GANGGUAN
SISTEM PENCERNAAN
DESKRIPSI
Pembahasan materi meliput gangguan pada
sistem gastro-intestinal, pankreas, hati dan
sistem empedu, berserta gangguan diare
Menjelaskan:
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Mulut (Lanjutan)
GANGGUAN MULUT
Mal-ocllusion = hubungan kurang normal, saat mulut
tertutup, antara gigi atas dan bawah.
Hanya malocllusion yang parah perlu terapi.
Ada tiga tipe:
Tipe 1 (tipe terumum) = rahang normal, namun gigi
tidak tersebar sempurna, terdorong ke atas, rotasi,
sehingga rahang atas dan bawah tidak tertemu
sempurna
Tipe 2 (retrognathism) = pada ini rahang bawah terlalu
terdorong ke belakang, gigi incisors jauh ke depan,
dan molar jauh ke belakang.
Tipe 3 (paling jarang) disebut: prognathism. Rahang
bawah terlalu terdorong ke depan, incisors ke dalam
dan molar jauh ke depan.
GANGGUAN ESOFAGUS
Berbagai gangguan esofagus seringnya
menimbulkan gejala klinis yang sama:
sulit menelan (dysphagia = disfagi) atau
rasa nyeri di belakang dada (nyeri ulu hati)
atau kedua-duanya timbul bersama.
Kadang ada laserasi dan varises yang dapat
menimbulkan perdarahan.
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AKHALASIA (Mega-esofagus)
Disfagi akibat gangguan motilitas esofagus karena
tiga sebab:
(1) peristaltik yang tak adekuat pada 2/3 bagian
bawah.
(2) relaksasi yang tidak adekuat dari otot spinter
esofagus bagian bawah.
(3) meningkatnya otot sphincter saat istirahat
Kelainan tersebut untuk waktu lama mega-esofagus.
Causa: gangguan saraf dan hormonal.
Gejala: disfagia didahului stres emosional berulangulang sakit di belakang sternum, regurgitasi bila
berbaring iritasi kronik Ca.
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Varises esofagus:
Sering terjadi perdarahan masif akut.
Causa:
ulkus peptikum,
gastritis erosiva,
varises esofagus,
laserasi esofagus.
Gejala: berlangsung tak bergejala ruptur (mendadak
terjadi perdarahan hebat tanpa rasa sakit).
Angka kematian lebih tinggi dari perforasi ulkus
peptikum.
Perdarahan baru fatal pada 6-8 minggu post
serangan pertama.
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LAMBUNG
Lambung (Stomach, Gaster)
Makanan masuk lambung dari esofagus dan keluar
ke dalam duodenum.
Selaput lendir lambung mengeluarkan gastric juice
(asam lambung HCL) dan mukus sebagai pelindung.
Bagian fundus lambung ialah lanjutan dari esofagus,
sedangkan bagian antrum menuju ke duodenum.
Masuk/keluar makanan dikontrol oleh esophageal
sphincters dan pyloric sphincter.
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GANGGUAN LAMBUNG
Gangguan bisa terkait lambung sebagai reservoir
makanan, proses mengeluarkan makanan, atau
terkait peran lambung sebagai menyedia makanan
untuk dicerna.
Infeksi:
HCL melindungi lambung dari serangan bakteri,
virus dan jamur yang masuk bersama makanan,
minuman.
Bila pertahanan kalah maka terjadi berbagai ragam
infeksi gastro-intestinal.
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Tumor:
Kanker lambung adalah sebab kematian 15.000/tahun
di USA. Gangguan pencernaan setelah usia 50 th.
sebaiknya diperiksa untuk kemungkinan adanya kanker
lambung (rasa penuh terus, sakit sebelum dan sesudah
makan, tidak ada/ hilang nafsu makan, mudah nausea)
Adanya tumor di bagian atas dekat esophagus akan
mengakibatkan obstruksi dan sulit menelan.
Tumor primer lambung kadang tidak menunjukkan
gejala, baru diketahuai setelah adanya tumor sekunder
di tempat lain. Tumor jinak bisa berupa polyps lambung.
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Ulceration:
HCL bersama getah pencernaan lain yang dihasilkan
lambung kadang menyerang selaput lambungnya
sendiri.
Proteksi terhadap lapisan ini adalah oleh mukus
hasil selaput penutup yang ada dan oleh cepatnya
regenerasi sel bagian dalam pengganti sel-sel
bagian permukaan yang rusak.
Banyak hal bisa mengganggu keseimbangan ini. Satu di
antaranya produk asam HCL lambung yang berlebih.
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Gangguan Autoimmune :
Anemia perniciosa timbul akibat selaput lambung
gagal menghasilkan faktor intrinsik yang
berperan sebagai fasilitator absorpsi vitamin
B12, akibat atropi selaput lambung yang juga
menimbulkan gagal memproduksi HCL lambung.
Perniciosa anemia timbul akibat gangguan autoimun.
Gangguan lain:
Pembesaran lambung bisa:
- akibat ulcus peptic chronic
- komplikasi stenosis pylorus
Gangguan lain: vovulus
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INVESTIGASI LAMBUNG
INVESTIGASI LAMBUNG
Barium X-ray untuk pemeriksaan lambung
Gastroscopy, dan
Biopsy bila perlu
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KANKER LAMBUNG
Tumor ganas primer lambung.
Causa: faktor lingkungan (diet, makan banyak makanan
diasinkan, acar, makanan yang diasap).
Megaloblastic anemia
Gastrectomy partial Blood group A.
Usia di atas 40 th, >laki dari wanita.
Diagnosis: X-ray, gastroscopy, biopsy.
Terapi: Gastrectomy
Yang inoperable radiasi dan obat antikanker.
Diagnostik pre-metastasis prognosis dapat diharapkan
baik.
Di Jepang, dilakukan mass screening dengan
gastroscopy, 85% laju harapan hidup ratarata 5 tahun post-operasi.
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Tumor-2 : - jarang
- lymphoma
- carcinoid (benign)
Colon: - Kanker colon
- Polyposis bisa jadi cancer
Gangguan Aliran Darah
Ischaemia
Obstruksi partial atau komplit arteria dinding
abdomen (atherosclerosis, thrombosis, embolism)
atau akibat pembuluh terjepit (bisa vovulus. Bisa
Intessuseption) atau hernia.
Kehilangan darah pada daerah usus gangrene ->
segera operasi.
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Obstruction:
Bisa akibat tertekan dari luar, gangguan dinding
ususnya (tumor, kanker, Crohns disease, atau
diverticular).
Sumbatan batu empedu, atau intessuception.
Satu yang paling umum adalah paralysis ileus
yang mengakibatkan kontraksi usus berhenti dan isi
usus tidak bisa didorong kembung (meteoristis)
Gangguan lain:
- Peptic ulcers duodenum.
- Ulcerasi usus halus terjadi pada infeksi typhoid dan
Crohns disease. rentan bleeding dan perforasi.
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GANGGUAN HATI
Penyebab utama penyakit hati adalah alkoholic =
alcoholic hepatitis dan cirrhosis
Di Asia. Afrika: sampai 20% populasi adalah carrier
hepatitis virus B, yang mengakibatkan cirrhosis dan
primary liver carcinoma.
Gangguan hati lain adalah
- kongesti, infeksi bakterial dan parasit,
- gangguan sirkulasi, dan metabolisme,
- keracunan dan autoimune.
Gagal hati bisa merupakan hasil akhir dari:
- acute hepatitis
- keracunan
- cirrhosis.
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Keracunan:
- Di luar alkohol, obat-obat yang dipecah di hati
bisa merusak sel hati.
Contoh: Usaha bunuh diri dengan obat analgetika
- Keracunan jamur, makanan tertentu.
Gangguan Autoimun
Masalah utama adalah terjadinya destruksi berlanjut dari
sel hati: - Kronik aktif hepatitis
- Progressive primary biliary cirrhosis
yang lambat laun/menaun.
- Sclerosing cholangitis.
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Gangguan Metabolik:
hemochromatosis
Wilsons disease (copper)
Tumor: - Kanker sekunder dari lambung, pancreas,
usus besar.
- Hepatosplenomegali adalah gejala umum
lymphoma, leukemia
- Hepatoma (kanker primer ganas) jarang.
Lain-lain: - Budd-Chiari Syndrome (sumbatan vena) ->
ascites
- Portal hypertension -> esophagus varices,
ascites, cirrhosis
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GANGGUAN PANCREAS
Keadaan serius terjadi bila fungsi pancreas sebagai
kelenjar terganggu.
Gangguan dan Defek Kongenital:
85% cystic fibrosis, tidak dapat menghasilkan getah
pencernaan malabsorpsi lemak dan protein
steatorrhea dan kemunduran otot.
Pancreatitis kronik, kadang bisa herediter, bisa
menimbulkan diabetes mellitus.
Infeksi: - Acute viral infection (> mump virus)
- Coxsackie virus (bisa DM), Echovirus.
Tumor:
Kanker pancreas adalah umum (sulit terdiagnose,
biasanya ditemukan setelah meluas)
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INVESTIGASI
Hati: -
pemeriksaan fisik
- liver biopsy
LFT
Ultrasound scanning, CT scanning
Empedu: - Cholecystography
Pancreas: - Ultrasound scanning
- Laboratorium darah atau cairan duodenum:
pemeriksaan enzyme pancreas.
- Endoscopy
- ERCP (Endoscopic Retrograde Cholengiopancreatograpgy) X-ray untuk melihat
sistem empedu berikut ductus pancreas.
Dilakukan bila CT-scan, atau US-scan gagal.
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Constitutional Symptoms
Nausea,
Vomiting,
Diarrhea,
Malaise
Fatique
Fever
Night blindness
Pallor
Diaphoresis
Dizziness
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CAUSES of DIARRHEA
1. Gangguan Malabsorption:
Pancreatitis
Pancreatic carcinoma
Crohns disease
2. Gangguan Neuromuscular:
Irritable bowel syndrome
Diabetic enteropathy
Hyperthyroidism
Caffeine
3. Infectious/Inflammatory:
Viral
Protozoal (Gardia)
Bacterial
Pelvic Inflammation
Parasitic
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4. Gangguan Mechanical:
Incomplte obstruction: Fecal impaction (scibala)
Muscular incompetence
Postsurgical effect:
Diverticulitis
neoplasm
adhesions
Stenosis
Heal bypass
Gastrectomy
Intestinal resection
Cholecystectomy
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5. Gangguan Non-Specific:
Crohns disease
Ulceration colitis
Diverticulitis
Diet
Laxative abuse
Food allergy
Antibiotics
Lactose intolerance
Food addictives
Food poisoning
Heavy metal poisoning
Drugs containing magnesium and sorbitol
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CAUSES OF CONSTIPATION
1. Gangguan Neurogenic:
Central nervous system lesions
Cord tumors
Cortical, voluntary, or involuntary evacuation
Multiple sclerosis
Tabes dorsalis
Traumatic spinal cord lesions
2. Gangguan Mechanical:
Bowel obstruction
Extra-alimentary tumors
Pregnancy
Colostomy
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3. Gangguan Muscular:
Amyloidosis
Atony
Dermatomyositis
Duchennes muscular dystrophy
Hypercalcemia
Hyperparathyroidism
Hyperthyroidism
Inactivity
Metabolic defects
Potassium depletion
Severe malnutrition
Systemic sclerosis
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SESI 9b
DESKRIPSI
Materi ajar ini membahas tentang hal-hal yang
harus menjadi perhatian dan harus dikerjakan
para fisioterapis terkait gangguan gastrointestinal
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3. Esophageal Cancer
Lymphatic vessels of the esophagus are continuous with
mediastinal structures and drain to the lymph nodes from
the neck of the celiac axis. Metastasis is via this
lymphatic drainage with tumors of the upper esophagus
metastasizing to the cervical, internal jugular, and
supraclavicular nodes.
The therapist may identify changes in lymph nodes,
requiring medical referral, during on upper-quarter
screening examination.
The usual precautions regarding clients with cancer
apply to neoplasms of the GI system. The primary
concern is the side effects of chemotherapy-induced
bone marrow suppression.
An exercise regimen including aerobic exercise of a
minimal level enhances the immune system and is
incorporated whenever possible.
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4. Esophageal Varices
The primary concerns in therapy are to avoid causing
rupture of varices and proper handling of clients with
known GI bleeding. Carefully instruct the client in proper
lifting techniques and avoid any activities that will
increase intra-abdominal pressure.
For the client with known esophageal varices, observe
closely for signs of behavioral or personality changes.
Report increasing stupor, lethargy, hallucinations, or
neuromuscular dysfunction. Watch for asterixis
(involuntary jerking movement0, a sign of developing
hepatic encephalopathy.
To assess fluid retention, inspect the ankles and sacrum
for dependent edema.
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5. Gastritis
Half of all clients receiving NSAIDs on a chronic basis
have acute gastritis (often asymptomatic).
The therapist should continue to monitor clients for any
symptoms of GI involvement indicating need for medical
referral.
For the client with known chronic GI bleeding, urge the
client to seek immediate attention for recurring.
Urge the client to take prophylactic medications as
prescribed by the physician. Steroids should be taken
with milk, food or antacids to reduce gastric irritation;
antacides can be taken between meals and at bedtime.
Aspirin-containing compounds should be avoided unless
specifically recommended by the physician.
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7. Gastric Adenocarcinoma
Epigastric or back pain, possibly relieved by antacids, is
a frequent complaint that the physician must differentiate
from peptic ulcer disease. Generally the first manifestations of carcinoma are caused by distant metastasis
when the condition is quite advanced.
The therapist may palpate the left supraclavicular lymph
node or the client may point out an umbilical nodule.
After surgery, position changes every 2 hours, deep
breathing, coughing, and incentive spirometry may be
used to prevent pulmonary complications. The semiFlower position (head of the bed raised 6 -> 12 in. with
knees slightly flexed) facilitates breathing and drainage
following any type of gastrectomy.)
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8. Malabsorption Syndrome
In the rehabilitation setting or for the acute care client who
has not been eating solid foods, diarrhea may develop
when the person begins to reestablish a normal diet.
Prolonged viral conditions can wash out the enzymes
normally present in the columnar epithelial cells.
Reestablishing normal eating may require
additional time to restore the enzymatic homeostasis
in the intestines.
9. Intestine Ischemia
Intestinal angina as a result of atherosclerotic plaque
induced ischemia can result in intermittent back pain
(usually at the thoracolumbar junction) with exertion.
Clinical presentation combined with past medical history,
the presence of coronary artery disease risk factors, and
the presence of peripheral vascular disease may also alert
the therapist to the need for a medical referral if the client
has not been medically diagnosed.
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10. Botulism
The sudden onset of rapidly progressive symptoms
associated with botulism is most likely to be reported to a
physician rather than to a therapist.
However, presentation of acute symetrical cranial nerve
impairment (ptosis, diplopia, dysarthria), followed by
descending weakness or paralysis of he muscles in the
extremities or trunk, and dyspnea from respiratory
muscle paralysis, requires immediate medical referral.
After the acute onset and initiation of medical treatment,
treatment is as for cranial nerve palsy. In mild to
moderate cases, there is a gradual recovery of muscle
strength which can take as long as a year after disease
onset; in severe cases, there is a 40% mortality.
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17. Appendicitis
When appendicitis is atypical the client may not
recognize the need for medical attention but will report
the symptoms to the therapist.
Early recognition o the need for medical referral is
important. In an athletic training or physical therapy
setting, appendicitis may present with symptoms of right
thigh, groin (testicular) pain, pelvic pain, or referred pain
to the hip.
In addition to screening for the presence of constitutional
symptoms, a variety of objective test procedures may be
employed by the therapist including the iliopsoas muscle
test and the obturator muscle test. Palpation of
McBurney point. Ask the client to cough: localization of
painful symptoms to the site of the appendix is typical.
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Appendicitis (Lanjutan)
18. Hernia
Early diagnosis is important in preventing incarceration
and strangulation.
Any client experiencing chronic cough pregnancy, or
back, hip, groin, or sacroiliac pain should be asked.
Have you ever been told you have a hernia, or do you
think you have a hernia now.
For the client recovering from surgical repair of a hernia,
heavy lifting and straining should be avoided for 4 to 6
weeks after surgery.
Transient anesthesia of the skin beneath the hernial
incision is a possible postoperative phenomenon.
Whether in the presence of an uncorrected hernia or
postoperatively, the client should avoid activities and
positions that produce painful symptoms associated with
the hernia.
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Hernia (Lanjutan-1)
Hernia (Lanjutran-1)
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Hernia (Lanjutan-2)
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Hernia (Lanjutan)
19. Peritonitis
Special considerations associated with peritonitis are
related to the underlying cause (e.g., liver or kidney
disease, postoperative, cancer) and resultant
complications (e.g., fluid and electrolyte imbalance
pulmonary compromise).
The client with peritonitis is usually hospitalized and
undergoing medical treatment.
The therapist should be familiar with implications
associated with the underlying cause and any
complications present.
Vital signs should be regularly monitored and a
semi-Fowlers position used to help the client breath
deeply with less pain to prevent pulmonary
complications.
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Peritonitis (Lanjutan)
21. Hemorrhoids
Clients involved in any activity requiring increased
abdominal support or causing increased intra-abdominal
pressure should be questioned as to the presence of
hemorrhoids.
For clients with hemorrhoids postoperatively, prone
position or side lying supported with pillows between the
knees and ankles is preferred.
Supine positioning and sitting for brief periods can be
accomplished with a rubber air ring under the buttocks
for support.
Fluid replacement during exercise is important in the
prevention of constipation.
Movement and exercise are also extremely helpful in
preventing constipation.
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