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Oligohydramnios is an abnormally decreased amount of amniotic fluid, complicating 1-2% of pregnancies. It is diagnosed sonographically by an amniotic fluid index (AFI) of ≤5 cm or deepest pocket ≤2 cm. Early onset is often due to fetal abnormalities or placental issues, impairing urine production. Later onset is usually due to placental dysfunction restricting fetal growth and urine output. Exposure to certain medications like ACE inhibitors and NSAIDs during the second or third trimester can also cause oligohydramnios. Decreased amniotic fluid before 22 weeks increases the risk of pulmonary hypoplasia. Management involves evaluating for fetal anomalies and growth complications
Oligohydramnios is an abnormally decreased amount of amniotic fluid, complicating 1-2% of pregnancies. It is diagnosed sonographically by an amniotic fluid index (AFI) of ≤5 cm or deepest pocket ≤2 cm. Early onset is often due to fetal abnormalities or placental issues, impairing urine production. Later onset is usually due to placental dysfunction restricting fetal growth and urine output. Exposure to certain medications like ACE inhibitors and NSAIDs during the second or third trimester can also cause oligohydramnios. Decreased amniotic fluid before 22 weeks increases the risk of pulmonary hypoplasia. Management involves evaluating for fetal anomalies and growth complications
Oligohydramnios is an abnormally decreased amount of amniotic fluid, complicating 1-2% of pregnancies. It is diagnosed sonographically by an amniotic fluid index (AFI) of ≤5 cm or deepest pocket ≤2 cm. Early onset is often due to fetal abnormalities or placental issues, impairing urine production. Later onset is usually due to placental dysfunction restricting fetal growth and urine output. Exposure to certain medications like ACE inhibitors and NSAIDs during the second or third trimester can also cause oligohydramnios. Decreased amniotic fluid before 22 weeks increases the risk of pulmonary hypoplasia. Management involves evaluating for fetal anomalies and growth complications
406172079 • This is an abnormally decreased amount of amnionic fluid • complicates approximately 1 to 2 percent of pregnancies • The sonographic diagnosis of oligohydramnios is usually based on an AFI ≤ 5 cm or on a single deepest pocket of amnionic fluid ≤ 2 cm (ACOG 2012) • AFI below the 5th or 2.5th percentile determined by a gestational-age-specific nomogram. Etiology Early onset • When amnionic fluid volume is abnormally decreased from the early second trimester fetal abnormality that precludes normal urination or • placental abnormality severe enough to impair perfusion After midpregnancy • amnionic fluid volume becomes abnormally decreased in the late second or in the third trimester, it more likely is associated with fetal-growth restriction, placental abnormality, • underlying etiology is often presumed to be uteroplacental insufficiency, which can impair fetal growth and reduce fetal urine output • Oligohydramnios is commonly encountered in postterm pregnancies, amnionic fluid volume decreased by approximately 8 percent per week beyond 40 week Medication • exposure to drugs that block the renin-angiotensin system include angiotensin converting enzyme (ACE) inhibitors and nonsteroidal antiinflammatory drugs (NSAIDs) • When taken in the 2nd or 3rd trimester, ACE inhibitors and angiotensin-receptor blockers fetal hypotension, renal hypoperfusion, renal ischemia • NSAIDs have been associated with fetal ductus arteriosus constriction and with decreased fetal urine production. In neonates, their use may result in acute and chronic renal insufficiency Pregnancy outcomes Pulmonary hypoplasia • When decreased amnionic fluid is first identified before the mid- second trimester, particularly before 20 to 22 weeks, pulmonary hypoplasia is a significant concern • Underlying etiology is a major factor in the prognosis for such pregnancies. Severe oligo- hydramnios secondary to a renal abnormality generally has a lethal prognosis Management • Initially, an evaluation for fetal anomalies and growth is essential. • Evidence for fetal or maternal compromise will override potential complications from preterm delivery. But, oligo- hydramnios detected before 36 weeks in the presence of normal fetal anatomy and growth may be managed expectantly