(INTERMEDIET RISK), T
PEMBIMBING Sp
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dr. Bunga
ANAMNESIS
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NYERI DADA memberat ± This is a placeholder. Type
3 hari SMRS, nyeri di Your text here
dada kiri (diitusuk-tusuk/ SESAK hampir ± 4 bulan,
ditimpa beban) menjalar
sampai ke bahu kiri dan
memberat ± 5 hari SMRS.
Muncul saat aktivitas berat
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mandi atau aktivitas tangga, berbaring, dan
ringan, berkurang saat pasien lebih nyaman tidur
istirahat. saat posisi badan setengah NYERI PERUT seperti
duduk, tidur menggunakan ± Marketing
diremas ± 3Research hari SMRS,
5 bantal. Sering terbangun terasa
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disertai here dan
mual,
keringat dingin, mudah lelah
muntah, terasa panas di
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kepala bagian belakang dan kerongkongan, dan
depan. bengkak pada kedua Set Business
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saat
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Pasien mempunyai riwayat Your text here
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Tidak ada anggota keluarga pasien
yang mengalamai keluhan atau
penyakit seperti pasien.
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PEMERIKSAAN FISIK
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Keadaan Umum : Tampak Sakit sedang Temuan abnormal This is a placeholder. Type
Kesadaran : Compos Mentis Mata : CA(+/+) Your text here
Berat Badan : 52 kg Leher : hepatojugular refluks (+), JVP
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Status Gizi : Normal (BMI: 20,31) Paru : ronkhi basah (+/+)
Tekanan Darah : 140/100 mmHg This is a placeholder. Type
Jantung : Batas pinggang jantung pada
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Denyut Nadi : 76 x/menit, Ireguler ICS III linea parasternalis sinistra, batas
Frekuensi Napas : 27 x/menit jantung kanan pada ICS III linea
Temperatur : 36,7’C
Saturasi O2 : 98% Marketing Research
parasternalis dextra, batas jantung kiri
pada ICS VI linea Axillaris Anterior, kesan
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melebar ke lateral Your text here
Abdomen: nyeri tekan (+) di epigastrium
dan kuadran kanan atas, hepar dan
limpa tidak teraba
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Ekstrimitas : edema piting pada tungkai
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+/+, CRT <2 detik Your text here
PEMERIKSAAN
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Hemoglobin : 12,3 g/dl Creatinin : 0,7 mg/dl
Eritrosit : 4,6 x 106/µl Troponin I : 0,03 ng/ml
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Trigliserida : 56 mg/dl
Leukosit : 7000/µl This is a placeholder. Type
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Hematokrit : 36,7 % LDL Kolesterol : 82 mg/dl
Trombosit : 203. 000/µl Gula Darah Sewaktu : 103 mg/dl
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PT : 14,9 detik This is a placeholder. Type
MCV : 79,3 fl Your text here
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Tulang dan jaringan lunak intak,
tidak tampakThis fraktur. CorType
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membesar ke lateral kiri
kranialisasi (+), kalsifikasi aorta
(-) sinus kiri kanan tajam dan
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diafragma kiri kanan dalam Research
batas
normal. Hilus paru tampak
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corakan bronkovaskular
bertambah, tampak infiltrat di
kedua paru
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Kesan kardiomegali Goals
curiga
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dengan edema Your text here
paru.
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MEDIS Write a Plan
• Pr (61 th) nyeri dada sebelah kiri yang memberat ± 3 This
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SMRS, seperti
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ditusuk/ditimpa beban, menjalar sampai ke punggung, muncul saat
aktivitas ringan, berkurang saat istirahat.
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• Sesak yang memberat ± 5 hari SMRS, dirasakan saat Thisberjalan, menaiki
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tangga, berbaring. tidur dengan ± 5 bantal, sering terbangun di malam
hari, mudah lelah, kadang keringat dingin, dan pingsan. Ditemukan
edema kedua tungkai
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• Tekanan darah 140/100 mmHg, denyut nadi 76 x/ This is a placeholder. Type
menit regular,
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frekuensi napas 27 x/menit. Temperatur 36,7oC.
• Hasil EKG ditemukan left ventrikel hipertrofi , dengan gelombang T inversi di lead
II, III, AVF, V5, V6. Set Business Goals
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• Hasil dari foto thorax ditemukan kardiomegali, dan edema paru.
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Diagnosa Klinis: ACS NSTEMI Killip II (Intermediet
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risk), ADHF prec. sicter ACS
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Diagnosis Anatomi: Iskemik inferior lateral
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Quo ad vitam : Dubia ad bonam Your text here
Quo ad sanactionam : Dubia ad malam
Quo ad functionam : Dubia ad malam
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NSTEMI depresi segmen ST ≥ 0,5 mm di V1-V3 dan ≥ 1 mm di sandapan
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lainnya. Selain itu dapat juga dijumpai elevasi segmen ST tidak persisten (<20
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menit), dengan amplitudo lebih rendah dari elevasi segmen ST pada STEMI.
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meningkat dalam
waktu 4 hingga 6
jam, mencapai
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saat 12
jam, danType
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sampai 2 hari
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Pada hasil EKG pasien ini ditemukan gelombang T inversi (tanpa ST elevasi)
Settroponin
pada sadapan II, III, aVF, V5 dan V6, dengan peningkatan Business Goals0,03
sebesar
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ng/ml, sehingga diagnosis NSTEMI inferior lateral dapat ditegakkan.
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Tabel Skor TIMI untuk APTS dan IMA-NEST Your text here
Parameter
Usia > 65 tahun 1
Lebih dari 3 faktor resiko 1
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Angiografi koroner sebelumnya menunjukkan 1 PadaThis
pasien ini, memiliki
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stenosis > 50 % skor TIMI 3
Intermediet risk.
Penggunaan aspirin dalam 7 hari terakhir 1
Setidaknya 2 episode nyeri saat istirahat dalam 24 1
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jam terakhir
Berdasarkan Klasifikasi
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Deviasi ST > 1 mm saat tiba 1 Your text here
GRACE pasien ini memiliki
Peningkatan biomarka jantung (CK, troponin) 1 skor 126 sehingga
*Faktor resiko : hipertensi, DM, merokok, riwayat dalam keluarga, dikategorikan dalam
dislipidemia Set Business
intermediet risk Goals
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Rekomendasi
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obat anti-
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iskemis fase
akut pada ACS This is a placeholder. Type
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NSTEMI
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• Calcium
Channel
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• Nitrat
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• Dual
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antiplatelet
• Antikoagulan
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• Statin
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A Usia 65-74(1) 0 Fungsi ginjal/hati abnormal 0
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Sc Jenis Kelamin :Perempuan (1) 1 Konsumsi obat (aspirin, NSAID) atau alkohol 1
TOTAL 4 Total 1
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Kompensasi
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Gagal
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ALUR DIAGNOSIS GAGAL
JANTUNG
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ALUR DIAGNOSIS
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JANTUNG
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TERAPI GAGAL
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Pada pasien ini fibrilasi
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atrium yang dialami
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adalah fibrilasi atrium
paroksismal
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Berdasarkan hasil anamnesis, pasien datang dengan keluhan kedua kaki bengkak,
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sesak napas dan nyeri dada. Sesak napas dan nyeri dada dirasakan melakukan
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aktivitas. Hasil pemeriksaan fisik dan penunjang foto thorax (kardiomegali, dan
curiga edema paru). Diagnosis klinis yaitu ACS NSTEMI Killip II (Intermediet risk),
dengan ADHF prec. sicter ACS dan Paroxysmal AF,
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2. BUSINESS STARTUP
Indonesia. 2018
Lilly LS. Acute Coronary Syndrome. Rhe JW, Sabatine MS and Lilly LS editor in: Write a Plan Heart Disease:
Pathophysiologyof
A Collaborative Project Of Medical Students And Faculty. 6th Ed. Philadelphia: Lippincott Williams & Wilkins.
2016 This is a placeholder. Type
3. ESC Guidelines for the management of acute coronary syndromes in patients presenting Your text here
without persistent ST-
segment elevation. Europe Heart Journal. 2016;37(3):267–315
4. Perhimpunan dokter spesialis kardiovaskular Indonesia 2015. Pedoman tatalaksana sindrom koroner akut.
PERKI; 2015 Find Investors
5. Pedoman Tatalaksana Fibrilasi Atrium. Edisi Pertama. Perhimpunan Dokter Spesialis Kardiovaskular Indonesia
2014. This is a placeholder. Type
6. Deirdre A.L. Use of the CHA2DS2-VASc and HAS-BLED scores to Aid Decision Making Yourfor
textTrombophylaxis
here in
Nonvalvular Atrial Fibrilation. CirculationAHA. UK. 2012
7. Griffin, Brian; Kapadia, Samir; Rimmerman, Curtis. Cardiology Board Review Second Edition. Cleveland Clinic;
2013
8. Joseph SM, Cedars AM, Ewald GA, Geltman EM, Mann DL. Acute Decompensated Marketing Research
Heart Failure: Contemporary
Medical Management. Texas Heart Institute Journal. 2009;36(6):510-520. This is a placeholder. Type
9. Kasper DL, Hauser SL, Jameson JL, Fauci AS, et al, editor. Harrison’s principles of internal medicine,
Your text here 19th ed. USA;
Mc Graw Hill Education; 2013.
10. ESC Guidelines for the diagnosis and treatment of acute and chronic heart failure. Europe Heart Journal. 2016
11. Siswanto BB, et al. Pedoman Tatalaksana Gagal Jantung, ed 1. Perhimpunan Dokter Spesialis Kardiovaskular
12.
Indonesia;2015. P:3-10.
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Nikoo MH, Amir A, Jorat MV. LBBB: State-of-the-Art Criteria. Int Cardio Res J. 2013 Jun; 7(2): 39-40.
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13. American Heart Association. 2013 ACC/ AHA Guideline on the Assesment of Cardiovascular Risk. Circulation.
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2014: 129: S49-S73
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