempunyai dasar adekuat,
b) tak dapat dipatahkan oeh argumentasi yang rasional
c) bukan suatu keyakinan konvensional
Obsesi (pikiran obsesif) pikiran tertentu yang berulang
muncul secara menetap, impuls atau bayangan yang
memasuki ingatan meskipun individu berusaha
mencegahnya. Fenomena obsesif yang diikuti dengan
tindakan (biasanya seperti ritual – membersihkan,
menghitung, menata) disebut kompulsi.
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Gangguan Pikiran Kualitatif
Divisi delusi/waham:
Berdasar onset
a) primer (waham terkait mood, persepsi)
b) sekunder (sistematik)
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Gangguan Pikiran Kualitatif
Delusi/waham:
Berdasar tema
a) paranoid (persecutory) - d. of reference, w. Cemburu, w.
dikendalikan, w. terkait pikiran yang diambil
b) megalomanik (grandiose, expansive) – w. tentang
kemampuannya, w. keistimewaan dirinya, w. kekuatannya,
w. asmara, w. kekuatan supranatural.........
c) depresif (mikromanik, melankolik) – w. bersalah, w. nihilistik
w. ketidakmampuan, w. hipokhondriakal.......
d) terkait kepemilikan pikiran
Sisip pikir (thought insertion)
Pikiran tersedot (thought withdrawal)
Siar pikir (thought broadcasting)
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Gangguan Daya Ingat
Penyimpanan Sensorik – menyimpan
informasi sensorik selama 0.5 detik
Ingatan jangka pendek – untuk informasi
verbal dan visual disimpan selama 15-20 detik,
kapasitas rendah
Ingatan jangka panjang – kapasitasnya lebih
luas dengan penyimpanan lebih permanen
– Ingatan deklaratif (explicit) – episodik (u peristiwa)
atau semantik (u bahasa dan pengetahuan)
– Ingatan prosedural – u motorik berirama
– Priming – ingatan bawah sadar
– Pembiasaan - conditioning – Klasik atau emosional
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Gangguan Daya Ingat
Gangguan daya ingat:
Amnesia – ketidakmampuan mengingat peristiwa
yang lalu
Jamais vu, déja vu
Confabulation, amnesic disorientation, Korsakov’s
syndrome
Pseudologia phantastica
Hypomnesia
Hypermnesia
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Gangguan Perhatian
Konsentrasi
Kapasitas
Tenasitas
Irritabilitas
Vigilitas
Gangguan intelek:
– retardasi mental
– demensi
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Gangguan Motorik
Gangguan Motorik sering terjadi pada GJ terutama
skizofrenia katatonik.
kuantitatif: kualitatif:
– mannerism
– hypoaktif – stereotipik
– hyperaktif – posturing
– perilaku agitatif – flexibilitas cerea
– ekhopraxia
– impuls skhizofrenik
– negativism
– short-circuit behaviour
– automatism
– agitation
– tics
– abulia
– compulsions
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Disorders of Volition
Disorders of volition:
– hypobulia
– abulia
– hyperbulia
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Disorders of Personality
Personality means a complex of persistent
mental and physical traits of a person
Disturbances of personality:
– transformation of personality
– appersonalization
– multiple personality (alteration of personality)
– specific personality disorder
– deprived personality
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Multistable perception
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Multistable perception
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Multistable perception
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Multistable perception
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Multistable perception
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Multistable perception
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Multistable perception
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Mental construction
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Mental construction
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Instruction to
manufacturing (straight
from the hell …)
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Schizophrenia
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Symptoms
Disorganized Thinking/Speech
Negative Symptoms – the absence of
normal behavior
Catatonia – immobility and “waxy
flexibility”
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Types of Schizophrenia
Disorganized Schizophrenia
Catatonic Schizophrenia
Paranoid Schizophrenia
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Brain and Schizophrenia
Larger than normal lateral ventricles – part of
the ventricular system; contain cerebrospinal
fluid
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Causes of Schizophrenia:
Genetics
Twin studies have shown that tendency for
both monozygotic twins to develop
schizophrenia is between 30 to 50%; the
tendency for dizygotic twins and siblings to
develop schizophrenia is 15%
Adoption Studies – 13% of biological
relatives of adoptees with schizophrenia had
the disease, but only 2% of biological
relatives of normal adoptees had
schizophrenia mulyowarnonagoro@msn.com
Causes of Schizophrenia:
Environment
Family Stress
Poor social interactions
Infections or viruses at an early age
Trauma at an early age
Genetic makeup combines with non-
genetic factors to cause schizophrenia
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Causes of Schizophrenia:
Neurotransmitters
Dopamine Theory – schizophrenia
caused by an overactive dopamine
system in the brain; dopamine is a
neurotransmitter that allows nerve
cells in the brain to send messages to
each other; dopamine imbalance may
affect the way a person’s brain can
react to stimuli
Many drugs for schizophrenia block
dopamine receptors
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Schizophrenia:
Medication
Antipsychotic medications – drugs do
not cure disease, but reduce
symptoms
Some drugs have Parkinson’s Disease-
like side effects
Counseling also helps
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Bipolar Disorder
Psychosis
Delusions
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Bipolar Disorder and the
Brain
Examination of brain tissue after people with BD have died
Brain imaging
New theory – mitochondrial malfunction
Mitochondria – vital organelle for energy production
2000 – Kato, University of Tokyo – based on abnormal brain
energy metabolism in bipolar patients measured by brain
scans and mitochondrial depletion in autopsied bipolar
brains; searched for mutant mitochondrial DNA – two suspect
genes
March 2004, Archives of General Psychiatry, Christine Konradi
- Harvard and McLean Hospital - studied brain tissue in
hippocampus and found expression of genes that coded for
mitochondrial proteins in bipolar patients decreased
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Famous People with BD
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Causes
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Case Study: Eve
http://www.mcmanweb.com
http://www.crimelibrary.com/criminal_
mind/psychology/multiples
http://faculty.washington.edu/chudler/
neurok.html
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