2.1 Definisi Attention Deficit Hyperactivity Disorder (ADHD) atau Hiperaktif.

ADHD adalah istilah popular, kependekan dari attention deficit hyperactivity disorder, (Attention = perhatian,
Deficit = berkurang, Hiperactivity = hiperaktif dan Disorder = gangguan). Diartikan dalam Bahasa Indonesia, ADHD
berarti gangguan pemusatan perhatian disertai hiperaktif.
Attention Deficit Hyperactivity Disorder (ADHD) atau Hiperaktif adalah gangguan perkembangan dalam
peningkatan aktifitas motorik anak-anak hingga menyebabkan aktifitas anak-anak yang tidak lazim dan cenderung
berlebihan. ADHD sekitar tiga kali lebih sering terjadi pada anak laki-laki dibandingkan anak perempuan.
Dr. Seto Mulyadi dalam bukunya “Mengatasi Problem Anak Sehari-hari“ mengatakan pengertian istilah anak
hiperaktif adalah suatu pola perilaku yang menetap pada seorang anak. Perilaku ini ditandai dengan sikap tidak mau
diam, tidak bisa berkonsentrasi dan bertindak sekehendak hatinya atau impulsif.
ADHD, juga dikenal sebagai gangguan perhatian defisit (ADD) atau gangguan hyperkinetic, telah ada lebih lama
daripada kebanyakan orang sadari. Bahkan, kondisi yang muncul untuk menjadi serupa dengan ADHD digambarkan oleh
Hippocrates, yang tinggal 460-370 SM. Nama Perhatian Defisit Disorder pertama kali diperkenalkan pada tahun 1980 di
DSM-III, edisi ketiga dari "Diagnostik dan Statistik Manual of Mental Disorders", digunakan dalam psikiatri. Pada tahun
1994 definisi telah diubah untuk memasukkan tiga kelompok dalam ADHD: jenis dominan hiperaktif-impulsif, tipe
didominasi inatentif, dan jenis gabungan. ADHD biasanya muncul pada masa kanak-kanak tetapi dapat didiagnosis pada
orang dewasa.

2.2 Karakteristik Attention Deficit Hyperactivity Disorder (ADHD) atau Hiperaktif.

Sekarang ini, anak ADHD dibedakan ke dalam tiga tipe. Pertama, tipe ADHD gabungan. Kedua, tipe ADHD
kurang memerhatikan. Ketiga, tipe ADHD hiperaktif impulsive.
1. Tipe ADHD gabungan
Untuk mengetahui ADHD tipe ini, dapat didiagnosis/dideteksi oleh adanya paling sedikit 6 di antara 9 kriteria
untuk ‘perhatian’, ditambah paling sedikit 6 di antara 9 kriteria untuk hiperaktivitas impulsifitas. Munculnya enam gejala
tersebut berkali-kali sampai dengan tingkat yang signifikan disertai adanya beberapa bukti, antara lain sebagi berikut.
a. Gejala-gejala tersebut tampak sebelum anak mencapai usia 7 tahun.
b. Gejala-gejala diwujudkan pada paling sedikit dua tempat yang berbeda.
c. Gejala yang muncul menyebabkan hambatan yang signifikan dalam kemampuan akademik.
d. Gangguan ini tidak dapat dijelaskan dengan lebih baik oleh kondisi psikologi atau psikiatri.
2. Tipe ADHD kurang memerhatikan dan Tipe ADHD hiperaktif impulsif.
Untuk mengetahui ADHD tipe ini, dapat didiagnosis/dideteksi oleh adanya paling sedikit 6 di antara 9 kriteria
untuk ‘perhatian’ dan mengakui bahwa individu-individu tertentu mengalami sikap kurang memerhatikan yang
mendalam tanpa hiperaktifvitas/impulsifitas. Hal ini merupakan salah satu alasan mengapa dalam beberapa buku teks,
kita menemukan ADHD ditulis dengan garis AD/HD. Hal ini membedakan, ahwa ADHD kurang memerhatikan dari jenis
ketiga yang dikenal dengan tipe hiperaktif impulsif.
3. Tipe ADHD hiperaktif impulsive
Tipe ketiga ini menuntut paling sedikit 6 diantara 9 gejala yang terdaftar pada bagian hiperaktif impulsifitas. Tipe
‘ADHD kurang memerhatikan’ ini mengacu pada anak-anak yang mengalami kesulitan lebih besar dengan memori
(ingatan) mereka dan kecepatan motor perceptual (persepsi gerak), cenderung untuk melamun, dan kerap kali menyendiri
secara sosial.

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 2.2.1 Kriteria ADHD dari DSM IV (1994)

Berikut ini kriteria ADHD berdasarkan Diagnostic Statistical Manual.

A.1 Kurang Perhatian
a. Seringkali gagal memerhatikan baik-baik terhadap sesuatu yang detail
b. Seringkali mengalami kesulitan dalam memusatkan perhatian terhadap tugas-tugas atau kegiatan bermain.
c. Seringkali tidak mendengarkan jika diajak bicara langsung
d. Seringkali tidak mengikuti baik-baik intruksi dan gagal dalam menyelesaikan pekerjaan sekolah, pekerjaan atau tugas di
tempat kerja.
e. Seringkali mengalami kesulitan dalam menjalankan tugas dan kegiatan.
f. Seringkali kehilangan barang/benda yang penting untuk tugas-tugas dan kegiatan.
g. Seringkali mengihndari, tidak menyukai atau enggan untuk melaksanakan tugas-tugas yang membutuhkan usaha mental
yang didukung.
h. Seringkali bingung/terganggu oleh rangsangan dari luar.
i. Seringkali lekas lupa dalam menyelesaikan kegiatan sehari-hari.
A.2 Hiperaktifitas Impulsifitas.
Paling sedikit enam atau lebih dari gejala-gejala hiperaktivitas impulsifitas berkutnya bertahan selama paling
sedikit 6 bulan sampai dengan tingkatan yang maladaptif dan tidak dengan tingkat perkembangan.
· Hiperaktivitas.
a. Seringkali gelisah dengan tangan atau kaki mereka dan sering mengggeliat di kursi.
b. Sering meninggalkan tempat duduk di dalam kelas
c. Sering berlarian atau naik-naik secara berlebihan dalam situasi di mana hal ini tidak tepat.
d. Sering mengalami kesulitan dalam bermain atau terlibat dalam kegiatan senggang secara tenang.
e. Sering ‘bergerak’ atau bertindak seolah-olah ‘dikendalikan oleh motor.
f. Sering berbicara berlebihan.
· Impulsifitas
a. Mereka sering memberi jawaban sebelum pertanyaan selesai.
b. Mereka sering mengalami kesulitan menanti giliran.
c. Mereka sering mengintrupsi orang lain.
B. Beberapa gejala hiperaktivitas impulsifitas atau kurang perhatian yang menyebabkan gangguan muncul sebelum anak
berusia 7 tahun.
C. Ada sesuatu di dua atau lebih setting/situasi.
D. Harus ada gangguan yang secara klinis, signifikan di dalam fungsi sosial, akademik, atau pekerjaan.
E. Gejala-gejala tidak terjadi selamanya berlaku PDD,skizofrenia, atau gangguan psikotik lainnya dan tidak dijelaskan
dengan lenbih baik oleh gangguan mental lainnya.

2.3 Faktor Penyebab Attention Deficit Hyperactivity Disorder (ADHD) atau Hiperaktif.
ADHD bukan disebabkan oleh parenting yang buruk, terlalu banyak asupan gula atau MSG, ataupun gara-gara
vaksin. ADHD itu berawal dari masalah biologis yang belum seratus persen dapat dipahami.
Dalam hal ini, tidak ada penyeba tunggal untuk ADHD. Para ahli telah meneliti beberapa kemungkinan dari faktor
genetik dan lingkungan. Berikut ini komentar orang tua mengenai ADHD, “Gangguan ADHD dapat merusak hidup anak,
menghabiskan banyak energy, menimbulkan rasa sakit secara emosional, menurunkan harga diri dan secara serius
merusak hubungan kekerabatan atau pertemanan.”
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 Beberapa hal yang dapat menyebabkan perilaku hiperaktif ialah :
a. Kondisi saat hamil & persalinan. Misalnya keracunan pada akhir kehamilan (ditandai dengan tingginya tekanan darah,
pembengkakan kaki & ekskresi protein melalui urine), cedera pada otak akibat komplikasi persalinan.
b. Cedera otak sesudah lahir,yang disebabkan oleh benturan kuat pada kepala anak.
c. Tingkat keracunan timbal yang parah dapat mengakibatkan kerusakan otak. Hal ini ditandai dengan kesulitan
konsentrasi, belajar dan perilaku hiperaktif. Polusi timbal berasal dari industri peleburan baterai, mobil bekas, asap
kendaraan atau cat rumah yang tua. Obat untuk mengeluarkan timbal dari dalam tubuh hanya diberikan dibawah
pengawasan dokter bagi anak kadar timbalnya sudah sangat tinggi, karena obat tersebut mempunyai efek samping.
d. Lemah pendengaran, yang disebabkan infeksi telinga sehingga anak tidak dapat mereproduksi bunyi yang didengarnya.
Akibatnya, tingkah laku menjadi tidak terkendali & perkembangan bahasanya yang lamban. Segeralah hubungi dokter
THT jika anak menunjukkan ciri berikut: perkembangan bahasa yang lambat, lebih banyak memperhatikan mimik lawan
bicara & lebih banyak berreaksi terhadap perubahan mimik & isyarat.
e. Faktor psikis, yang lebih banyak dipengaruhi oleh hubungan anak dengan dunia luar. Meskipun jarang, hubungan
dengan anggota keluarga dapat pula menjadi penyebab hiperaktivitas. Contoh kasus, orang tua yang bersikap sangat
tegas menyuruh anak berdiri 15 menit di pojok ruangan untuk mengatasi ketidakdisiplinannya. Tapi setelah 15 menit
berlalu, maka anak malah mempunyai energi berlebih yang siap meledak dengan akibat lebih negatif dibanding kesalahan
sebelumnya.
Faktor-faktor penyebab hiperaktif pada anak
1. Faktor neurologik
Insiden hiperaktif yang lebih tinggi didapatkan pada bayi yang lahir dengan masalah-masalah prenatal seperti
lamanya proses persalinan, distres fetal, persalinan dengan cara ekstraksi forcep, toksimia gravidarum atau eklamsia
dibandingkan dengan kehamilan dan persalinan normal. Di samping itu faktor-faktor seperti bayi yang lahir dengan berat
badan rendah, ibu yang terlalu muda, ibu yang merokok dan minum alkohol juga meninggikan insiden hiperaktif
Terjadinya perkembangan otak yang lambat. Faktor etiologi dalam bidang neuoralogi yang sampai kini banyak dianut
adalah terjadinya disfungsi pada salah satu neurotransmiter di otak yang bernama dopamin. Dopamin merupakan zat aktif
yang berguna untuk memelihara proses konsentrasi.
2. Faktor toksik
Beberapa zat makanan seperti salisilat dan bahan-bahan pengawet memilikipotensi untuk membentuk perilaku
hiperaktif pada anak. Di samping itu, kadar timah (lead) dalam serum darah anak yang meningkat, ibu yang merokok dan
mengkonsumsi alkohol, terkena sinar X pada saat hamil juga dapat melahirkan calon anak hiperaktif.

3. Faktor genetik
Didapatkan korelasi yang tinggi dari hiperaktif yang terjadi pada keluarga dengan anak hiperaktif. Kurang lebih
sekitar 25-35% dari orang tua dan saudara yang masa kecilnya hiperaktif akan menurun pada anak. Hal ini juga terlihat
pada anak kembar.
4. Faktor Kultural dan psikososial
a. Pemanjaan.
Pemanjaan dapat juga disamakan dengan memperlakukan anak terlalu manis, membujuk-bujuk makan,
membiarkan saja, dan sebagainya. Anak yang terlalu dimanja itu sering memilih caranya sendiri agar terpenuhi
kebutuhannya.
b. Kurang disiplin dan pengawasan.

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 Anak yang kurang disiplin atau pengawasan akan berbuat sesuka hatinya, sebab perilakunya kurang dibatasi. Jika
anak dibiarkan begitu saja untuk berbuat sesuka hatinya dalam rumah, maka anak tersebut akan berbuat sesuka hatinya
ditempat lain termasuk di sekolah dan orang lain juga akan sulit untuk mengendalikannya.
c. Orientasi kesenangan.
Anak yang memiliki kepribadian yang berorientasi kesenangan umumnya akan memiliki ciri-ciri hiperaktif
secara sosio-psikologis dan harus dididik agak berbeda agar mau mendengarkan dan menyesuaikan diri. Anak yang
mempunyai orientasi kesenangan ingin memuaskan kebutuhan atau keinginan sendiri.
2.4 Pelayanan Pembelajaran Attention Deficit Hyperactivity Disorder (ADHD) atau Hiperaktif.
Menurut Moeslichatoen ada beberapa metode yang cocok untuk membimbing dan
mengarahkan anak. Adapun keseluruhan metode tersebut akan dijelaskan di bawah ini
sebagaimana Moeslichatoen menjelaskan dalam bukunya “Metode pengajaran di
Taman Kanak-Kanak”.

1. Metode Bercerita
Metode cerita juga digunakan oleh Allah untuk mengajarkan kepada manusia tentang
prinsip-prinsip rohani. Dalam cerita terjadi peristiwa yang menarik. Metode cerita bagi
anak-anak usia 3-5 tahun merupakan salah satu pemberian pengalaman belajar
bagi anak secara lisan. Metode bercerita bagi anak usia ini dalam mengajarkan tentang
kebenaran haruslah menarik, mengundang perhatian dan tidak lepas dari konsep
bercerita. Dunia kehidupan anak itu penuh sukacita, maka kegiatan bercerita haruslah
diusahakan dapat memberikan perasaan, gembira, lucu, dan mengasyikkan. Karena
dunia kehidupan anak itu dapat berkaitan dengan lingkungan keluarga, sekolah dan
diluar lingkungannya.
Moeslichatoen mengatakan bahwa ada beberapa macam teknik bercerita yang dapat
dipergunakan antara lain guru atau orang tua dapat membaca langsung dari buku,
menggunakan illustrasi dari buku gambar, menggunakan papan flanel, menggunakan
boneka, bermain peran dalam suatu cerita.
2. Metode tanya-jawab.
Dengan adanya metode tanya-jawab ini akan membuat antara anak dan guru ada
komunikasi. Itu juga diperlukan persiapan yang baik agar dapat memberikan jawaban
yang sesuai dengan kebenarannya. Kadang kala ada anak hiperaktif menanyakan
sesuatu yang dapat membuat guru menjadi bingung untuk menjawabnya. Saat anak
yang memiliki perilaku yang berlebihan itu tidak bisa diam, guru dapat langsung
bertanya kepada anak mengenai cerita yang baru saja diceritakan. Dengan cara ini
maka anak tersebut akan memberikan perhatiannya kepada guru yang bertanya.
Walaupun rentang konsentrasi anak seperti itu sangat singkat.
3. Metode pekerjaan tangan.
Guru/pembimbing anak dapat memberikan metode pekerjaan tangan ini kepada anak
yang memiliki perlaku berlebihan atau yang tidak mau diam, seperti membuat bentuk
dari lilin, melukis dengan kanji yang berwarna warni. Hal tersebut harus dibuat oleh
anak sesuai dengan apa yang diajarkan oleh guru. Dengan adanya metode ini maka
anak yang tidak mau diam tadi dapat diberikan kegiatan diatas, sehingga anak itu tidak
lagi mengganggu teman yang lainnya saat berada di kelas.
4. Metode pemberian tugas.
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 Metode pemberian tugas merupakan tugas atau pekerjaan yang sengaja diberikan
kepada anak yang tidak mau diam, supaya kesempatan si anak untuk mengganggu
temannya mulai berkurang. Pemberian tugas itu juga harus jelas dan penentuan batas
yang tepat diberikan secara nyata. Banyak anak yang mengalami hambatan untuk
memperoleh kemajuan belajar karena tidak menentunya batas tugas yang diberikan
oleh guru untuk diselesaikan. Kejelasan penentuan batas tugas yang harus
diselesaikan anak akan memperkecil kemungkinan anak membuang-buang waktu dan
tenaga untuk suatu kegiatan yang tidak membuahkan hasil dan tidak bermakna bagi
anak. Pemberian tugas kepada anak seperti ini juga harus dapat membangkitkan minat
anak untuk mengembangkan tugas itu secara kreatif. Anak itu tidak akan melakukan
tugas bila yang diberikan oleh guru baginya itu tidak menarik. Pemberian tugas secara
tepat dan profesional akan dapat meningkatkan bagaimana cara belajar yang benar,
sehingga keinginan anak untuk melakukannya timbul pada dirinya sendiri. Bila
pemberian tugas itu menggunakan bahan yang bervariasi, dan sesuai dengan
kebutuhan dan minat anak, maka akan memberikan arti yang besar bagi anak tersebut.
5. Metode bermain.
Metode bermain juga sangat baik diberikan kepada anak tersebut karena anak akan belajar mengendalikan diri
sendiri, memahami dunianya. Dengan menggunakan metode bermain kepada anak seperti ini diperlukan guru-guru
yang harus menemaninya. Melalui kegiatan bermain anak dapat mengembangkan
kreativitasnya, yaitu melakukan kegiatan yang dapat menyalurkan bakat si anak.
Bagi anak seperti ini, metode ini dapat diberikan dan anak akan merasa sangat senang.
Karena anak itu dapat dengan bebas melakukan kegiatannya yang dirasakan cukup
baik bagi dirinya. Melalui kegiatan bermain ini anak dapat menggunakan otot kasar.
Bermacam cara dan teknik dapat dipergunakan dalam kegiatan tersebut seperti
merayap, berlari, merangkak, berjalan, melompoat, menendang, melempar
Guru/pembimbing anak dapat melakukan metode bermain ini sehingga anak tersebut
tidak cepat bosan dengan cara yang diberikan oleh guru. Seperti mengajak anak untuk
bernyanyi yang menggunakan aturan main. Anak seperti ini akan tertarik untuk
melakukannya.
Kegiatan bermain dapat membantu penyaluran kelebihan tenaga. Setelah melakukan
kegiatan bermain anak memperoleh keseimbangan antara kegiatan dengan
menggunakan kekuatan tenaga dan kegiatan yang memerlukan ketenangan. Anak
dapat menyalurkan rasa ingin tahunya dengan menggunakan metode bermain ini
seperti bagaimana caranya memasak, mengapa pohon layu bila tidak diberi air, dan
sebagainya.
Kegiatan menggambar dapat juga diberikan kepada anak hiperaktif termasuk didalam
kegiatan bermain. Anak dalam menggambar dapat menggunakan pensil warna dan
kertas gambar. Cara seperti ini merupakan salah satu kegiatan yang dapat
menyalurkan tenaga pada dirinya.

2.5 Penanganan dan Alternatif Penyembuhan Attention Deficit Hyperactivity Disorder (ADHD) atau Hiperaktif.
Melihat penyebab ADHD yang belum pasti terungkap dan adanya beberapa teori penyebabnya, maka tentunya
banyak sekali terapi atau cara dalam penanganannya sesuai dengan landasan teori penyebabnya. Ada satu hal yang perlu
diketahui, bahwa tak ada penyembuhan ADH. Beberapa terapi untuk anak hiperaktif :
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 1. Terapi Farmakologi
Adalah penanganan dengan menggunakan obat- obatan. Terapi ini hendaknya hanya sebagai penunjang dan
sebagai kontrol terhadap kemungkinan timbulnya impuls impils hiperaktif yang tidak terkendali. Rencana pengobatan
harus dibuat secara individual, tergantung gejala dan efeknya terhadap kehidupan sehari-hari. Penelitian jangka panjang
menunjukkan bahwa kombinasi obat dan terapi lain memberi hasil paling baik.
Pengobatan diberikan bila gejala impulsivitas, agresivitas, dan hiperaktivitas cukup berat sehingga menyebabkan
gangguan di sekolah, di rumah, atau hubungan dengan teman. Pengobatan bertujuan menghilangkan gejala dan sangat
memudahkan terapi psikologis. Lamanya pengobatan tergantung ada atau tidaknya gejala yang ingin dihilangkan.
Saat ini tersedia beragam tipe pengobatan yang dapat digunakan dalam menangani ADHD.
a. Stimulans.
Merupakan jenis obat yang paling sering digunakan dan sudah lebih 50 tahun dipakai dalam menangani ADHD.
Dosis yang diberikan pun bervariasi, ada yang setiap 4 jam adapula yang lebih dari 12 jam . Sementara efek samping
yang mungkin timbul, antara lain, nafsu makan menurun, sakit perut, snewen dan insomnia.
b.NonStimulans
Mulai dianjurkan untuk menangani ADHD pada tahun 2003. Obat ini diketahui mempunyai efek samping yang
lebih sedikit dibandingkan tipe stimulant, dan masa paruh pakainya lebih dari 24 jam.
c. Antidepressants.
Terkadang dijadikan salah satu opsi dalam menangani ADHD, tetapi pada tahun 2004 Food and Drug
Administration (FDA) mengeluarkan peringtan bahwa obat ini dapat memicu gejala aneh yang meningkatkan risiko
terjadinya bunuh diri di antara anak dan remaja yang menggunakannya.
Jika anak kita diberi resep antidepressant, maka penting sekali untuk mendiskusikan risiko-risiko diatas dengan
dokter.
Sebagai catatan, efek yang diberikan dari obat-obatan di atas akan berbeda-beda pada setiap anak. Efek samping
seperti hilangnya selera makan, kesulitan tidur, atau mengantuk didalam kelas kerap kali dapat dikendalikan melalui
penyesuaian dosis obat-obatan. Dengan demikian, untuk menemukan dosis yang paling tepat, dokter akan mencoba
beragam jenis obat dan dosisnya khusunya, bila menangani anak ADHD yang disertai dengan kelainan lainnya.
2. Terapi Bermain
Terapi bermain sangat penting untuk mengembangkan keterampilan, kemampuan gerak , minat dan terbiasa
dengan suasana kompetitif dan kooperatif dalam melakukan kegiatan kelompok. Bermain juga dapat dipakai untuk sarana
persiapan beraktivitas dan bekerja saat dewasa.
3. Terapi Perilaku
Terapi psikososial/perilaku, seperti pelatihan kemampuan sosial, dapat dianjurkan sebagai terapi awal bila gejala
ADHD cukup ringan, diagnosis ADHD belum pasti, atau keluarga memilih terapi ini. Namun, untuk jangka panjangnya,
terapi perilaku saja tidak cukup dalam menangani ADHD.
Berikut beberapa contoh strategi-strategi perilaku yang dapat membantu anak dengan ADHD.
a. Menjadwal Rutinitas Harian.
Cobalah untuk megikuti “jadwal” kegiatan yang sama setiap hari dari bangun tidur sampai tidur lagi. Taruh jadwal
tersebut di tempat yang dapat dilihat dengan mudah sehingga anak pun tahu.
b. Keteraturan dan kerapian.
Taruh tas sekolah, pakaian dan mainan di tempat yang telah ditentukan. Dengan demikian, risiko kehilangan benda-
benda milik pribadi anak menjadi kecil.

c. Mengurangi Distraksi.
Matikan TV, radio dan game komputer khususnya pada saat anak sedang belajar atau mengerjakan PR.
6
d. Batasi pilihan-pilihan.
Jika menawarkan sesuatu, batasi pilihan yang ditawarkan menjadi DUA pilihan saja. Misal, “ Dede mau es krim atau es
cendol”. Kebiasaan di atas dapat mencegah anak dari kebingungan dan overstimulasi.
e. Mengubah Gaya Interaksi dengan Anak.
Anak ADHD lebih mudah memahami perintah dan petunjuk yang ringkas, jelas, dan singkat daripada perintah dan
petunjuk yang banyak penjelasan dan bertele-tele.
f. Buat daftar Goals dan Rewards.
Buat daftar perilaku baik yang berhasil ditampilkan anak sesuai dengan permintaan kita, kemudian beri imbalan yang
pantas kepadanya.
Tetapi, pastikan goal yang kita inginkan itu realistis karena tidak ada yang namanya sukses dalam semalam!
g. Menerapkan Disiplin yang efektif.
Daripada memarahi atau memukul, lebih baik gunakan mettode timeouts atau mengurangi fasilitas-fasilitas yang biasa
kita berikan pada anak sebagai konsekuensi untuk perilaku buruk yang ditampilkan anak.
Untuk anak yang masih kecil, cukup kita alihkan perhatian atau tunggu saja, dia pun akan normal kembali.

h. Bantu Anak Menemukan Bakatnya.

Semua anak perlu mendapat perasaan sukses agar dapat merasa baik terhadap diri mereka sendiri. Temukan dan beri
pujian pada setiap pencapaian yang berhasil ditampilkan anak, apakah itu di bidang olahraga, musik, menggambar,
mengarang dan lain-lain.
Tindakan seperti ini dapat memacu perkembangan keahlian sosial anak serta dapat menumbuhkan rasa percaya diri
anak.

Signs and symptoms

Children with ADHD tend to get distracted from schoolwork rather easily, and they often behave disruptively.[21]

Inattention, hyperactivity, disruptive behavior and impulsivity are common in ADHD.[22][21] Academic difficulties are also
frequent.[21] The symptoms are especially difficult to define because it is hard to draw a line at where normal levels of
inattention, hyperactivity, and impulsivity end and clinically significant levels requiring intervention begin.[23] To be
diagnosed with ADHD, symptoms must be observed in two different settings for six months or more and to a degree that
is greater than other children of the same age.[24]
The symptom categories yield three potential classifications of ADHD—predominantly inattentive type, predominantly
hyperactive-impulsive type, or combined type if criteria for both subtypes are met:[25]
Predominantly inattentive type symptoms as listed by the U.S. National Institute of Mental Health may include:[26]

 Be easily distracted, miss details, forget things, and frequently switch from one activity to another
 Have difficulty maintaining focus on one task
 Become bored with a task after only a few minutes, unless doing something enjoyable

7
 Have difficulty focusing attention on organizing and completing a task or learning something new or trouble
completing or turning in homework assignments, often losing things (e.g., pencils, toys, assignments) needed to
complete tasks or activities
 Not seem to listen when spoken to
 Daydream, become easily confused, and move slowly
 Have difficulty processing information as quickly and accurately as others
 Struggle to follow instructions.

Predominantly hyperactive-impulsive type symptoms may include:[26]

 Fidget and squirm in their seats

 Talk nonstop
 Dash around, touching or playing with anything and everything in sight
 Have trouble sitting still during dinner, school, and story time
 Be constantly in motion
 Have difficulty doing quiet tasks or activities

and also these manifestations primarily of impulsivity:[26]

 Be very impatient
 Blurt out inappropriate comments, show their emotions without restraint, and act without regard for consequences
 Have difficulty waiting for things they want or waiting their turns in games

Most people exhibit some of these behaviors, but not to the degree where such behaviors significantly interfere with a
person's work, relationships, or studies—and in the absence of significant interference or impairment, a diagnosis of
ADHD is normally not appropriate. The core impairments are consistent even in different cultural contexts. [27]
Some children, adolescents, and adults with ADHD have an increased risk of experiencing difficulties with social skills,
such as social interaction and forming and maintaining friendships. About half of children and adolescents with ADHD
experience rejection by their peers compared to 10-15 percent of non-ADHD children and adolescents. Training in social
skills, behavioural modification and medication may have some limited beneficial effects. The most important factor in
reducing emergence of later psychopathology, such as major depression, criminality, school failure, andsubstance use
disorders is formation of friendships with people who are not involved in delinquent activities.[28] Adolescents with
ADHD are more likely to have difficulty making and keeping friends due to impairments in processing verbal
and nonverbal language.[29]
Handwriting difficulties seem to be common in children with ADHD.[30] Delays in speech and language as well as motor
development occur more commonly in the ADHD population.[30][31][32] A 2009 study found that children with ADHD
move around a lot because it helps them stay alert enough to complete challenging tasks.[33][34]

Associated disorders

Inattention and hyperactive behavior are not necessarily the only problems in children with ADHD. ADHD exists alone
in only about 1/3 of the children diagnosed with it. The combination of ADHD with other conditions can greatly
complicate diagnosis and treatment. Many co-existing conditions require other courses of treatment and should be
diagnosed separately instead of being grouped in the ADHD diagnosis.
Some of the associated conditions are:

 Oppositional defiant disorder and conduct disorder, which occur with ADHD at a rate of 50 percent and 20 percent
respectively,[35] are characterized by antisocial behaviors such as stubbornness, aggression, frequent temper tantrums,

8
 deceitfulness, lying, or stealing,[36] inevitably linking these comorbid disorders with antisocial personality
disorder (ASPD); about half of those with hyperactivity and ODD or CD develop ASPD in adulthood.[37] However,
modern brain imaging technology indicates that conduct disorder and ADHD are two distinct disorders. [38]
 Borderline personality disorder, which was according to a study on 120 female psychiatric patients diagnosed and
treated for BPD associated with ADHD in 70 percent of those cases.[39]
 Primary disorder of vigilance, which is characterized by poor attention and concentration, as well as difficulties
staying awake. These children tend to fidget, yawn and stretch and appear to be hyperactive in order to remain alert
and active.[36]
 Mood disorders (especially Bipolar disorder and Major depressive disorder). Boys diagnosed with the combined
ADHD subtype have been shown to be more likely to suffer from a mood disorder.[40]
 Bipolar disorder. Adults with ADHD sometimes have co-morbid bipolar disorder, which requires careful assessment
in order to accurately diagnose and treat both conditions.[41]
 Anxiety disorders, have been found to occur more commonly in the ADHD population.[40]
 Obsessive-compulsive disorder. OCD can co-occur with ADHD and shares many of its characteristics.[36]
 Substance use disorders. Adolescents and adults with ADHD are at a significantly increased risk of developing a
substance abuse problem which can interfere with the evaluation and treatment of ADHD. The most commonly
misused substances by the ADHD population are alcohol and cannabis; serious substance misuse problems should be
treated first due to the serious risks and impairments that occur,[14]:p.38[42] with long-term alcohol misuse and long-
term cannabis misuse and other drug misuse.

There is a strong association between persistent bed wetting and ADHD[43] as well as dyspraxia with up to 50 percent of
dyspraxics having ADHD.[44] Multiple research studies have also found a significant association between ADHD
and language delay.[45] Anxiety and depression are some of the disorders that can accompany ADHD. Academic studies,
and research in private practice suggest that depression in ADHD appears to be increasingly prevalent in children as they
get older, with a higher rate of increase in girls than in boys, and to vary in prevalence with the subtype of ADHD. Where
a mood disorder complicates ADHD, it would be prudent to treat the mood disorder first, but parents of children with
ADHD often wish to have the ADHD treated first, because the response to treatment is quicker.[46]

In children who have a high IQ

There has been some controversy as to whether ADHD children with a high IQ have significant impairments. Research
has shown that high IQ children with ADHD are more likely to repeat grades and have more social and functional
impairments; more than half require additional academic support compared to children without ADHD. Additionally,
more than half of high IQ ADHD people experience major depressive disorder or oppositional defiant disorder at some
point in their lives. Generalised anxiety disorder, separation anxiety disorder and social phobia is also more common in
high IQ ADHD individuals. There is some evidence that high IQ ADHD individuals are not at an increased risk of
substance abuse and conduct disorder compared to low and average IQ ADHD young people. High IQ children and
adolescents with ADHD can have their high intelligence level missed when standard testing is performed; high IQ ADHD
people tend to require more comprehensive testing to detect their true intelligence level.[47] High IQ ADHD children have
a unique neuropsychological profile which typically shows a gap of 20 points or more between the verbal IQ and the
performance IQ when tested on the Wechsler Intelligence Scale for Children; high IQ children without ADHD do not
usually present with this sizable gap.[48]

Cause

The specific causes of ADHD are not known.[49] There are, however, a number of factors that may contribute to, or
exacerbate ADHD. They include genetics, diet and the social and physical environments.

9
Genetics

Twin studies indicate that the disorder is highly heritable and that genetics are a factor in about 75 percent of all
cases.[14] Hyperactivity also seems to be primarily a genetic condition; however, other causes have been identified. [50]
A large majority of ADHD cases may arise from a combination of various genes, many of which
affect dopamine transporters. Candidate genes include DAT1, DRD4, DRD5, 5HTT, HTR1B, andSNAP25. There is also
strong heterogeneity for the associations between ADHD and DAT1, DRD4, DRD5, dopamine beta
hydroxylase, ADRA2A, 5HTT, TPH2, MAOA, and SNAP25.[51] A common variant of a gene called LPHN3 is estimated
to be responsible for about 9% of the incidence of ADHD, and ADHD cases where this gene is present are particularly
responsive to stimulant medication.[52]

Executive functions

Cognitive processes that regulate, control, and manage other cognitive processes are termed "executive functions"
(EF).[53] Examples of such regulated processes are planning, working memory,attention, inhibition, mental flexibility, and
initiation and monitoring of actions.[54] One of the most noticeable neuropsychologic theories of ADHD suggests that its
symptoms arise from a primary deficit in executive functions (EF).[55] A 2005 review found that groups with ADHD
showed significant impairment on all EF tasks. The strongest effects were on measures of response inhibition, vigilance,
working memory, and planning. However, the effect sizes were moderate and there were also individual
differences.[55] Not all individuals with ADHD show deficits in executive functioning;[56] the criteria for an executive
function deficit are met in 30–50% of children and adolescents with ADHD.[57] Furthermore, deficits in EF are not unique
to ADHD.[56] For example, deficits in executive function are seen in individuals with oppositional defiant disorder and
conduct disorder.[56] One study found that 80% of individuals with ADHD was impaired in at least one EF task, compared
to 50% for individuals without ADHD.[58] The 2005 review concluded that deficits in certain executive functions play an
important role in ADHD; however, "EF weaknesses are neither necessary nor sufficient to cause all cases of ADHD". [55]

Evolution

See also: Hunter vs. farmer hypothesis

At more than 1 percent of the population, researchers have proposed that the high prevalence of ADHD may be due
to natural selection having favoured ADHD, possibly because the individual traits may be beneficial on their own, and
only become dysfunctional when these traits combine to form ADHD.[59] The high prevalence of ADHD may in part be
because women in general are more attracted to males who are risk takers, thereby promoting ADHD in the gene pool.[60]
Further evidence that hyperactivity might be evolutionarily beneficial was put forth in a 2006 study finding that it may
carry specific benefits for certain forms of society. In these societies, those with ADHD are hypothesized to have been
more proficient in tasks involving risk, competition, and/or unpredictable behavior (i.e. exploring new areas, finding new
food sources, etc.), where these societies may have benefited from confining impulsive or unpredictable behavior to a
small subgroup. In these situations, ADHD would have been beneficial to society as a whole even while severely
detrimental to the individual.[60] More recent research suggests that because ADHD is more common in mothers who are
anxious or stressed that ADHD is a mechanism of priming the child with the necessary traits for a stressful or dangerous
environment, such as increased impulsivity and explorative behaviour etc.[61] A genetic variant associated with ADHD
(DRD4 48bp VNTR 7R allele) has been found to be at higher frequency in more nomadic populations and those with
more of a history of migration.[62] Consistent with this, another group of researchers observed that the health status of
nomadic Ariaal men was higher if they had the ADHD associated genetic variant (7R alleles). However in recently
sedentary (non-nomadic) Ariaal those with 7R alleles seemed to have slightly worse health.[63]

Environmental

10
ADHD is predominantly a genetic disorder with environmental factors contributing a small role to the etiology of
ADHD. Twin studies have shown that ADHD is largely genetic with 76 percent of the phenotypic variance being
explained by inherited genetic factors.[64][65] Alcohol intake during pregnancy can cause the child to have a fetal alcohol
spectrum disorder which can include symptoms similar to ADHD.[66] Exposure to tobacco smoke during pregnancy
impairs normal development of the feotus including the central nervous system and can increase the risk of the child
being diagnosed with ADHD.[67] Many children exposed to tobacco do not develop ADHD or else only have mild
symptoms which do not reach the threshold of a diagnosis of ADHD. A combination of a genetic vulnerability to
developing ADHD as well as the toxic developmental effects of tobacco on the foetus explain why some children
exposed to tobacco smoke in utero develop ADHD and others don't.[68] Children exposed to lead, even relatively low
levels of lead develop neurocognitive deficits which resemble ADHD and these children can fulfill the diagnostic criteria
for ADHD. There is also some evidence that exposure to polychlorinated biphenyls during childhood causes
developmental damage and can cause ADHD type symptoms which are the diagnosed as ADHD.[69] Exposure to
the organophosphate insecticides chlorpyrifos and dialkyl phosphate is associated with an increased risk of ADHD.
However, the evidence is not definitive as 5 of 17 studies failed to find an association.[70]
Very low birth weight, premature birth and exceptional early adversity increase the risk of the child having ADHD.[71] At
least 30 percent of children who experience a paediatric traumatic brain injury develop ADHD.[72] Infections during
pregnancy, at birth, and in early childhood are linked to an increased risk of developing ADHD. These include various
viruses (measles, varicella, rubella,enterovirus 71) and streptococcal bacterial infection.[73]

Diet

Main article: Diet and attention deficit hyperactivity disorder

Concerns were first raised by Benjamin Feingold, a paediatric allergist that food colourings and additives may affect
children's behaviour in 1973. There is evidence suggesting that some food colourings may make some children
hyperactive. However, the evidence for a link between food colourings and hyperactive behaviour remains uncertain. The
FDA interpreted the evidence as being inconclusive as to whether food colours caused hyperactivity or not. The FDA
review of food colours has been criticised for only doing a very narrow investigation into food colourings and their
possible association with causing hyperactivity instead of investigating their possible effect on neurobehaviour in
general.[74] It is possible that certain food colourings act as a trigger for ADHD symptoms in subgroup of children who
have a genetic vulnerability. The U.K, followed by the European Union took regulatory action on food colourings due to
concerns about their possible adverse effects in children.[75] According to the Food Standards Agency, the food regulatory
agency in the UK, food manufacturers were encouraged to voluntarily phase out the use of most artificial food colors by
the end of 2009. Sunset yellow FCF (E110), quinoline yellow (E104), carmoisine (E122), allura red (E129), tartrazine
(E102) and ponceau 4R (E124) are collectively called the "Southampton six". Following the FSA’s actions, the European
Commission ruled that any food products containing the contentious colourings must display warning labels on their
packaging by 2010.[76]

Social

The World Health Organization states that the diagnosis of ADHD can represent family dysfunction or inadequacies in
the educational system rather than individual psychopathology.[77] Other researchers believe that relationships with
caregivers have a profound effect on attentional and self-regulatory abilities. A study of foster children found that a high
number of them had symptoms closely resembling ADHD.[78] Researchers have found behavior typical of ADHD in
children who have suffered violence and emotional abuse.[14][79] Individuals with posttraumatic stress syndrome(PTSD)
show deficits in executive functions and in attention, and children with PTSD can be misdiagnosed with ADHD.[80]

Social construct theory of ADHD

11
Main article: Social construct theory of ADHD

Social construction theory states that it is societies that determine where the line between normal and abnormal behavior
is drawn. Thus society members including physicians, parents, teachers, and others are the ones who determine which
diagnostic criteria are applied and, thus, determine the number of people affected.[81] This is exemplified in the fact that
the DSM IV arrives at levels of ADHD three to four times higher than those obtained with use of the ICD 10. [11] Thomas
Szasz, a proponent of this theory, has argued that ADHD was "invented and not discovered."[82][83]

Pathophysiology

The pathophysiology of ADHD is unclear and there are a number of competing theories.[84] Research on children with
ADHD has shown a general reduction of brain volume, but with a proportionally greater reduction in the volume of the
left-sided prefrontal cortex. These findings suggest that the core ADHD features of inattention, hyperactivity, and
impulsivity may reflect frontal lobe dysfunction, but other brain regions in particular the cerebellum have also been
implicated.[85] Neuroimaging studies in ADHD have not always given consistent results and as of 2008 are used only for
research and not diagnostic purposes.[86] A 2005 review of published studies involving neuroimaging, neuropsychological
genetics, and neurochemistry found converging lines of evidence to suggest that four connected frontostriatal regions
play a role in the pathophysiology of ADHD: The lateral prefrontal cortex, dorsalanterior cingulate cortex, caudate,
and putamen.[87]
It should be noted that stimulant medication itself may affect growth factors of the central nervous system.[88]
The same laboratory had previously found involvement of the "7-repeat" variant of the dopamine D4 receptor gene,
which accounts for about 30 percent of the genetic risk for ADHD, in unusual thinness of the cortex of the right side of
the brain; however, in contrast to other variants of the gene found in ADHD patients, the region normalized in thickness
during the teen years in these children, coinciding with clinical improvement.[89]
Previously it was thought that the elevated number of dopamine transporters seen in ADHD patients was part of the
pathophysiology of ADHD but it now appears that the reason for elevated striatal dopamine transporter density in ADHD
individuals is due to neuroadaptations occurring due to the continuous exposure to stimulants such as methylphenidate or
dexamphetamine as the body tries to counter-act the effects of the stimulants by developing a tolerance to the stimulant
medications.[90] One interpretation of dopamine pathway tracers is that the biochemical "reward" mechanism works for
those with ADHD only when the task performed is inherently motivating; low levels of dopamine raise the threshold at
which someone can maintain focus on a task that is otherwise boring.[91] There is evidence that people with ADHD have
a low arousal threshold and compensate for this with increased stimuli, which in turn results in disruption of attentional
capacity and an increase in hyperactive behaviour. The reason for this is due to abnormalities in how the dopamine
system in central nervous system responds to stimuli.[92]
Critics, such as Jonathan Leo and David Cohen, who reject the characterization of ADHD as a disorder, contend that the
controls for stimulant medication usage were inadequate in some lobar volumetric studies, which makes it impossible to
determine whether ADHD itself or psychotropic medication used to treat ADHD is responsible for the decreased
thickness observed[93] in certain brain regions. While the main study in question used age-matched controls, it did not
provide information on height and weight of the subjects. These variables it has been argued could account for the
regional brain size differences rather than ADHD itself.[94][95] They believe many neuroimaging studies are oversimplified
in both popular and scientific discourse and given undue weight despite deficiencies in experimental methodology.[94][96]

Diagnosis

ADHD is diagnosed via a psychiatric assessment; to rule out other potential causes or comorbidities, physical
examination, radiological imaging, and laboratory tests may be used.[97]

12
In North America, the DSM-IV criteria are often the basis for a diagnosis, while European countries usually use the ICD-
10. If the DSM-IV criteria are used, rather than the ICD-10, a diagnosis of ADHD is 3–4 times more likely.[11] Factors
other than those within the DSM or ICD however have been found to affect the diagnosis in clinical practice. For
example, children who are the youngest in a class are much more likely to be diagnosed as having ADHD compared to
their older counterparts in the same year. This is because these children may behave more hyperactive not because they
have ADHD but because they are younger and more developmentally behind. It is estimated that about 20 percent of
children who are given a diagnosis of ADHD are misdiagnosed because of the month that they were born in.[98]
Children who display the behavioural symptoms of ADHD but who do not have any significant functional impairments
compared to their age matched peers cannot be diagnosed as having the psychiatric disorder, ADHD.[99]
The previously used term ADD expired with the most recent revision of the DSM. As a consequence, ADHD is the
current nomenclature used to describe the disorder as one distinct disorder that can manifest itself as being a primary
deficit resulting in hyperactivity/impulsivity (ADHD, predominantely hyperactive-impulsive type) or inattention (ADHD,
predominantely inattentive type) or both (ADHD combined type).[24]

Classification

ADHD may be seen as one or more continuous traits found normally throughout the general population. [14] It is
a developmental disorder in which certain traits such as impulse control lag in development. Using magnetic resonance
imaging of the prefrontal cortex, this developmental lag has been estimated to range from 3 to 5 years.[100] However, the
definition of ADHD is based on behaviour and it does not imply a neurological disease.[14] ADHD is classified as
a disruptive behavior disorder along with oppositional defiant disorder, conduct disorder and antisocial personality
disorder.[101]
The Diagnostic and Statistical Manual of Mental Disorders (DSM-IV-TR) classifies ADHD according to the
predominance of symptoms of:

1. Inattention;
2. Hyperactivity–impulsivity;
3. or a combination of both (Combined type).[24]

This subdivision is based on presence of at least six out of nine long-term maladaptive symptoms (lasting at least 6
months) of either inattention, hyperactivity–impulsivity, or both. Thus, a child who is diagnosed with the inattention
subtype may also show signs of hyperactivity–impulsivity, and vice-versa. To be considered, the symptoms must have
appeared before the age of 6, manifest in more than one environment (e.g. at home and at school or work), and not be
better explained by another mental disorder.[24]
Most children with ADHD have the combined type. Children with the inattention subtype are less likely to act out or have
difficulties getting along with other children. They may sit quietly, but without paying attention to what they are doing.
Therefore, the child may be overlooked, and parents and teachers may not notice symptoms of ADHD.[24]

DSM-IV

As with many other psychiatric and medical disorders, the formal diagnosis is made by a qualified professional in the
field based on a set number of criteria. In the USA these criteria are laid down by the American Psychiatric Association
in their Diagnostic and Statistical Manual of Mental Disorders (DSM-IV), 4th edition. Based on the DSM-IV criteria
listed below, three types of ADHD are classified:[99]

1. ADHD, Combined Type is a combination of the two other ADHD subtypes.[99]

2. ADHD Predominantly Inattentive Type presents with symptoms including being easily distracted, forgetful,
daydreaming, disorganisation, poor concentration, and difficulty completing tasks.[99]

13
 3. ADHD, Predominantly Hyperactive-Impulsive Type presents with excessive fidgetiness and restlessness,
hyperactivity, difficulty waiting and remaining seated, immature behaviour; destructive behaviors may also be
present.[99]

For a diagnosis of ADHD to be made the signs must not be due to the course of a Pervasive Developmental
Disorder, Schizophrenia, or other Psychotic Disorder. The signs are not better accounted for by another mental disorder
(such as Mood Disorder, Anxiety Disorder, Dissociative Identity Disorder, or a Personality Disorder).[24]

ICD-10

In the tenth edition of the International Statistical Classification of Diseases and Related Health Problems (ICD-10) the
signs of ADHD are given the name "hyperkinetic disorders". When aconduct disorder (as defined by ICD-10[31]) is
present, the condition is referred to as "hperkinetic conduct disorder". Otherwise the disorder is classified as "disturbance
of activity and attention", "other hyperkinetic disorders" or "hyperkinetic disorders, unspecified". The latter is sometimes
referred to as, "hyperkinetic syndrome".[31]

Other guidelines

The American Academy of Pediatrics Clinical Practice Guideline for children with ADHD emphasizes that a reliable
diagnosis is dependent upon the fulfillment of three criteria:[102]

 The use of explicit criteria for the diagnosis using the DSM-IV-TR.
 The importance of obtaining information about the child’s signs in more than one setting.
 The search for coexisting conditions that may make the diagnosis more difficult or complicate treatment planning.

All three criteria are determined using the patient's history given by the parents, teachers and/or the patient.
Adults often continue to be impaired by ADHD. Adults with ADHD are diagnosed under the same criteria, including the
stipulation that their signs must have been present prior to the age of seven. Questioning parents or guardians as to how
the person behaved and developed as a child also forms part of the clinical assessment of the individual; a family history
of ADHD also adds weight to a diagnosis of adult ADHD due the strong heritability of ADHD.[103] While the core
symptoms of ADHD are similar in children and adults they often present differently in adults than in children, for
example excessive physical activity seen in children may present as feelings of restlessness and constant mental activity
in adults.[103]
The American Academy of Child Adolescent Psychiatry (AACAP) considers it necessary that the following be present
before attaching the label of ADHD to a child:[104]

 The behaviors must appear before age 7.

 They must continue for at least six months.
 The symptoms must also create a real handicap in at least two of the following areas of the child’s life:
 in the classroom,
 on the playground,
 at home,
 in the community, or
 in social settings.

If a child seems too active on the playground but not elsewhere, the problem might not be ADHD. It might also not be
ADHD if the behaviors occur in the classroom but nowhere else. A child who shows some symptoms would not be
diagnosed with ADHD if his or her schoolwork or friendships are not impaired by the behaviors.[104]

Differential

14
To make the diagnosis of ADHD, a number of other possible medical and psychological conditions must be excluded.
Medical conditions that must be excluded as causing the ADHD symptoms include: hypothyroidism, anemia, lead
poisoning, chronic illness, hearing or vision impairment, substance abuse,medication side-effects, sleep
disorders and child abuse,[105] and cluttering (tachyphemia) among others.
As with other psychological and neurological issues, the relationship between ADHD and sleep is complex. In addition to
clinical observations, there is substantial empirical evidence from a neuroanatomic standpoint to suggest that there is
considerable overlap in the central nervous system centers that regulate sleep and those that regulate
attention/arousal.[106] Primary sleep disorders play a role in the clinical presentation of symptoms of inattention and
behavioral dysregulation. There are multilevel and bidirectional relationships among sleep, neurobehavioral functioning
and the clinical syndrome of ADHD.[107]
Behavioral manifestations of sleepiness in children range from the classic ones (yawning, rubbing eyes), to externalizing
behaviors (impulsivity, hyperactivity, aggressiveness), to mood lability and inattentiveness. [106][108][109] Many sleep
disorders are important causes of symptoms that may overlap with the cardinal symptoms of ADHD; children with
ADHD should be regularly and systematically assessed for sleep problems.[106][110]
From a clinical standpoint, mechanisms that account for the phenomenon of excessive daytime sleepiness include:

 Chronic sleep deprivation, that is insufficient sleep for physiologic sleep needs,
 Fragmented or disrupted sleep, caused by, for example, obstructive sleep apnea (OSA) or periodic limb movement
disorder (PLMD),
 Primary clinical disorders of excessive daytime sleepiness, such as narcolepsy and
 Circadian rhythm disorders, such as delayed sleep phase syndrome (DSPS). A study in the Netherlands compared
two groups of unmedicated 6-12-year-olds, all of them with "rigorously diagnosed ADHD". 87 of them had problems
getting to sleep, 33 had no sleep problems. The larger group had a significantly later dim light melatonin
onset (DLMO) than did the children with no sleep problems.[111]
Management

Main article: Attention deficit hyperactivity disorder management

Methods of treatment often involve some combination of Positive behavior support (PBS), life-style changes, counseling,
and medication. A 2005 study found that medical management and behavioral treatment is the most effective ADHD
management strategy, followed by medication alone, and then behavioral treatment.[112] While medication has been
shown to improve behavior when taken over the short term, they have not been shown to alter long-term
outcomes.[113] Medications have at least some effect in about 80% of people.[114] Dietary modifications may also be of
benefit.[115]

Psychosocial

The evidence is strong for the effectiveness of behavioral treatments in ADHD.[116] It is recommended first line in those
who have mild symptoms and in preschool-aged children.[117] Psychological therapies used
include psychoeducational input, behavior therapy, cognitive behavioral therapy (CBT), interpersonal
psychotherapy (IPT), family therapy, school-based interventions, social skills training, parent management
training,[14] neurofeedback,[118] and nature exposure.[119][120] Parent training and education have been found to have short-
term benefits.[121] There is a deficiency of good research on the effectiveness of family therapy for ADHD, but the
evidence that exists shows that it is comparable in effectiveness to treatment as usual in the community and is superior to
medication placebo.[122] Several ADHD specific support groups exist as informational sources and to help families cope
with challenges associated with dealing with ADHD.

15
Medication

methylphenidate (Ritalin) 10 mg tablets

Stimulant medications are the medical treatment of choice.[123][124] There are a number of non-stimulant medications, such
as atomoxetine, that may be used as alternatives.[123] There are no good studies of comparative effectiveness between
various medications, and there is a lack of evidence on their effects on academic performance and social
behaviors.[125] While stimulants and atomoxetine are generally safe, there are side-effects and contraindications to their
use.[123] Medications are not recommended for preschool children, as their long-term effects in such young people are
unknown.[14][126] There is very little data on the long-term benefits or adverse effects of stimulants for ADHD.[127] Any
drug used for ADHD may haveadverse drug reactions such as psychosis and mania,[128] though methylphenidate-induced
psychosis is uncommon. Regular monitoring of individuals receiving long-term stimulant therapy for possible treatment
emergent psychosis has been recommended.[129] Tolerance to the therapeutic effects of stimulants can occur,[90] and abrupt
withdrawal is not recommended.[130] People with ADHD have an increased risk of substance abuse, and stimulant
medications reduce this risk.[131][132] Stimulant medications in and of themselves however have the potential for abuse
and dependence.[133] Guidelineson when to use medications vary internationally, with the UK's National Institute of
Clinical Excellence, for example, recommending use only in severe cases, while most United States guidelines
recommend medications in nearly all cases.[134] Deficiency in zinc is more commonly found in ADHD children compared
to other children. There is evidence that zinc supplementation can benefit ADHD children who have low zinc levels. [135]

Prognosis

Children diagnosed with ADHD have significant difficulties in adolescence, regardless of treatment.[136][137] In the United
States, 37 percent of those with ADHD do not get a high school diploma even though many of them will receive special
education services.[138] A 1995 briefing citing a 1994 book review says the combined outcomes of the expulsion and
dropout rates indicate that almost half of all ADHD students never finish high school.[139] Also in the US, less than 5
percent of individuals with ADHD get a college degree[140] compared to 28 percent of the general population.[141] The
proportion of children meeting the diagnostic criteria for ADHD drops by about 50 percent over three years after the
diagnosis. This occurs regardless of the treatments used and also occurs in untreated children with
ADHD.[105][142][143] ADHD persists into adulthood in about 30 to 50 percent of cases.[7] Those affected are likely to
develop coping mechanisms as they mature, thus compensating for their previous ADHD.[8]

Epidemiology

ADHD's global prevalence is estimated at 3 to 5 percent in people under the age of 19. There is, however, both
geographical and local variability among studies. Children in North America appear to have a higher rate of ADHD than
children in Africa and the Middle East.[145] Published studies have found rates of ADHD as low as 2 percent and as high
as 14 percent among school-aged children.[146] The rates of diagnosis and treatment of ADHD are also much higher on the
east cast of the United States than on its west coast.[147] The frequency of the diagnosis differs between male children
(10%) and female children (4%) in the United States.[148] This difference between genders may reflect either a difference
in susceptibility or that females with ADHD are less likely to be diagnosed than males.[149]
Rates of ADHD diagnosis and treatment have increased in both the UK and the US since the 1970s. In the UK an
estimated 0.5 per 1,000 children had ADHD in the 1970s, while 3 per 1,000 received ADHD medications in the late

16
1990s. In the US in the 1970s 12 per 1,000 children had the diagnosis, while in the late 1990s 34 per 1,000 had the
diagnosis and the numbers continue to increase.[14]
In the UK in 2003 a prevalence of 3.6 percent is reported in male children and less than 1 percent is reported in female
children.[14]:134

History

Hyperactivity has long been part of the human condition. Sir Alexander Crichton describes "mental restlessness" in his
book An inquiry into the nature and origin of mental derangement written in 1798.[150][151] The terminology used to
describe the symptoms of ADHD has gone through many changes over history including: "minimal brain damage",
"minimal brain dysfunction" (or disorder),[152] "learning/behavioral disabilities" and "hyperactivity". In the DSM-II
(1968) it was the "Hyperkinetic Reaction of Childhood". In the DSM-III "ADD (Attention-Deficit Disorder) with or
without hyperactivity" was introduced. In 1987 this was changed to ADHD in the DSM-III-R and subsequent
editions.[153] The use of stimulants to treat ADHD was first described in 1937.[154]

Society and culture

A number of notable individuals have given controversial opinions on ADHD. Scientologist Tom Cruise's interview
with Matt Lauer was widely watched by the public in 2005. In this interview he spoke about postpartum depression and
also referred to Ritalin and Adderall as being "street drugs" rather than as ADHD medication.[155] In
England Baroness Susan Greenfield, a leading neuroscientist, spoke out publicly in 2007 in the House of Lords about the
need for a wide-ranging inquiry into the dramatic increase in the diagnosis of ADHD in the UK and possible causes
following a BBC Panorama programme that highlighted US research (The Multimodal Treatment Study of Children with
ADHD by the University of Buffalo) suggesting drugs are no better than other forms of therapy for ADHD in the long
term.[156] However, in 2010 theBBC Trust criticized the 2007 BBC Panorama programme for summarizing the US
research as showing "no demonstrable improvement in children's behaviour after staying on ADHD medication for three
years" when in actuality "the study found that medication did offer a significant improvement over time" although the
long-term benefits of medication were found to be "no better than children who were treated with behaviour therapy."[157]
As of 2009, eight percent of all Major League Baseball players have been diagnosed with ADHD, making the disorder
common among this population. The increase coincided with the League's 2006 ban on stimulants (q.v. Major League
Baseball drug policy).[158]

Legal status of medications

Stimulants legal status was recently reviewed by several international organizations:

 Internationally, methylphenidate is a Schedule II drug under the Convention on Psychotropic Substances.[159]

 In the United States, methylphenidate and amphetamines are classified as Schedule II controlled substances, the
designation used for substances that have a recognized medical value but present a high likelihood for abuse because
of their addictive potential.[160]
 In the United Kingdom, methylphenidate is a controlled 'Class B' substance, and possession without prescription is
illegal, with a sentence up to 14 years and/or an unlimited fine.[161]
 In Australia, stimulants such as methylphenidate and dexamphetamine are Schedule 8 controlled poisons, and as a
result have strict prescribing rules due to their potential for abuse.[162]
Controversies

Main article: Attention-deficit hyperactivity disorder controversies

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ADHD and its diagnosis and treatment have been considered controversial since the 1970s.[15][16][163] The controversies
have involved clinicians, teachers, policymakers, parents and the media. Opinions regarding ADHD range from not
believing it exists at all[164] to believing there are genetic and physiological bases for the condition as well as
disagreement about the use of stimulant medications in treatment.[16][17][164] Some sociologists consider ADHD to be a
"classic example of the medicalization of deviant behavior, defining a previously nonmedical problem as a medical
one".[15] Most healthcare providers in U.S. accept that ADHD is a genuine disorder with debate in centering mainly
around how it is diagnosed and treated.[18][19][20]
The British Psychological Society said in a 1997 report that physicians and psychiatrists should not follow the American
example of applying medical labels to such a wide variety of attention-related disorders: "The idea that children who
don’t attend or who don’t sit still in school have a mental disorder is not entertained by most British clinicians." [165][166] In
2009, the British Psychological Society, in collaboration with the Royal College of Psychiatrists, released a set of
guidelines for the diagnosis and treatment of ADHD.[167] In its guideline, it states that available evidence indicate that
ADHD is a valid diagnosis and that medication should be the first-line treatment for adults, for children with severe
ADHD, or for children with mild ADHD who do not respond to non-medication interventions. However, it states that the
diagnosis lack any biological basis and that "[c]ontroversial issues surround changing thresholds applied to the definition
of illness as new knowledge and treatments are developed and the extent to which it is acknowledged that clinical
thresholds are socially and culturally influenced and determine how an individual's level of functioning within the 'normal
cultural environment' is assessed". It further states that "the acceptable thresholds for impairment are partly driven by the
contemporary societal view of what is an acceptable level of deviation from the norm."
Others have included that it may stem from a misunderstanding of the diagnostic criteria and how they are utilized by
clinicians,[168]teachers, policymakers, parents and the media.[164] Debates center around key controversial issues; whether
ADHD is a disability or merely a neurological description, the cause of the disorder, the changing of the diagnostic
criteria, the rapid increase in diagnosis of ADHD, and the use of stimulants to treat the disorder.[169] Possible long-term
side-effects of stimulants and their usefulness are largely unknown because of a lack of long-term studies.[170] Some
research raises questions about the long-term effectiveness and side-effects of medications used to treat ADHD.[171]
In 1998, the US National Institutes of Health (NIH) released a consensus statement on the diagnosis and treatment of
ADHD. The statement, while recognizing that stimulant treatment is controversial, supports the validity of the ADHD
diagnosis and the efficacy of stimulant treatment. It found controversy only in the lack of sufficient data on long-term use
of medications, and in the need for more research in many areas.[172]
With a "wide variation in diagnosis across states, races, and ethnicities"[173] some investigators suspect that factors other
than neurological conditions play a role when the diagnosis of ADHD is made.[173][174] Two studies published in 2010
suggest that the diagnosis is more likely to be made in the younger children within a grade; the authors propose that such
a misdiagnosis of ADHD within a grade may be due to different states of maturity and may lead to potentially
inappropriate treatment.[173][174] A further study involving a million children in British Columbia (Canada) published in
2012 using data from 1997 to 2008 unambiguously confirmed the phenomenon, finding children born in December (the
youngest) 39% more likely to be diagnosed with ADHD than those born in January (the oldest).[175]

In adults

Main article: Adult attention deficit hyperactivity disorder

Between 2 and 5 percent of adults have ADHD.[176] Between one-third,[177] and up to 80 percent of the children diagnosed
with ADHD continue having symptoms well into adulthood.[178] Many adults, however, remain untreated.[179] Untreated
adults with ADHD often have chaotic lifestyles, may appear to be disorganized and may rely on non-prescribed drugs and
alcohol to get by.[180]They often have such associated psychiatric comorbidities as depression, anxiety disorder, substance
abuse, or a learning disability.[180] Recognized as occurring in adults in 1978, it is currently not addressed separately from
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ADHD in childhood. Obstacles that clinicians face when assessing adults who may have ADHD include developmentally
inappropriate diagnostic criteria, age-related changes, comorbidities and the possibility that high intelligence or
situational factors can mask ADHD.

Pengertian dan Dinamika Gangguan ADHD (1)

1. Pengertian
ADHD adalah kepanjangan dari Attention Deficit Hiperactivity Disorder. Sedangkan ADD adalah kepanjangan
dari Attention Deficit Disorder. Menurut Maslim (2001: 136) hiperaktif merupakan kegelisahan yang relative berlebihan,
khususnya situasi yang menuntut ketenangan. Batasan hiperaktifitas adalah tingkah laku berlebihan dalam konteks yang
diharapkan pada saat itu dibandingkan dengan anak yang sama usia dan IQnya.
Menurut Widodo, definisi ADHD adalah suatu peningkatan aktifitas motorik hingga pada tingkatan tertentu yang
menyebabkan gangguan perilaku yang terjadi, setidaknya pada dua tempat dan suasana yang berbeda. Aktifitas anak yang
tidak lazim dan cenderung berlebihan yang ditandai dengan gangguan perasaan gelisah, selalu menggerak-gerakkan jari-
jari tangan, kaki, pensil, tidak dapat duduk dengan tenang dan selalu meninggalkan tempat duduknya meskipun pada saat
dimana dia seharusnya duduk degan tenang.. Terminologi lain yang dipakai mencakup beberapa kelainan perilaku
meliputi perasaan yang meletup-letup, aktifitas yang berlebihan, suka membuat keributan, membangkang dan destruktif
yang menetap.
2. Dinamika Gangguan
1.
1. a. Proses Perkembangan ADHD
Untuk memahami jalannya perkembangan ADHD, maka teori mengemukakan bahwa anak ADHD memiliki kekurangan
motivasi, arousal (dorongan), self-regulasi dan penahanan respon (Respon Inhibition). Adapun penjelasannya sebagai
berikut:
a) Motivation Deficit : Anak ADHD menunjukan gangguan pada kesensitifan mereka terhadapreward dan punishment,
b) Arousal Deficit: Anak ADHD pada umumnya memiliki dorongan yang sangat rendah,
c) Self-regulation: Adanya kerusakan pada otak sehingga mereka impulsive dan memiliki kekuatan usaha yang buruk,
d) Behavior Inhibition: Anak ADHD mengalami kesulitan ketika harus mempertahankan responnya terhadap sesuatu.
Hal ini berpengaruh pada kemampuan kognitif, bahasa dan motorik.
Dinamika gangguan ini dapat diawali dari pengaruh genetik. Ibu yang mengkonsumsi alkohol semasa hamilnya atau
mengalami komplikasi kehamilan, akan menyebabkan kerusakan pada dopamin dan kerusakan pada lobus frontalis dan
ganglia basalis. Seluruh kerusakan tersebut memicu gagalnya menekan respon yang tidak tepat, sehingga kemampuan
kognitif (seperti memori, self-directed dan self-regulation menjadi berkurang. Jika kemampuan kognitif mengalami
kemunduran, maka anak akan mengalami inattention (sulit memusatkan perhatian pada sesuatu), hiperaktif (susah diam)
dan impulsif (mengambil suatu tingkah laku tanpa dipikirkan terlebih dahulu).
Perkembangan social anak yang seharusnya berjalan normal menjadi terhambat, karena biasanya anak yang mengalami
hiperaktif, tidak mampu memperhatikan dan impulsive dijauhi oleh peergroupnya. Karena tuntutan tersebut, maka orang
tua anak membentuk suatu pola pengasuhan yang bias saja kurang tepat untuk menangani gangguan tersebut. Pada
akhirnya, anak akan mengalami gangguan tingkah laku (Mash, 2005: 129).
1. b. Komorbid ADHD
Terkadang gangguan ADHD ini tidak hanya menyerang sendiri, terdapat beberapa gangguan lain yang mungkin/biasanya
muncul bersamaan dengan ADHD. Untuk mengetahui komorbid ADHD ini, maka kita harus mengetahui karakteristik
umum dari ADHD ini, diantaranya adalah:
a) Kurangnnya kemampuan kognitif yang berisikan empat fungsi eksekutif (proses control pada otak yang dapat
mengaktifkan, mengintegerasikan dan mengatur fungsi otak lain), meliputi:
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i. Proses kognitif: berkaitan dengan memori, berpikir fleksibel, menggunakan strategi,planning dan antisipasi,
ii. Proses bahasa: meliputi kelancaran berbahasa, komunikasi dan menggunakan self-directed speech,
iii. Proses motorik: melakukan usaha, mengikuti instruksi, mempertahankan respond an mengkoordinasikan
motorik,
iv. Proses emosional: self-regulation untuk dorongan tingkah laku, mentoleransi frustasi dan alasan moral.
b) Kurangnya intelektual: Biasanya anak ADHD memiliki IQ yang rendah,
c) Kerusakan fungsi akademik: Anak ADHD mengalami kesulitan sekolah,
d) Gangguan belajar: Anak ADHD sulit memfokuskan diri pada penjelasan guru sehingga sulit menyelesaikan tugas,
e) Self persepsi yang terganggu (Mash, 2005: 118).
Setelah mengetahui karakteristik yang berhubungan dengan ADHD, maka komorbid pun dapat diketahui. Dalam buku
Wenar (2005: 185-186) menjelaskan komorbid ini diantaranya adalah:
a) ADHD/Distruptive Behavior
Destuptive behavior ini meliputi ODD (Oppositional Defiant Dsorder) dan CD (Conduct Disorder). Presentasinya
mencapai 30-50% subjek. Terdapat sedikit perbedaan antara ADHD dan gangguan tingkah laku ini. ADHD lebih pada
perilaku tidak mengerjakan tugas, kelemahan kognitif, prestasi rendah, prognosis jangka panjang baik. Sedangkan
gangguan tingkah laku, dimanapun anak berada, dia lebih agresif, mungkin memiliki orang tua yang antisosial, rumah
tangga penuh kekerasan, sosioekonomi rendah dan berpotensi melakukan delinkuensi dan penggunaan narkoba
(Davidson, 2006: 679).
b) ADHD/Gangguan Kecemasan
Sekira 10-40% anak ADHD mengalami gangguan kecemasan. Kecemasan berpotensial menimbulkan gejala gangguan
tingkah laku.
c) ADHD/Gangguan Mood
Sekitar 20-30% anak ADHD mengalami pengalaman depresi, sehingga memungkinkan anak tersebut mengalami
perubahan mood yang drastic.
d) ADHD/Gangguan Belajar
Sekitar 19-26% anak ADHD mengalami kesulitan dalam menyelesaikan tugas-tugas akademik karena mereka sulit untuk
memusatkan perhatian.

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