JURNAL READING

Contact-lens Induced Endofthalmitis. A Case Report

Disusun Oleh :
Muhammad Taufiqurrachman (21904101057)

Dosen Pembimbing Klinik :

dr. Agustin Wijayanti, Sp.M

KEPANITERAAN KLINIK MADYA

LABORATORIUM ILMU KESEHATAN MATA
RSUD MARDI WALUYO KOTA BLITAR
FAKULTAS KEDOKTERAN UNIVERSITAS ISLAM MALANG
2021
 KATA PENGANTAR

Assalamu’alaikum warahmatullahi wabarakatuh,

Puji syukur kami panjatkan kehadirat Allah SWT yang telah melimpahkan rahmat,
taufik, dan hidayah-Nya, sholawat serta salam yang kami junjungkan kepada Nabi
Muhammad SAW yang telah menuntun kita menuju jalan kebenaran sehingga dalam
penyelesaian tugas ini kami dapat memilah antara yang baik dan buruk. Kami mengucapkan
terima kasih kepada dosen pembimbing pada Laboratorium Ilmu Mata, yaitu dr. Agustin
Wijayanti, Sp.M yang memberikan bimbingan dalam menempuh pendidikan ini. Tak lupa
pula kami mengucapkan terima kasih kepada semua pihak sehingga dalam penyusunan
laporan kasus ini dapat terselesaikan.

Kami menyadari dalam ini belum sempurna secara keseluruhan oleh karena itu kami
dengan tangan terbuka menerima masukan-masukan yang membangun sehingga dapat
membantu dalam penyempurnaan dan pengembangan penyelesaian selanjutnya.

Demikian pengantar kami, semoga makalahini dapat bermanfaat bagi semua. Amin.

Wassalamu’alaikum warahmatullahi wabarakatuh,

Malang, 12 Juli 2021

Penyusun
CASE REPORT A Rare Case of Bilateral Optic Neuritis and Guillain-Barré Syndrome Post Mycoplasma
pneumoniae Infection Aravindhan Baheerathana , Amy Ross Russella , Fion Bremnerb , and Simon F.
Farmera a Department of Neurology, National Hospital for Neurology and Neurosurgery, London,
United Kingdom; b Department of Neuroophthalmology, National Hospital for Neurology and
Neurosurgery, London, United Kingdom ABSTRACT Neurological complications are the most
commonly encountered extra-pulmonary manifestation of infection with Mycoplasma pneumoniae
(M. pneumoniae). Here the authors report the case of a 39-yearold woman who was admitted with
acute-onset bilateral visual loss coinciding with ascending numbness. Clinical examination,
neurological imaging, and nerve conduction studies revealed a syndrome of bilateral optic neuritis
and Guillain-Barré syndrome (GBS). Serological testing confirmed recent exposure to M.
pneumoniae. The patient did not experience any clinical benefit with pulsed intravenous
methylprednisolone but demonstrated marked clinical and radiological improvement following 5
days of plasma exchange. This report will explore the diagnostic and therapeutic approach to
patients with neuro-ophthalmological and neurological complications of M. pneumoniae infection in
addition to discussing previously encountered cases. ARTICLE HISTORY Received 15 June 2016
Revised 13 September 2016 Accepted 14 September 2016 KEYWORDS Mycoplasma pneumoniae;
post-infectious inflammatory neuropathy; post-infectious optic neuropathy; plasma exchange; optic
neuritis; Guillain-Barré syndrome Case report A 39-year-old Afro-Caribbean woman was admitted to
our neurological department from the local ophthalmic hospital casualty department, having
presented with acute-onset bilateral visual loss. Fifteen days prior to presentation, she had
experienced a dry cough with flu-like symptoms, predominantly myalgia, in association with a dull
headache. The patient had been prescribed 5 days of amoxicillin in primary care. These symptoms
had resolved, but were followed 5 days later by bilateral eye pain, pain on eye movements, and a
reduction of vision in each eye over the subsequent 5 days. She also described pins and needles in
her fingers and toes, gradually progressing over the same time course. Upon initial assessment she
reported no perception of light in either eye. Pupillary reflexes were absent. She had bilateral
swelling of the optic discs with splinter haemorrhages (Figure 1a). There was bilateral globe
tenderness. The remainder of the cranial nerve examination was normal. Examination of the arms
demonstrated normal tone and full power. She had reduced light touch and pin prick to the level of
the wrists. Reflexes were present and symmetrical. Examination of the legs showed normal tone and
full power. Light touch and pin prick sensation was reduced to the ankles. Reflexes were present on
day 1, but ankle-knee, supinator, and bicep reflexes had become absent by day 4. Plantar reflexes
were flexor bilaterally. Magnetic resonance imaging (MRI) of the brain and orbits demonstrated
enlargement of both optic nerves with gadolinium enhancement (Figure 2a). The reminder of the
brain parenchyma was normal with no evidence of leptomeningeal enhancement. Positron emission
tomography–computed tomography (PET-CT) of the whole body did not show any pathological
uptake. Chest X-ray was unremarkable. Electrophysiological investigation demonstrated a motor-
predominant acquired distal demyelinating polyneuropathy, consistent with Guillain-Barré
syndrome (GBS). Lumbar puncture gave an opening pressure of 16 cm H2O. Cerebrospinal fluid (CSF)
showed protein of 0.21 g/L, white cell count of 4, and negative culture and cytology. Full blood
count, electrolytes, and renal function and liver