Nama : Ny. R A
Usia : 38 thn
Status Pernikahan : Menikah
Pekerjaan : Ibu Rumah Tangga
Agama : Islam
Alamat : JL. Angke Jaya 1 RT 7001/006 No. 21
Anamesa
Dilakukan pada tanggal 11 Mei 2018 pk 10.37 secara autoanamesa
Keluhan utama :
Kontrol Hamil, dirujuk dari puskesmas Angke untuk USG
Riwayat Penyakit Sekarang
G1P0A0 H.35-36 minggu dirujuk dari puskesmas untuk periksa USG
HPHT pasien 6 September 2017, Hari Perkiraan Lahir 13 Juni 2018
TP-USG pada usia kehamilan 4 bulan dikatakan pasien adalah 12 Juni 2018
Gerak janin sekarang dikatakan aktif
Pasien mengatakan ada bengkak pada kedua tangan dan kaki sejak sekitar 3 hari
yang lalu
Sakit kepala, pusing, gangguan penglihatan, nyeri pada abdomen kanan atas
disangkal
Pasien menikah 1x, sudah 14 tahun dengan suami sekarang
Rutin melakukan pemeriksaan tiap bulannya di puskesmas
Tidak ada perdarahan, ataupun mual dan muntah yang berlebihan.
Nafsu makan dikatakan baik, tidak ada demam, batuk, ataupun pilek.
Riwayat Penyakit Dahulu
Riwayat Hipertensi sebelumnya disangkal oleh pasien
Riwayat DM, TBC, Asma, Penyakit Jantung, Alergi (-)
Riwayat Penyakit Keluarga
Riwayat hipertensi, DM (+)
Riwayat keluhan serupa disangkal oleh pasien
Riwayat alergi, TBC, asma, penyakit jantung (-)
Riwayat Obstetri dan Ginekologi
Haid dikatakan teratur, setiap 28 hari
G1 P1 A0 Riwayat KB suntik dan pil
Pasien menikah 1 kali, dengan suami selama total 5 tahun
sekarang sudah 14 tahun
HPHT 6 September 2017
HPL 13 Juni 2018
No. Tahu Tempat Umur Jenis Penolong Penyuli Anak
n Kehamilan t
JK BB Keadaan sekarang
1 Hamil ini
Riwayat Kebiasaan & Asupan Nutrisi
HIV NR
HbsAg -
Urinalisa
Janin :
Janin Tunggal Hidup Presentasi Kepala dengan TBJ 2362
gram
Rencana Diagnostic
Lab darah rutin + BT + CT + Golongan Darah
USG
Urinalisa (cek kuantitatif produksi protein urin per 24 jam atau cek
protein urin)
Lab Kimia Darah (SGPT/SGOT)
Cek Serum Kreatinin
Rencana terapi farmakologis
-
Terapi Non Farmakologis Rencana Evaluasi
- Evaluasi tekanan darah di
puskesmas (Jaga tekanan darah
<160/110 mmHg)
Datang kembali minggu depan
untuk kontrol kehamilan
Edukasi
Menjelaskan kepada pasien tentang apa itu hipertensi dalam
kehamilan
Menjelaskan kepada pasien apa saja tanda-tanda dari preeklamsia
Menjelaskan kepada pasien dampak dan bahaya dari Hipertensi dalam
kehamilan kepada ibu dan bayinya
Menjelaskan kepada pasien untuk kontrol tekanan darahnya di
puskesmas dan datang kembali minggu depan untuk evaluasi ulang
kondisi serta kontrol kehamilan
Prognosis
Ad vitam – dubia ad Bonam
Ad sanationam – Bonam
Ad functionam – Bonam
Hypertension in pregnancy
Hypertension in pregnancy
Is among the most significant and intriguing unsolved problems in
obstetrics
Complicate 5-10% of all preganancies
PE either alone or superimposed on chronic HTN -> most dangerous
PE is identified in 3.9% of all pregnancies
16% maternal death were reported to be due to HTN disorders
hemorrhage—13%, abortion—8%, and sepsis—2% = HTN is higher
Classification
4 types :
Gestational hypertension—evidence for the preeclampsia syndrome does
not develop and hypertension resolves by 12 weeks postpartum
Preeclampsia and eclampsia syndrome
Chronic hypertension of any etiology
Preeclampsia superimposed on chronic hypertension
Diagnosis of Hypertensive Disorders
Hypertension is diagnosed empirically when appropriately taken
blood pressure exceeds 140 mm Hg systolic or 90 mm Hg diastolic.
women who have a rise in pressure of 30 mm Hg systolic or 15 mm
Hg diastolic should be observed more closely because eclamptic
seizures develop in some of these women whose blood pressures have
stayed < 140/90 mm Hg (Alexander, 2006)
Gestational Hypertension
women whose blood pressures >= 140/90 mm Hg for the first time
after midpregnancy, but in whom proteinuria is not identified
Almost ½ -> develop Preeclampsia syndrome (findings = headaches
or epigastric pain, proteinuria, and thrombocytopenia)
reclassified by some as transient hypertension if evidence for
preeclampsia does not develop and the blood pressure returns to
normal by 12 weeks postpartum
Preeclampsia syndrome
Best described as = a pregnancy-specific syndrome that can affect virtually every organ
system
appearance of proteinuria remains an important diagnostic criterion
-> ? -> proteinuria = an objective marker and reflects the system-wide endothelial leak,
which characterizes the preeclampsia syndrome
Abnormal protein excretion = defined by 24-hour urinary excretion exceeding 300 mg; a
urine protein:creatinine>= 0.3; or persistent 30 mg/dL (1+ dipstick) protein in random
urine samples (Lindheimer, 2008a).
urine concentrations vary widely during the day, as do dipstick readings
assessment may show a dipstick value of 1+ to 2+ from concentrated urine specimens
from women who excrete <300 mg/day
overt proteinuria may not be a feature in some women with the preeclampsia syndrome
(Sibai, 2009)
Headaches or visual disturbances such as scotomata can be premonitory
symptoms of eclampsia
Epigastric or right upper quadrant pain frequently accompanies hepatocellular
necrosis, ischemia, and edema that ostensibly stretches Glisson capsule ->
frequently accompanied by elevated serum hepatic transaminase levels
thrombocytopenia is also characteristic of worsening preeclampsia as it
signifies platelet activation and aggregation as well as microangiopathic
hemolysis
renal or cardiac involvement and obvious fetal-growth restriction, which also
attests to its duration
Eclampsia
Preeclampsia + convulsion that can not be attributed to another cause
-> eclampsia
Convulsions are generalized, may appear before, during, or after labor
10% (Sibai, 2005), or 25% (Chames, 2002) -> x develop seizure until
after 48 hrs postpartum
Preeclampsia Superimposed on Chronic Hypertension
Hospitalisation
for women
with new
onset HT
Elective Cesarean Delivery
labor induction and vaginal delivery have traditionally been
considered ideal
Several concerns, including an unfavorable cervix, a perceived sense
of urgency because of preeclampsia severity, and a need to coordinate
neonatal intensive care, have led some to advocate cesarean delivery
Whenever it appears induction almost certainly will not succeed or
attempts have failed, then cesarean delivery is indicated
Hospitalization vs Outpatient Management
mild to moderate stable hypertension— whether or not preeclampsia
has been confirmed—surveillance is continued in the hospital, at
home for some reliable patients, or in a day-care unit
Bed rest was associated with a significantly reduced relative risk—
RR 0.27—of developing preeclampsia
complete bed rest is not recommended by the 2013 Task Force
AntiHypertrensive Therapy
Future Pregnancies
Hypertensive disorders may serve as markers for subsequent preterm labor
and fetal-growth restriction
even in subsequent nonhypertensive pregnancies, women who had
preterm preeclampsia are at increased risk for preterm birth
Sibai and colleagues (1986, 1991) found that nulliparas diagnosed with
preeclampsia before 30 weeks have a recurrence risk as high as 40 percent
during a subsequent pregnancy
In a prospective study of 500 women previously delivered for
preeclampsia at 37 weeks, the recurrence rate in a subsequent gestation
was 23 percent (Bramham, 2011).
Future Pregnancies
Women whose first pregnancy was complicated by preeclampsia
between 32 and 36 weeks had a significant twofold increased
incidence of preeclampsia in their second pregnancy’
preterm delivery and fetal-growth restriction in the first pregnancy
significantly increased the risk for preeclampsia in the second
pregnancy
Long Term Consequences